Hepatic encephalopathy (HE) is a kind of neuropsychiatric syndrome caused by acute or chronic liver failure or portosystemic venous shunt (hereinafter referred to as portosystemic shunt), which can lead to the occurrence of functional impairment, personality and behavioral abnormalities, coma, and even death. Most patients with cirrhosis combined with HE have spontaneous portosystemic shunt (SPSS), especially those with recurrent or persistent HE. Internal medicine's current standard of treatment for HE associated with SPSS in cirrhotic patients is unsatisfactory, and even after treatment, recurrent HE episodes may still occur. Although interventional therapy has shown significant results and has been applied in clinical practice for many years for SPSS-associated HE, the number of treatment-related cases is relatively small, and there is a lack of large samples and well-designed research. Currently, interventional therapy for SPSS-associated HE in patients with cirrhosis is still under continuous exploration.

Hepatic encephalopathy (HE) is a kind of neuropsychiatric syndrome caused by acute or chronic liver failure or portosystemic venous shunt (hereinafter referred to as portosystemic shunt), which can lead to the occurrence of functional impairment, personality and behavioral abnormalities, coma, and even death. Most patients with cirrhosis combined with HE have spontaneous portosystemic shunt (SPSS), especially those with recurrent or persistent HE. Internal medicine's current standard of treatment for HE associated with SPSS in cirrhotic patients is unsatisfactory, and even after treatment, recurrent HE episodes may still occur. Although interventional therapy has shown significant results and has been applied in clinical practice for many years for SPSS-associated HE, the number of treatment-related cases is relatively small, and there is a lack of large samples and well-designed research. Currently, interventional therapy for SPSS-associated HE in patients with cirrhosis is still under continuous exploration.

China ; epidemiology ; Heat Stroke ; epidemiology ; Humans

A 56-year-old male patient was given traditional Chinese herbal medicines in the form of decoction for arrhythmia.One week later, Squama Manitis 10 g was added into the prescriptions.The erythema, papules with itching appeared in the left leg the next day.That condition was not controlled by stopping taking Chinese herbal medicines and topical use of compound dexamethasone acetate cream.On the third day, the rash even spread to the trunk, arms and legs.The patient was given oral loratadine 10 mg once daily and vitamin C 0.3 g thrice daily.Twenty days later, the symptoms disappeared.The rash in the left leg appeared again 3 days after the patient took Chinese herbal medicines with Squama Manitis by himself.He was cured by using the same treatment.

A 56-year-old male patient was given traditional Chinese herbal medicines in the form of decoction for arrhythmia.One week later, Squama Manitis 10 g was added into the prescriptions.The erythema, papules with itching appeared in the left leg the next day.That condition was not controlled by stopping taking Chinese herbal medicines and topical use of compound dexamethasone acetate cream.On the third day, the rash even spread to the trunk, arms and legs.The patient was given oral loratadine 10 mg once daily and vitamin C 0.3 g thrice daily.Twenty days later, the symptoms disappeared.The rash in the left leg appeared again 3 days after the patient took Chinese herbal medicines with Squama Manitis by himself.He was cured by using the same treatment.