Currently, thoracic endovascular aortic repair (TEVAR) is recommended for type B aortic dissection to reduce long-term aneurysmal progression and lethal aortic events, however, preoperative risk and managements are still unclear. In this report, we present a case of recurrent embolic cerebral infarction associated with stent graft-induced new entry tear (SINE), which required additional surgical treatment. The patient was a 61-year-old male who underwent Zone 3 TEVAR 34 days after the onset of acute type B dissection. Over the course of approximately 1.5 years, he experienced six episodes of recurrent embolic cerebral infarction. A follow-up contrast-enhanced CT scan revealed migration of the stent graft to the distal side and a SINE (stent-induced new entry) in the left subclavian artery distal to the major curvature. Furthermore, a 4D-MRI showed retrograde blood flow from the proximal end of the stent graft extending to the brachiocephalic artery. Therefore, the patient underwent ascending aortic arch replacement with an open stent graft technique. False lumen thrombus caused by SINE in the distal aortic arch has potential risk of repeat cerebral infarction. Early and precise diagnosis, as well as preventive treatment strategies are warranted.

We employed vacuum-assisted closure (VAC) as a treatment modality for wound complications after cardiovasular surgery. Between March and December 2004, 9 patients were treated with VAC, 8 of whom were men, and the mean age was 69.6 years old. Seven patients underwent off-pump coronary artery bypass, and 2 underwent a valve replacement. Six of them had diabetes, 5 had renal dysfunction (4 were dialysis patients), and 2 had chronic obstructive lung disease. Six cases were classified as superficial sternal infection (Superficial) and 3 as a deep sternal infection (Deep). Superficial cases were healed with wound closure after a short period of VAC treatment. However, Deep cases required long duration of VAC treatment and wound closure with a myocutaneous flap in 2 cases, although all of them did not develop mediastinitis requiring closed irrigation and drainage. In 9 cases, with numerous risk factors for poor healing, we found that VAC treatment facilitated wound healing and reduced frequent painful wound care.

A 71-year-old man had been repeatedly admitted to our hospital with congestive heart failure, cerebral infarction and pneumonia. Under a diagnosis of mitral regurgitation and tricuspid regurgitation by echocardiography and catheter examination, mitral valve replacement and tricuspid annuloplasty were performed. Pathohistological study revealed a direct insertion of the papillary muscle into the anterior mitral leaflet (DPM) in addition to post-rheumatic valvular disease. These findings suggest that the increased rigidity of the scarring valve leaflets in combination with direct insertion of DPM lead to inadequate leaflet coaptation and apposition. This is the first report of mitral valve replacement for mitral regurgitation due to post-inflammatory valvular disease with DPM.