[Objective] To explore the clinical characteristics and pathogenic mechanism of sensory ataxia form of GBS.[Methods] To Summarize clinica1 data of 19 cases with sensory ataxia form of GBS.[Result] The main clinical manifestations were sensory ataxia.The disease relieved and recurred easily and had long course.The protein in CSF increased significantly.Pathological feature was same with general CIDP.Treatment of glucocortieoid was satisfied.[Conclusions] Sensory ataxia form of GBS was one sub-type of CIDP.Pathogenic mechanism was perhaps that immunoreaction attacked proproioceptive sense fibre of radix dorsalis.