SMAD4 depletion enhances NHEJ by regulating BRCA1 expression through ARIH1 in pancreatic cancer.
10.1016/j.apsb.2025.03.035
- Author:
Yiran SONG
1
;
Yazhi HE
1
;
Tianyu YU
1
;
Yang WANG
1
;
Liwei AN
2
;
Yang SHI
3
;
Yingqun ZHOU
4
;
Junyi JU
5
;
Feng WANG
1
Author Information
1. Department of Gastroenterology, Huadong Hospital, Fudan University, Shanghai 200040, China.
2. Department of Medical Ultrasound, Department of Stomatology, Shanghai Tenth People's Hospital, Tongji University Cancer Center, Tongji University School of Medicine, Shanghai 200072, China.
3. Department of Polymer Therapeutics, Institute for Experimental Molecular Imaging, RWTH Aachen University Clinic, Aachen 52074, Germany.
4. Department of Gastroenterology, Shanghai Tenth People's Hospital, Tongji University School of Medicine, Shanghai 200072, China.
5. The Affiliated Taizhou People's Hospital of Nanjing Medical University, Taizhou School of Clinical Medicine, Nanjing Medical University, Taizhou 225300, China.
- Publication Type:Letter
- Keywords:
BRCA1;
Homologous recombination;
Non-homologous end joining;
Pancreatic cancer;
SMAD4
- From:
Acta Pharmaceutica Sinica B
2025;15(5):2787-2790
- CountryChina
- Language:English
