Delayed anoxic encephalopathy after carbon monoxide (CO) poisoning is characterized by neurological deterioration that occurs after recovery from acute CO intoxication. There has been no established therapy. We report a patient recovered from acute CO intoxication developed various neurological symptoms. After the administration of high dose prednisolone and anticholinesterase inhibitor, the therapeutic effect was remarkable and confirmed by quantitative analysis of diffusion-tensor imaging (DTI). DTI could be used to evaluate the therapeutic effect for delayed anoxic encephalopathy after CO poisoning.
Carbon Monoxide Poisoning ; Carbon Monoxide ; Carbon ; Diffusion Tensor Imaging ; Humans ; Hypoxia, Brain ; Leukoencephalopathies ; Magnetic Resonance Imaging ; Poisoning ; Prednisolone