1.A Huge Left Ventricular Spontaneous Echo Contrast during Ventricular Fibrillation.
Il Suk SOHN ; Jong Chun PARK ; Kye Hun KIM ; Hyung Wook PARK
Journal of Cardiovascular Ultrasound 2006;14(1):36-37
No abstract available.
Echocardiography
;
Ventricular Fibrillation*
2.A Huge Left Ventricular Spontaneous Echo Contrast during Ventricular Fibrillation.
Il Suk SOHN ; Jong Chun PARK ; Kye Hun KIM ; Hyung Wook PARK
Journal of Cardiovascular Ultrasound 2006;14(1):36-37
No abstract available.
Echocardiography
;
Ventricular Fibrillation*
3.Near-Sudden Unexpected Death of Epilepsy (SUDEP) Caused by Ventricular Fibrillation Following Seizure in a Post-Stroke Epilepsy Patient.
Jong Wook SHIN ; In Chul BAEK ; Ji Eun OH ; Kyoung Jae LEE ; Hye Seon JEONG ; Jae Moon KIM
Journal of the Korean Neurological Association 2011;29(4):368-370
No abstract available.
Epilepsy
;
Humans
;
Seizures
;
Ventricular Fibrillation
5.The design of the external defibrillator using the truncated exponential biphasic waveform.
Er-mei SONG ; Xiao-mei WU ; Cui-wei YANG ; Zu-xiang FANG
Chinese Journal of Medical Instrumentation 2006;30(1):25-28
The external defibrillator is an emergency instrument used very widely in clinics. It plays an important role in rescuing ventricle fibrillation (VF) patients. We have designed an external defibrillator using the truncated exponential biphasic waveform. The system consists of three parts: the ECG collection module, the control module and the defibrillator module. They are introduced respectively, listing the main problems and the methods to solve them. Some experiments have been done and the corresponding results are given.
Animals
;
Defibrillators
;
Equipment Design
;
Swine
;
Ventricular Fibrillation
6.The Effects of Propranolol and MJ 1999 against Ventricular Fibrillation during Piofound Hypothermia in Dogs .
Korean Journal of Anesthesiology 1974;7(1):19-33
Profound hypothermia (15 degrees C) was induced in 36 mogrel dogs anesthetized with nitrous oxide-oxygen-d-tubocurarine. Prcpranolol (0.3 mg/kg) or MJ 1999(0.8 mg/kg) was administered intravenously 10 minutes prior to the start of cooling. Significant protection against ventricular fibrillation was provided by MJ 1999, but not by propranolol. With available evidence that hypothermia causes increased catecholamine release and that MJ 1999 is more specific in its beta-adrenergic blocking prcperties than propranolol, the authors findings suggest that ventricular fibrillation during hypothermia is due to beta-adrenergic hyperactivity.
Animals
;
Dogs*
;
Hypothermia*
;
Propranolol*
;
Sotalol*
;
Ventricular Fibrillation*
9.Dispersion of QT Interval and Other Repolarization Indexes in Acute Myocardial Infarction.
Hwee CHOI ; Tae Joon CHA ; Seon Mi PARK ; Jin KIM ; Hwan Jun CHOI ; Ho Dae YOO ; Seon Ja PARK ; Yang Soo KIM ; Seung Jae JOO ; Jae Woo LEE
Korean Circulation Journal 1997;27(12):1289-1297
BACKGROUND: It is known that QT dispersion represents asynchronous repolarization of ventricle which is related to ventricular fibrillation. The incidence of ventricular arrhythmia is increased after acute myocardial infarction. So this study compared QT dispersion and other repolarization indexes for detection of asynchronous repolarization in acute myocardial infarction. We also investigated which portion of repolarization is the key portion of the asynchrony. METHODS: In 37 acute myocardial infarction patients and 38 angina patients dispersion of QT, JT, JTpeak and QTpeak were measured. We also measured maximum adjacent dispersion of same parameters in precordial leads. In 20 survived patients and 17 dead patients after acute myocardial infarction were also compared. We also investigated correlation of PVC's on Holter monitoring with these repolarization parameters. RESULTS: 1) All ventricular repolarization indexes(QT, QTc, JT, JTpeak, QT peak and TpeakTend dispersion) were significantly increased in acute myocardial infarction group than compared with those of angina group(p<0.05). 2) Maximal precordial dispersion(QT, QTc, JT, JTpeak and QTpeak) were also significantly increased in acute myocardial infarction group than angina group(p<0.05). 3) Dead patient group after myocardial infarction showed significantly increased QTc and TpeskTend dispersion compared with those of survived patient group(p<0.05). 4) Multivariate linear correlation showed that TpeakTend dispersion and JT dispersion was correlated with QT dispersion. CONCLUSIONS: There were asynchronous myocardial repolarization changes in acute myocardial infarction. Our study demonstrated that T wave change was major determinant of dispersion of myocardial repolarization.
Arrhythmias, Cardiac
;
Electrocardiography, Ambulatory
;
Humans
;
Incidence
;
Myocardial Infarction*
;
Ventricular Fibrillation
10.J Wave Syndromes: History and Current Controversies.
Tong LIU ; Jifeng ZHENG ; Gan Xin YAN
Korean Circulation Journal 2016;46(5):601-609
The concept of J wave syndromes was first proposed in 2004 by Yan et al for a spectrum of electrocardiographic (ECG) manifestations of prominent J waves that are associated with a potential to predispose affected individuals to ventricular fibrillation (VF). Although the concept of J wave syndromes is widely used and accepted, there has been tremendous debate over the definition of J wave, its ionic and cellular basis and arrhythmogenic mechanism. In this review article, we attempted to discuss the history from which the concept of J wave syndromes (JWS) is evolved and current controversies in JWS.
Brugada Syndrome
;
Death, Sudden, Cardiac
;
Electrocardiography
;
Ventricular Fibrillation