Amyand’s hernia is the presence of the appendix within an inguinal hernia sac. A surgical rarity, this accounts for approximately 1% of all inguinal hernias, mostly in children, and presents a unique set of diagnostic and management challenges. This case report details a 61-year-old male presenting with suppurative appendicitis within a recurrent right inguinal hernia, managed through a hybrid approach involving a combination of open and laparoscopic surgical techniques. The purpose of this case report is to raise awareness among surgeons regarding the management of Amyand’s hernia with suppurative appendicitis and to recognize the need for further research pertaining to the use of biosynthetic meshes as an alternative to tension-free tissue-based repairs in such cases.

We present a case series of two patients who developed unilateral cranial nerve III (CNIII) palsy following non-aneurysmal SAH (NASAH). Subarachnoid hemorrhage (SAH) can present with various signs and symptoms. Early diagnosis is paramount to determine treatment course. Thus, clinicians must be aware of the variable clinical presentations of this condition. Two patients were admitted to a single institution for SAH. Patient 1, 52-year-old male, presented with headache, left eye ptosis, and painless diplopia. A non-contrast head computed tomography (CT) demonstrated a SAH within the left sylvian fissure and blood surrounding the mesencephalon and falx. Patient 2, 70-year-old male, presented with mild headache, acute onset of blurry vision, and right eye ptosis. A non-contrast head CT demonstrated a diffuse SAH predominantly in the Sylvian and suprasellar cisterns. Patients were admitted to the neuro intensive care unit and underwent diagnostic angiograms to identify possible aneurysms. Magnetic resonance imaging and angiograms for both patients were negative. Patients were managed with best medical therapy and followed up in the outpatient setting. Unilateral CNIII palsy in the setting of NASAH was identified in both patients. Diagnostic angiograms were negative for aneurysms; therefore, SAH were determined to be spontaneous. We propose that unilateral CNIII palsy is a possible sign of NASAH.