3.Pulmonary epithelioid hemangioendothelioma: report of a case.
Huan-fen ZHAO ; Chun-nian HE ; Ping LI ; Jin-ping ZHAI ; Xiu-zhi ZHANG
Chinese Journal of Pathology 2010;39(4):277-277
Adenocarcinoma, Bronchiolo-Alveolar
;
metabolism
;
pathology
;
Antigens, CD34
;
metabolism
;
Diagnosis, Differential
;
Female
;
Hemangioendothelioma, Epithelioid
;
metabolism
;
pathology
;
surgery
;
Hemangioma
;
metabolism
;
pathology
;
Hemangiosarcoma
;
metabolism
;
pathology
;
Humans
;
Lung Neoplasms
;
metabolism
;
pathology
;
surgery
;
Middle Aged
;
Vimentin
;
metabolism
4.Isolation and Identification of Seven Symbiotic Bacteria from Local Entomopathogenic Nematodes
Xiu-Fen YANG ; Zheng LIU ; Ran ZHANG ; Huai-Wen YANG ; Jing-Jing YUAN ; Heng JIAN ;
Microbiology 1992;0(02):-
The symbiotic bacterium exists in the intestines of entomopathogenic nematodes and is a potential biological agent.Systematic classification of these bacteria is scarce in China.In this paper,seven strains of symbiotic bacteria from local entomopathogenic nematodes were identified by both observation of mor-phology,physiological,biochemical characteristics and sequence analysis of 16S rDNA fragments.
5.Primary Hodgkin lymphoma of the intestinal tract: report of a case.
Wei-Dong SHI ; Chun-Nian HE ; Huan-Fen ZHAO ; Ping LI ; Chen CHEN ; Xiu-Zhi ZHANG ; Shu-Yan ZHANG
Chinese Journal of Pathology 2008;37(7):493-494
Aged
;
Female
;
Hodgkin Disease
;
diagnosis
;
pathology
;
Humans
;
Reed-Sternberg Cells
;
pathology
6.Effect of fluvastattn on extracelluar signal-regulated kinase activity in glomerular mesangial cells exposed to high glucose concentration
Ying LI ; Hui-Jun DUAN ; Tao ZHANG ; Li-Hong ZHANG ; Mao-Dong LIU ; Xiu-Fen WANG ; Qiong-Zhen LIN ;
Chinese Journal of Endocrinology and Metabolism 2001;0(05):-
When the glomerular mesangial cells of rats were cultured in vitro at high glucose concentration,the activity of extraeellular signal-regulated kinase(ERK),the expression of transforming growth factor-?_1(TGF-?_1)mRNA and the content of typeⅣcollagen in the supematant were higher than those at normal glucose concentration.These effects were inhibited by fluvastatin.The results showed that the activation of ERK signal transduction pathways appeared to play a role in the onset and progression of diabetic nephropathy. Furthermore,fluvastatin could protect the kidney by inhibiting ERK signal transduction pathway and TGF-?_1 expression.
7.Clinicopathologic study of aortic valves in children.
Ping HUANG ; Hong-wei WANG ; Zhen-lu ZHANG ; Xiu-fen HU ; Yan-ping LI ; Pei-xuan CHENG ; Jian-ying LIU
Chinese Journal of Pathology 2006;35(10):623-624
Adolescent
;
Aortic Valve
;
abnormalities
;
Aortic Valve Insufficiency
;
complications
;
pathology
;
surgery
;
Aortic Valve Stenosis
;
complications
;
pathology
;
surgery
;
Child
;
Endocarditis
;
complications
;
pathology
;
surgery
;
Female
;
Heart Defects, Congenital
;
complications
;
pathology
;
surgery
;
Heart Valve Prosthesis Implantation
;
Humans
;
Male
;
Rheumatic Heart Disease
;
complications
;
pathology
;
surgery
8.Screening on high arsenic water source and epidemiological investigation on threatened population of lower reaches of Yellow River in Shangdong Province
Xiu-hong, WANG ; Jian-chao, BIAN ; You-zhang, XIANG ; Shu-fen, GUAN ; Lin, WANG ; Shu-liang, SONG ; Fu-rong, QU
Chinese Journal of Endemiology 2008;27(5):529-531
Objective To screen the arsenic content situation of drinking water in lower reaches of Yellow River and survey the amount of threatened people drinking high arsenic water and the condition of endemic arsenism.Methods Four counties of Yuncheng,Jiaxiang,Dongchangfu and Boxing were selected to colleft the water samples by CroOSS-sectional survey method.The water arsenic content wag determined by semi-quantitative rapid kit.All water samples having arsenic were re-determined by atomic fluorescence spectrometry.And the nurober of threatened people who drinking high arsenic water were investigated.Results In 4765 water wells screened,303 water samples had contained arsenic,arsenic content of 35 samples Was≥0.030 mg/L,12 samples were exceeding the international standard (arsenic content≥0.050 ms/L),they distributed in 3 counties of Dongchangfu,Yuncheng and Jiaxiang.The residents drinking water wells of arsenic content≥0.030 mg/L were surveyed by epidemiological investigation.And in the 28 villages 13 032 residents and 11 Bu8picious patient8 wlere checked out.Conclusion The wells with excesive water arsenic content are existing in the lower reaches of Yellow River and people suspicious of endemic arsenism need to be further identified.
10.Methylation of FHIT gene promoter region in DNA from plasma of patients with myelodysplastic syndromes and demethylating effect of decitabine.
Yin-Fen DENG ; Lei ZHANG ; Xiu-Qun ZHANG ; Ming-Qiu HU ; Dan DAI ; Xue-Zhong ZHANG ; Yan-Li XU
Journal of Experimental Hematology 2012;20(5):1144-1148
This study was aimed to detect the methylation status of FHIT gene promoter region in the DNA from plasma of patients with myelodysplastic syndrome (MDS), and to investigate the demethylating effect of decitabine. Methylation-specific PCR method was used to detect the methylation status of FHIT gene promoter region in the DNA from plasma of 4 patients with MDS before and after treatment with decitabine plus semis CAG therapy (among them, 1 case of newly diagnosed MDS, 3 cases progressed into acute leukemia). The results indicated that 3 cases were found to have an increased methylation in the promoter region. After treatment with decitabine plus semis CAG, increased methylation was reversed in 2 cases. In 4 cases, 2 cases displayed clinical response. It is concluded that FHIT gene hypermethylation is associated with MDS pathogenesis. Decitabine has demethylating effect on the FHIT gene hypermethylation of plasma from MDS patients. Detecting the methylation status of FHIT gene in DNA from plasma may play a role in MDS auxiliary diagnosis or prognosis.
Acid Anhydride Hydrolases
;
genetics
;
Adult
;
Aged
;
Azacitidine
;
analogs & derivatives
;
therapeutic use
;
DNA
;
blood
;
DNA Methylation
;
Female
;
Humans
;
Male
;
Middle Aged
;
Myelodysplastic Syndromes
;
blood
;
drug therapy
;
Neoplasm Proteins
;
genetics
;
Promoter Regions, Genetic