No abstract available.
Capsid Proteins* ; Capsid* ; Genome*

Most subdural hematoma with significantly differed attenuation from that of adjacent brain tissue can beaccurately diagnosed by CT. Difficulty arises when the hematoma is isodense that is exhibited similar attenuationto that of brain. Unilateral isodense subdural hematoma can be identified by indirect sign such as mass effect.Occasionally, the use of intravenous contrast material to aid in identifying isodense subdural hematomas has metwith variable success. Moreover, bilateral isodense subdural hematoma may be more difficult. We therfore considerdit of interest to evalute the diagnostic efficiency of CT in isodense subdural hematomas. We have analysed 13cases surgically provened cases of isodense subdural hematoma examined at Korea General Hospital from Oct. 1981 toApril. 1982. The resuls were as follows; 1. One hundred twenty seven cases of subdural hematomas were studied byCT, 13 cases (19.2%) of which were isodense. 2. The age distribution was from 29 years to 69 years and mean agewas 52 years. The sex ratio was 11 male to 2 female. 3. Seven(53.8%) of 13 cases has a history of head trauma. 4.The time interval which subdural hematoma became isodense was from 1 week to 4 months and peak time interval wasfrom 1 week to 3 weeks. 5. The precontrast CT of isodense subdural hematoma appeared shifting of midlinestructure, compression and deformity of the ventricles in all 13 cases, effacement of cerebral sulci in 10 cases(76.9%) and dilatation of contralateral ventricles in 4 cases (30.8%). 6. The postcontrast CT scan demonstratedenhancement of the medial margin of the lesion in 4(30.8%) of 13 cases and displacement of cortical vein away fromthe inner table of the skull in 3 (23.1%) of 13 cases. 7. Bilateral isodense subdural hematomas were 2 (15.4%) of13 cases.
Age Distribution ; Brain ; Congenital Abnormalities ; Craniocerebral Trauma ; Dilatation ; Female ; Hematoma ; Hematoma, Subdural ; Hospitals, General ; Humans ; Korea ; Male ; Sex Ratio ; Skull ; Tomography, X-Ray Computed ; Veins

Calcific tendinitis of the shoulder joint is common disease causing acute pain, mainly involving the supraspinatus or infraspinatus muscle, and less frequently the teres minor or subscapularis muscle. This study reports on the satisfactory arthroscopic removal of calcium deposits as well as infraspinatus and supraspinatus muscle repair without relapse via minimal incision using suture anchors. This was a case of atypically extensive calcific tendinitis involving the infraspinatus muscle, with a bursal side partial rupture of the supraspinatus muscle in a 61-year-old female whose chief complaint was chronic pain of the right shoulder exacerbated by limited movement.
Acute Pain ; Calcium ; Chronic Pain ; Female ; Humans ; Middle Aged ; Recurrence ; Rupture ; Shoulder Joint ; Shoulder* ; Suture Anchors ; Tendinopathy*

No abstract available.

No abstract available.
Lymphomatoid Papulosis* ; Mycobacterium* ; Tuberculosis*

No abstract available.
Lymphomatoid Papulosis* ; Mycobacterium* ; Tuberculosis*

No abstract available.
Adult* ; Humans ; Life Style*

The Farnsworth-Munsell 100 Hue Test is very useful clinical color discrimination test for detecting color deficient person in quantitative and qualitative diagnosis. In this study 45 subjects were examined by the F-M Test and of these 22 were diagnosed as normal and 23 as color anomalous. The 23 anomalous persons were composed of 6 deutans, 9 protans and 8 unclassified red-green defects. These 23 color anomalous subjects were examined by the three kinds of pseudoisochromatic plates), Ishihara (15 plates), Dvorine (15 plates) and Ao H-R-R (20 plates), and their results were compared with the results of the Farnsworth-Munsell test.
Diagnosis ; Discrimination (Psychology) ; Humans

"It was reported that polymorphism of TNF alpha gene was present in promoter region and involves the substitution of guanine by adenosine in the uncommon (TNFA 2) allele. In this study, we investigated the significance of TNFA gene polymorphism in relation to various clinical characteristics and autoantibody profiles in SLE as well as comparing it with that of other countries, and also studied its association with peripheral TNF-a production in vitro. TNFA genotyping was performed in 126 SLE patients and 300 controls using DNA extracted from peripheral leucocytes. The biallelic polymorphism at position -308 of the TNFA promotor was determined by Ncol- RFLP. Peripheral mononuclear cell production of TNF-a was investigated by bioassay using L-929 cell cytotoxicity. The TNFA ""1 homozygote was a predominant allele (77.0%) in SLE, which was not different from the controls. TNFA ""2 homozygate was extremely rare in both patients and controls (0.8%, 1.3% respectively). The clinical manifestations between TNFA '1 and TNFA""2 did not differ. The production of autoantibodies including dsDNA, anti-La, anti-nRNP and anti-Sm was not different between two alleles, whereas anti- Ro antibody was more frequent in TNFA""1/TNFA '1 than in TNFA'1/TNFA'2 (62.1% vs 38.4%, P=0.022). The polymorphism of TNFA gene did not influence the lipopolysaccharide stimulated peripheral mononuclear cell production of TNF-a (1356+/-293 vs 1119+/-385 pg/ml; TNFA'1/TNFA'1, TNFA'1/TNFA'2 respectively). These results suggested that promoter polymorphism of TNFA was not directly involved in the susceptibility of SLE and was not responsible for differential peripheral TNF-a production, but TNFA ' may be associated with anti-Ro antibody production."
Adenosine ; Alleles ; Autoantibodies ; Biological Assay ; DNA ; Guanine ; Homozygote ; Humans ; Lupus Erythematosus, Systemic* ; Polymorphism, Restriction Fragment Length ; Promoter Regions, Genetic ; Tumor Necrosis Factor-alpha*

No abstract available.