1.Baseball elbow and elbow joint instability.
KAZUSHI TEZUKA ; YUKINORI TOMODA ; TOKUHIDE DOI ; AKIRA HIRAOKA ; HIROKO NAKAGAWA ; HARUHI KIKUCHI ; TORU FUKUBAYASHI ; YUTAKA KAMIMAKI ; HITOSHI SHIMOJO ; ATSUSHI MASUJIMA
Japanese Journal of Physical Fitness and Sports Medicine 1988;37(1):37-45
97 baseball players from high school, universities and companies were subjected to this study in order to attempt to clarify the relationship between derangement of the elbow joint and lateral instabilitly by measuring various conventional parameters and the degree of lateral instability.
From the survey by questionnaire, 29 out of 97 players complained of elbow pain, paticularily on the medial side of the joint. The painful phases of the throwing motions were the acceleration phase (23 players, 61%) and release phase (7 players, 46%) . The hyperexten sion of the elbow on the dominant side was significantly smaller than that observed on the undominant side. X-ray examination revealed osteophyte formation at the tip of olecranon and medial joint space.
On the application of 63 kg⋅cm torque force, the varus deflection angle for the dominant side was 8.3° and that for the undominant side was 8.8°. The valgus angle for the dominant side was 12.2° and that for the undominant side was 10.8°. The varus stiffness (kg⋅cm/angle) was 9.5 for the dominant side and 8.4 for the undominant side. Valgus stiffness were 6.3 for the dominant side and 9.2 for the undominant side.
Thses results indicate that the lateral side become stiffer and medial side become looser in the dominant elbow than in the undominant one.
The unphysiological valgus stress at the acceleration phase would induce the osteophyte formation as well as the elongation of the medial collateral ligament on the medial side of the elbow. At the same time the impigement of the olecranon at follow through phase, would induce the osteophyte formation around the olecranon. Thses two factors would considerably influence the range of motion and the instability of the elbow joint.
2.School-aged asthma children with high fractional exhaled nitric oxide levels and lung dysfunction are at high risk of prolonged lung dysfunction
Mari SAITO ; Yutaka KIKUCHI ; Alan Kawarai LEFOR
Asia Pacific Allergy 2019;9(1):e8-
BACKGROUND: Long-term management of bronchial asthma based on the fractional exhaled nitric oxide (FeNO) value alone is not conclusive yet. Therefore, we combined FeNO testing and spirometry, a commonly used test in routine practice, to evaluate acute exacerbation and respiratory function in children with bronchial asthma. OBJECTIVE: We combined FeNO testing and spirometry, commonly used in routine practice, to evaluate acute exacerbations and respiratory function in children with bronchial asthma. METHODS: Subjects were school aged children 7 years and older with bronchial asthma who underwent FeNO testing in January 2015 to May 2016. We evaluated the changes in the frequency of acute exacerbations and respiratory function in the 30 subsequent months. Subjects were divided into 2 groups: those with initial FeNO levels ≥ 21 parts per billion (ppb) (high FeNO) and < 20 ppb (normal FeNO) groups. RESULTS: There were 48 children (33 boys) in the high FeNO group and 68 children (46 boys) in the normal FeNO group. Spirometry was conducted on 83 children (72%) prior to the initial FeNO test, revealing no difference in the ratio of detecting lung dysfunction between the 2 groups. The observation period was 25.8 ± 0.7 and 24.7 ± 0.6 months for the high and normal FeNO groups, respectively. The children in the high FeNO group with lung dysfunction in the initial FeNO test continued to exhibit lung dysfunction at the test at 30 months. In the normal FeNO group, even if lung dysfunction was observed at the initial FeNO, it improved within the 20-month point, and the improvement was maintained thereafter. CONCLUSION: Children with bronchial asthma with high FeNO levels and lung dysfunction are at a higher risk of prolonged lung dysfunction.
Asthma
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Child
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Humans
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Lung
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Nitric Oxide
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Spirometry
3.Gait Instability and Compensatory Mechanisms in Parkinson’s Disease Patients With Camptocormia: An Exploratory Study
Hideyuki URAKAMI ; Yasutaka NIKAIDO ; Yuta OKUDA ; Yutaka KIKUCHI ; Ryuichi SAURA ; Yohei OKADA
Journal of Movement Disorders 2025;18(2):127-137
Objective:
Camptocormia contributes to vertical gait instability and, at times, may also lead to forward instability in experimental settings in Parkinson’s disease (PD) patients. However, these aspects, along with compensatory mechanisms, remain largely unexplored. This study comprehensively investigated gait instability and compensatory strategies in PD patients with camptocormia (PD+CC).
Methods:
Ten PD+CC patients, 30 without camptocormia (PD-CC), and 27 healthy controls (HCs) participated. Self-paced gait tasks were analyzed using three-dimensional motion capture systems to assess gait stability as well as spatiotemporal and kinematic parameters. Unique cases with pronounced forward gait stability or instability were first identified, followed by group comparisons. Correlation analysis was performed to examine associations between trunk flexion angles (lower/upper) and gait parameters. The significance level was set at 0.05.
Results:
Excluding one unique case, the PD+CC group presented a significantly lower vertical center of mass (COM) position (p=0.019) increased mediolateral COM velocity (p=0.004) and step width (p=0.013), compared to the PD-CC group. Both PD groups presented greater anterior‒posterior margins of stability than did the HCs (p<0.001). Significant correlations were found between lower/upper trunk flexion angles and a lower vertical COM position (r=-0.690/-0.332), as well as increased mediolateral COM velocity (r=0.374/0.446) and step width (r=0.580/0.474).
Conclusion
Most PD+CC patients presented vertical gait instability, increased fall risk, and adopted compensatory strategies involving greater lateral COM shift and a wider base of support, with these trends intensifying as trunk flexion angles increased. These findings may guide targeted interventions for gait instability in PD+CC patients.
4.Gait Instability and Compensatory Mechanisms in Parkinson’s Disease Patients With Camptocormia: An Exploratory Study
Hideyuki URAKAMI ; Yasutaka NIKAIDO ; Yuta OKUDA ; Yutaka KIKUCHI ; Ryuichi SAURA ; Yohei OKADA
Journal of Movement Disorders 2025;18(2):127-137
Objective:
Camptocormia contributes to vertical gait instability and, at times, may also lead to forward instability in experimental settings in Parkinson’s disease (PD) patients. However, these aspects, along with compensatory mechanisms, remain largely unexplored. This study comprehensively investigated gait instability and compensatory strategies in PD patients with camptocormia (PD+CC).
Methods:
Ten PD+CC patients, 30 without camptocormia (PD-CC), and 27 healthy controls (HCs) participated. Self-paced gait tasks were analyzed using three-dimensional motion capture systems to assess gait stability as well as spatiotemporal and kinematic parameters. Unique cases with pronounced forward gait stability or instability were first identified, followed by group comparisons. Correlation analysis was performed to examine associations between trunk flexion angles (lower/upper) and gait parameters. The significance level was set at 0.05.
Results:
Excluding one unique case, the PD+CC group presented a significantly lower vertical center of mass (COM) position (p=0.019) increased mediolateral COM velocity (p=0.004) and step width (p=0.013), compared to the PD-CC group. Both PD groups presented greater anterior‒posterior margins of stability than did the HCs (p<0.001). Significant correlations were found between lower/upper trunk flexion angles and a lower vertical COM position (r=-0.690/-0.332), as well as increased mediolateral COM velocity (r=0.374/0.446) and step width (r=0.580/0.474).
Conclusion
Most PD+CC patients presented vertical gait instability, increased fall risk, and adopted compensatory strategies involving greater lateral COM shift and a wider base of support, with these trends intensifying as trunk flexion angles increased. These findings may guide targeted interventions for gait instability in PD+CC patients.
5.Gait Instability and Compensatory Mechanisms in Parkinson’s Disease Patients With Camptocormia: An Exploratory Study
Hideyuki URAKAMI ; Yasutaka NIKAIDO ; Yuta OKUDA ; Yutaka KIKUCHI ; Ryuichi SAURA ; Yohei OKADA
Journal of Movement Disorders 2025;18(2):127-137
Objective:
Camptocormia contributes to vertical gait instability and, at times, may also lead to forward instability in experimental settings in Parkinson’s disease (PD) patients. However, these aspects, along with compensatory mechanisms, remain largely unexplored. This study comprehensively investigated gait instability and compensatory strategies in PD patients with camptocormia (PD+CC).
Methods:
Ten PD+CC patients, 30 without camptocormia (PD-CC), and 27 healthy controls (HCs) participated. Self-paced gait tasks were analyzed using three-dimensional motion capture systems to assess gait stability as well as spatiotemporal and kinematic parameters. Unique cases with pronounced forward gait stability or instability were first identified, followed by group comparisons. Correlation analysis was performed to examine associations between trunk flexion angles (lower/upper) and gait parameters. The significance level was set at 0.05.
Results:
Excluding one unique case, the PD+CC group presented a significantly lower vertical center of mass (COM) position (p=0.019) increased mediolateral COM velocity (p=0.004) and step width (p=0.013), compared to the PD-CC group. Both PD groups presented greater anterior‒posterior margins of stability than did the HCs (p<0.001). Significant correlations were found between lower/upper trunk flexion angles and a lower vertical COM position (r=-0.690/-0.332), as well as increased mediolateral COM velocity (r=0.374/0.446) and step width (r=0.580/0.474).
Conclusion
Most PD+CC patients presented vertical gait instability, increased fall risk, and adopted compensatory strategies involving greater lateral COM shift and a wider base of support, with these trends intensifying as trunk flexion angles increased. These findings may guide targeted interventions for gait instability in PD+CC patients.
6.Report of the second workshop on continuing medical education.
Arito TORII ; Hiroshi KIKUCHI ; Toru ITO ; Tsutomu IWABUCHI ; Kenichi UEMURA ; Michio OGASAWARA ; Kenichi KOBAYASHI ; Shouichi SUZUKI ; Masahiko HATAO ; Shigeru HAYASHI ; Yutaka HIRANO ; Motokazu HORI ; Susumu TANAKA
Medical Education 1987;18(2):97-106
8.Continuing Medical Education in Universities. Questionnaire Analysis of Present Status. (The 2nd Report).
Kenichi KOBAYASHI ; Tsutomu IWABUCHI ; Hiroshi KIKUCHI ; Masahiko HATAO ; Shigeru HAYASHI ; Yutaka HIRANO ; Hiroshi HAMADA ; Takao NAKAGI ; Kazuo SAITO ; Osamu NISHIZAKI ; Ryoichi NISHIMURA ; Arito TORII
Medical Education 1992;23(1):50-54
9.Objectives for Practicing Physicians' Continuing Medical Education.
Yutaka HIRANO ; Hiroshi KIKUCHI ; Kenichi KOBAYASHI ; Masahiko HATAO ; Tsutomu IWABUCHI ; Akira KURAI ; Naohiko MIYAMOTO ; Takao NAKAKI ; Osamu NISHIZAKI ; Hirosuke SUZUKI ; Kyoichi UENO ; Daizo USHIBA
Medical Education 1994;25(6):365-367
10.Check List for Practicing Physicians' Continuing Medical Education.
Yutaka HIRANO ; Hiroshi KIKUCHI ; Kenichi KOBAYASHI ; Masahiko HATAO ; Tsutomu IWABUCHI ; Akira KURAI ; Naohiko MIYAMOTO ; Takao NAKAKI ; Osamu NISHIZAKI ; Hirosuke SUZUKI ; Kyoichi UENO ; Daizo USHIBA
Medical Education 1995;26(1):51-61