1.Clinical Significance of Hypercapnia during Mechanical Ventilation.
The Korean Journal of Critical Care Medicine 2005;20(2):105-113
No abstract available.
Hypercapnia*
;
Respiration, Artificial*
2.Postoperative delayed hypercapnia and respiratory failure after robot-assisted lower anterior resection.
Korean Journal of Anesthesiology 2013;65(6 Suppl):S115-S116
No abstract available.
Hypercapnia*
;
Respiratory Insufficiency*
3.Postoperative delayed hypercapnia and respiratory failure after robot-assisted lower anterior resection.
Korean Journal of Anesthesiology 2013;65(6 Suppl):S115-S116
No abstract available.
Hypercapnia*
;
Respiratory Insufficiency*
4.Unidirectional valve malfunction by the breakage or malposition of disc: two cases report.
Chol LEE ; Kyu Chang LEE ; Hye Young KIM ; Mi Na KIM ; Eun Kyung CHOI ; Ji Sub KIM ; Won Sang LEE ; Myeong Jong LEE ; Hyung Tae KIM
Korean Journal of Anesthesiology 2013;65(4):337-340
Malfunction of the unidirectional valve in a breathing circuit system may cause hypercapnia from the rebreathing of expired gas, ventilation failure, and barotrauma. Capnography is a useful method for monitoring the integrity of the unidirectional valve. We experienced two cases of malfunction of a unidirectional valve which caused leakage and reverse flow, diagnosed early as a change of the capnographic waveform. One case was caused by expiratory unidirectional valve breakage. The other was caused by an incorrectly-assembled inspiratory unidirectional valve.
Barotrauma
;
Capnography
;
Hypercapnia
;
Respiration
;
Ventilation
5.Thrombotic Complications during Interventional Lung Assist: Case Series.
Eun Jung KIM ; Woo Hyun CHO ; Eun Young AHN ; Dae Gon RYU ; Seung Eun LEE ; Doo Soo JEON ; Yun Seong KIM ; Bong Soo SON ; Do Hyung KIM
Tuberculosis and Respiratory Diseases 2015;78(1):18-22
Interventional lung assist (iLA) effectively reduces CO2 retention and allows protective ventilation in cases of life-threatening hypercapnia. Despite the clinical efficacy of iLA, there are a few major limitations associated with the use of this approach, such as bleeding, thrombosis, and catheter-related limb ischemia. We presented two cases in which thrombotic complications developed during iLA. We demonstrated the two possible causes of thrombotic complications during iLA; stasis due to low blood flow and inadequate anticoagulation.
Extremities
;
Hemorrhage
;
Hypercapnia
;
Ischemia
;
Lung*
;
Thrombosis
;
Ventilation
6.Experiences Using Airway Pressure Release Ventilation for Pneumonia with Severe Hypercapnia or Postoperative Pulmonary Edema.
Kyung Sook HONG ; Young Joo LEE
Korean Journal of Critical Care Medicine 2017;32(1):83-87
No abstract available.
Continuous Positive Airway Pressure*
;
Hypercapnia*
;
Pneumonia*
;
Pulmonary Edema*
7.Experiences Using Airway Pressure Release Ventilation for Pneumonia with Severe Hypercapnia or Postoperative Pulmonary Edema
Kyung Sook HONG ; Young Joo LEE
The Korean Journal of Critical Care Medicine 2017;32(1):83-87
No abstract available.
Continuous Positive Airway Pressure
;
Hypercapnia
;
Pneumonia
;
Pulmonary Edema
8.Brain Stem Blood Flow During Hypercapnia, Hypocapnia, Hypoxia, and Hemorrhagic Hypotension in Newborn Piglet.
Journal of Korean Neurosurgical Society 1995;24(12):1463-1468
Changes of brain stem blood flow(BBF) during physiologic stimulation, such as hypoxia, hypo-and hypercapnia, and hemorrhagic hypotension, were studied by hydrogen clearance technique through closed clival window in the brain stem of nerborn piglet. Induction of moderate hypercapnia(PaCO2=65.4+/-2.11mmHg) caused prominent increases in BBF(354+/-39%;p<0.0001). Conversely, in response to moderate hypocapnia(PaCO2=23.9+/-0.64mmHg), a significant decreases in BBF(-30.38+/-3.22%;p<0.0001) was observed. During severe hypoxia(PaO2=3.7+/-1.4mmHg)moderate increases in BBF(161+/-51.4%;p=0.005) occurred. Interestingly, in response to systemic hypotension(Mean Arterial Blood Pressure=34+/-0.7mmHg), a nonsignificant reduction of BBF was recorded(-14+/-10%;p=0.240). These findings suggest that CO2 reactivity of newborn piglet posterior circulation is high;however, hypoxic reactivity is relatively moderate, and BBF is autoregulated during systemic hypotension.
Anoxia*
;
Brain Stem*
;
Brain*
;
Humans
;
Hydrogen
;
Hypercapnia*
;
Hypocapnia*
;
Hypotension*
;
Infant, Newborn*
9.Management of tracheal compression that's caused by an innominate artery aneurysm: A case report.
Hyun Ju JUNG ; Jong Bun KIM ; Kyong Shil IM ; Duk Ja KIM ; Jae Myeong LEE
Korean Journal of Anesthesiology 2009;57(6):762-764
An aneurysm of the innominate artery could compress the tracheal lumen, and this requires special care. Intubation without intensive monitoring and antihypertensive agents could aggravate the hypercapnia and completely rupture the aneurysm. There are few reports on the airway management of tracheal compression that's caused by an innominate artery aneurysm. We report here on a patient who had a severe hypercapnia after endotracheal intubation above the stenotic area of the tracheal compression, which was caused by an artery aneurysm. Permissive hypercapnia was inadvertently enforced without our knowledge, but the patient recovered without any neurologic problems.
Airway Management
;
Aneurysm
;
Antihypertensive Agents
;
Arteries
;
Brachiocephalic Trunk
;
Humans
;
Hypercapnia
;
Intubation
;
Intubation, Intratracheal
;
Rupture
10.Changes of Gas Values in the CSF and Blood during Sustained Hypoventilation .
Korean Journal of Anesthesiology 1991;24(2):395-403
To evaluate the regulation of and in the CSF during respiratory acidosis, the changes in cisternal CSF and arterial plasma acid-base status were asaessed in anesthetized, paralyzed, mechanically ventilated dogs rendered hypercapneic (PaCO2 of 60~70 mmHg) by hypoventilation. The electrochemical potential difference (u) between the CSF and blood for H+ and HCO2- was calculated from values for and in CSF and arterial plasma, and CSF/plasma DC potential difference was calculated. Simultaneously arterial blood and the CSF samples were drawn at 0, 1, 2, 3, 4, 6, 8 hours after hypercapnia. After 8 hours of respiratory acidosis, the arterial pH declined 0.09 units to 7.26 whereas the CSF pH fell by 0.07 units to 7.29. The CSF rose to 34 mEq/L whereas arterial plasma increased to 29 mEq/L. Therefore, increase of the CSF was almost 11 mEq/L, while the arterial plasma HCO, had increased 7 mE/L. So, much increase in CSF bicarbonate maintained the spinal fluid significantly less acidic than the blood during sustained hypercapnia. CSF/plasma PD was not only increased during acute respiratory acidosis, but also remained high as long as the plasma pH was acid. Values of p for H+ and HCO at 8 hours had returned to +1.1 and -0.1 mV of the control value. The attainment of steady-state values for u close to the control value may be compatible with passive distribution of these ions between the CSF and blood. But active mechanism can not be ruled out, because CSF/plasma PD remained high during the study.
Acidosis, Respiratory
;
Animals
;
Dogs
;
Hydrogen-Ion Concentration
;
Hypercapnia
;
Hypoventilation*
;
Ions
;
Plasma