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Performance comparison of estimated glomerular filtration rate equations.

Beom Seok KIM

Kidney Research and Clinical Practice.2014;33(3):168-169. doi:10.1016/j.krcp.2014.07.002

No abstract available.
Glomerular Filtration Rate*

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Improvement of Erdheim-Chester disease-related renal failure after treatment with anakinra.

Manuel Alfredo PODESTA ; Giorgio GRAZIANI ; Francesco REGGIANI ; Michele BUEMI ; Salvatore BADALAMENTI ; Claudio PONTICELLI

Kidney Research and Clinical Practice.2014;33(3):165-167. doi:10.1016/j.krcp.2014.07.007

Erdheim-Chester disease (ECD) is a rare non-Langerhans cell histiocytosis characterized by infiltrates of lipid-laden CD68+/CD1- histiocytes, affecting heart, lungs, central nervous system, and bones. Kidney and adjacent structures can also be affected, leading to renal failure in about 30% of cases. The diagnosis is challenging, and treatment is generally based on administration of interferon-alpha (IFNalpha), but preliminary results also showed the therapeutic efficacy of anakinra, an antagonist of the receptor of interleukin-1 (IL-1). We report the case of an elderly patient with ECD and severe involvement of the heart and kidneys who was successfully treated with anakinra.
Aged ; Central Nervous System ; Diagnosis ; Erdheim-Chester Disease ; Heart ; Histiocytes ; Histiocytosis ; Humans ; Interferon-alpha ; Interleukin 1 Receptor Antagonist Protein* ; Interleukin-1 ; Kidney ; Lung ; Renal Insufficiency*

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Nephrotic syndrome complicated with portal, splenic, and superior mesenteric vein thrombosis.

Bong Soo PARK ; Sihyung PARK ; Kyubok JIN ; Gibok CHOI ; Kang Min PARK ; Kyeong Min JO ; Yang Wook KIM

Kidney Research and Clinical Practice.2014;33(3):161-164. doi:10.1016/j.krcp.2014.07.001

Thromboembolism is a major complication of nephrotic syndrome. Renal vein thrombosis and deep vein thrombosis are relatively common, especially in membranous nephropathy. However, the incidence of portal vein and superior mesenteric vein (SMV) thrombosis in patients with nephrotic syndrome is very rare. To date, several cases of portal vein thrombosis treated by anticoagulation therapy, not by thrombolytic therapy, have been reported as a complication of nephrotic syndrome. Here, we report a case of portal, splenic, and SMV thrombosis in a patient with a relapsed steroid dependent minimal change disease who was treated successfully with anticoagulation and thrombolytic therapy using urokinase. Radiologic findings and his clinical conditions gradually improved. Six months later, a complete remission of the nephrotic syndrome was observed and the follow-up computed tomography scan showed the disappearance of all portal vein, splenic vein, and SMV thrombi.
Follow-Up Studies ; Glomerulonephritis, Membranous ; Humans ; Incidence ; Mesenteric Veins* ; Nephrosis, Lipoid ; Nephrotic Syndrome* ; Portal Vein ; Renal Veins ; Splenic Vein ; Thromboembolism ; Thrombolytic Therapy ; Thrombosis* ; Urokinase-Type Plasminogen Activator ; Venous Thrombosis

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Remission of secondary membranous nephropathy in a patient with Kimura disease after surgical resection.

Sunhwa LEE ; Yong Jin YI ; Hyung Ah JO ; Hyuk HUH ; Kyung Hwan KIM ; Dong Ki KIM ; Hajeong LEE

Kidney Research and Clinical Practice.2014;33(3):157-160. doi:10.1016/j.krcp.2014.06.002

Kimura disease (KD) is an eosinophilic, granulomatous, benign, chronic inflammatory disease with an unknown etiology. A 33-year-old woman visited our hospital because of a palpable, left subclavian mass, a left scapulo-anterior pseudoaneurysm, and nephrotic syndrome. Her subclavian lymph node biopsy examination result was consistent with KD, and results of a renal biopsy indicated secondary membranous nephropathy. After renal histological examination confirmed nephropathy, treatment with prednisolone and cyclosporine was initiated, which was maintained for over 1 year. However, this therapy only provided a transient improvement in proteinuria. One year after commencing the treatment, both proteinuria and azotemia aggravated as the left axillary mass doubled in size. Finally, the mass was surgically excised, following which the azotemia rapidly normalized and proteinuria resolved within 1 month. This case shows that tumor resection in a patient with KD with secondary nephropathy may resolve secondary renal manifestations. Furthermore, reversible renal dysfunction may be caused by unknown secreted molecules.
Adult ; Aneurysm, False ; Angiolymphoid Hyperplasia with Eosinophilia* ; Azotemia ; Biopsy ; Cyclosporine ; Eosinophils ; Female ; Glomerulonephritis, Membranous* ; Humans ; Lymph Nodes ; Nephrotic Syndrome ; Prednisolone ; Proteinuria ; Surgical Procedures, Operative

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A new connecting technique in partial replantation of a ruptured peritoneal dialysis catheter.

Yaeni KIM ; Prashant C DHEERENDRA ; Yong Soo KIM

Kidney Research and Clinical Practice.2014;33(3):154-156. doi:10.1016/j.krcp.2014.06.001

Peritoneal dialysis catheter ruptures have been managed by immediate removal and subsequent reinsertion of the catheter which inevitably entails interruption in peritoneal dialysis and a need for vascular access. A 36-year-old man on continuous ambulatory peritoneal dialysis complaining of dialysate leakage was found to have a small rupture near the outer cuff of the peritoneal dialysis catheter. Rather than employing the traditional method of exchanging the whole catheter, a partial replantation procedure to salvage the still-functioning conduit was performed. Two peritoneal dialysis adaptors were used to connect the end of the remaining old catheter to a new extraperitoneal segment of a new catheter and a piece of a transfer set to connect the adaptors. A novel, yet simple and safe, means of partial peritoneal dialysis catheter replantation when managing catheter injuries is suggested.
Adult ; Catheters* ; Humans ; Kidney Failure, Chronic ; Peritoneal Dialysis* ; Peritoneal Dialysis, Continuous Ambulatory ; Replantation* ; Rupture

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Emphysematous pyelonephritis initially presenting as a spontaneous subcapsular hematoma in a diabetic patient.

Ji Won MIN ; Soon Kyu LEE ; Yu Mi KO ; Ki Wook KWON ; Jeong Uk LIM ; Yeong Bok LEE ; Hye Won LEE ; Yoo Dong WON ; Young Ok KIM

Kidney Research and Clinical Practice.2014;33(3):150-153. doi:10.1016/j.krcp.2014.05.001

Emphysematous pyelonephritis (EPN) is a life-threatening infection characterized by the formation of gas. Complications of EPN include septic shock, acute renal failure, and disseminated intravascular coagulation. Spontaneous subcapsular hematoma (SCH) has also been reported as a rare complication of EPN, although there have been no reports to date of this occurring prior to the presentation of EPN. We report a case of EPN that initially presented as spontaneous SCH. The patient was admitted for left flank pain, and initial computed tomography revealed SCH without any air shadows. Laboratory findings and clinical symptoms suggested the presence of urinary tract infection and the patient was started on antibiotics. Fever developed 24 hours after admission. On follow-up computed tomography 7 days later, EPN was newly observed, and a percutaneous drain was inserted. Blood, urine, and drainage fluid cultures all revealed growth of extended-spectrum beta-lactamase-negative Escherichia coli.
Acute Kidney Injury ; Anti-Bacterial Agents ; Disseminated Intravascular Coagulation ; Drainage ; Emphysema ; Escherichia coli ; Fever ; Flank Pain ; Follow-Up Studies ; Hematoma* ; Humans ; Pyelonephritis* ; Shock, Septic ; Urinary Tract Infections

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The association between acute kidney injury in renal infarction and progression to chronic kidney disease.

Yun Kyu OH

Kidney Research and Clinical Practice.2016;35(3):192-192. doi:10.1016/j.krcp.2016.08.001

No abstract available.
Acute Kidney Injury* ; Infarction* ; Renal Insufficiency, Chronic*

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Henoch–Schönlein purpura nephritis and colitis in an adult patient with alcoholic liver cirrhosis.

Jong Hwan JUNG

Kidney Research and Clinical Practice.2016;35(3):190-191. doi:10.1016/j.krcp.2016.04.003

No abstract available.
Adult* ; Alcoholics* ; Colitis* ; Humans ; Liver Cirrhosis, Alcoholic* ; Nephritis* ; Purpura*

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Cardiac arrest caused by nafamostat mesilate.

Hyo Shik KIM ; Kyung Eun LEE ; Ji Hyun OH ; Chan Sung JUNG ; Dughyun CHOI ; Yunsuek KIM ; Jin Seok JEON ; Dong Cheol HAN ; Hyunjin NOH

Kidney Research and Clinical Practice.2016;35(3):187-189. doi:10.1016/j.krcp.2015.10.003

A 65-year-old man was transferred from the Department of Vascular Surgery to Nephrology because of cardiac arrest during hemodialysis. He underwent incision and drainage for treatment of a buttock abscess. Nafamostat mesilate was used as an anticoagulant for hemodialysis to address bleeding from the incision and drainage site. Sudden cardiac arrest occurred after 15 minutes of dialysis. The patient was treated in the intensive care unit for 5 days. Continuous veno-venous hemodiafiltration was started without any anticoagulant in the intensive care unit. Conventional hemodialysis was reinitiated, and nafamostat mesilate was used again because of a small amount of continued bleeding. Ten minutes after hemodialysis, the patient complained of anaphylactic signs and symptoms such as dyspnea, hypotension, and facial swelling. Epinephrine, dexamethasone, and pheniramin were injected under the suspicion of anaphylactic shock, and the patient recovered. Total immunoglobulin E titer was high, and skin prick test revealed weak positivity for nafamostat mesilate. We first report a case of anaphylactic shock caused by nafamostat mesilate in Korea.
Abscess ; Aged ; Anaphylaxis ; Buttocks ; Death, Sudden, Cardiac ; Dexamethasone ; Dialysis ; Drainage ; Dyspnea ; Epinephrine ; Heart Arrest* ; Hemodiafiltration ; Hemorrhage ; Humans ; Hypotension ; Immunoglobulin E ; Immunoglobulins ; Intensive Care Units ; Korea ; Mesylates* ; Nephrology ; Renal Dialysis ; Skin

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Rhabdomyolysis in a patient taking nebivolol.

Ye Jin KIM ; Hae Ri KIM ; Hong Jae JEON ; Hyun Jun JU ; Sarah CHUNG ; Dae Eun CHOI ; Kang Wook LEE ; Ki Ryang NA

Kidney Research and Clinical Practice.2016;35(3):182-186. doi:10.1016/j.krcp.2015.09.003

β Blockers such as propranolol and labetalol are known to induce toxic myopathy because of their partial β₂ adrenoceptor agonistic effect. Nebivolol has the highest β1 receptor affinity among β blockers, and it has never been reported to induce rhabdomyolysis until now. We report a patient who developed rhabdomyolysis after changing medication to nebivolol. A 75-year-old woman was admitted to our hospital because of generalized weakness originating 2 weeks before visiting. Approximately 1 month before her admission, her medication was changed from carvedilol 12.5 mg to nebivolol 5 mg. Over this time span, she had no other lifestyle changes causing rhabdomyolysis. Her blood chemistry and whole body bone scan indicated rhabdomyolysis. We considered newly prescribed nebivolol as a causal agent. She was prescribed carvedilol 12.5 mg, which she was previously taking, instead of nebivolol. She was treated by hydration and urine alkalization. She had fully recovered and was discharged.
Aged ; Chemistry ; Female ; Humans ; Labetalol ; Life Style ; Muscular Diseases ; Nebivolol* ; Propranolol ; Rhabdomyolysis*

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