1.Clinical efficiency of three types repair methods on repairing large area defects of deciduous teeth
WANG Shuai ; GAO Yuguang ; LIU Yusan ; ZHANG Bin
Journal of Prevention and Treatment for Stomatological Diseases 2018;26(2):109-112
Objective :
To investigate the clinical effects of different restoration methods on large area defect of deciduous molars.
Methods :
A total of 150 deciduous molars were selected and randomly divided into three groups: A, B and C. Group A was repaired with glass ionomer and compound resin, B group was repaired by Hall technique, and C group was repaired with metal preformed crown. The successful rate of restoration in 6 and 12 months was compared between the three groups.
Results:
There was no significant difference between three groups in A, B and C (P > 0.05) in 6 months; the successful rate of 12 months repair in group B and C was significantly higher than that in group A (P < 0.05).
Conclusion
The success rate of Hall technique and metal performed crown is higher than that of glass ionomer and composite resin on the repair of large defects of deciduous molars.
2.Correlation of LAIR-1 and TGF-βexpression in the peripheral blood of OLP patients
Lihui ZHANG ; Yanjun LI ; Ping YU ; Aili FU ; Jiangnan XUE ; Yusan LIU
Journal of Practical Stomatology 2014;(6):760-763
Objective:To investigate the expression of LAIR-1 on Treg cells and TGF-βin the peripheral blood of patients with oral lichen planus(OLP).Methods:The peripheral blood was obtained from 22 OLP patients and 22 healthy controls.Flow cytometry was used to evaluate the proportion of LAIR-1 on CD4 +Foxp3 +Treg cells,and the level of TGF-βwas measured by ELISA.Results:In the peripheral blood of OLP patients,the proportion of LAIR-1 on CD4 +Foxp3 +Treg cells and TGF-βcontent were significantly higher than those of the healthy controls(P <0.05),The proportion of LAIR-1 on CD4 +Foxp3 +Treg cells was positively correlated with the level of TGF-β(r =0.43,P <0.05).Conclusion:The high expression of LAIR-1 on CD4 +Foxp3 +Treg cells and TGF-βin the pe-ripheral blood of OLP patients may be responsible for the pathogenesis of OLP.


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