Back ground: The hypertension may lead the changes of the left ventricular geometry and the left ventricular diastolic dysfunction. There were many investigations about these abnormalities. Moreover, the treatment of the hypertensive patients with or without the left ventricular diastolic dysfunction is quite different. In Binh dinh general hospital, however, the left ventricular dysfuntion were neglected in practice. Thus, in this study, we evaluate the prevalence of these abnormalities in hypertensive patients. Subjects and method: We prospectively studied, from 6/2002 to 11/2003, 200 consecutive hypertensive patients admitted to the Internal Deparment at the Binh Dinh General Hospital. Echocardiography was performed on a Hewlett-Packard Image-Points with 2.5 and 3.5 MHz transducers. Measurements of the size of the ventricular walls are according to ASE. Doppler echocardiography is used to evaluate the characteristics of diastolic transmitral blood flow from the apical four-chaber view. The peak velocities of blood flow during early diastolic filling (E wave) and atrial contraction (A wave), the E-wave decelerating time and parameters of pulmonary venous flow revesal during atrial contraction are measured. The isovolumic relaxation time is measured from the apical-five chamber view. The diagnosis and classification of the left ventricular geometric changes and the abnormal diastolic filling patterns are based on the criteria of Devereux. Results: Prevalence of the left ventricular diastolic dysfunction was 61.5% of the hypertensive patients with the most frequent impaired relaxation pattern (48%). 72.5% of patients have the geometric changes in which the remodelling type consists of 47.5% concentric hypertrophy 25%.
Ventricular Dysfunction, Left ; Hypertension

No abstract available.
Breast* ; Humans ; Ventricular Dysfunction, Left*

No abstract available.
Humans ; Thrombosis* ; Ventricular Dysfunction, Left*

Study on 66 men with type 2 diabetic at Military Hospital 108 and 103 healthy persons (with similar age, BIM, HATTh, HATTr, frequence of heart). Results: In type 2 diabetic group, A-C distance front of bicuspid valve extend, slash volume significant reducing compared with control group. Left autrial size, intraventricular septum wall thickness, ventricular wall thickness, left ventricular mass index (LVMI) significant changing compared with control group. Non THA diabetic group include 3 patients ventricular hypertrophy (LVMI > 131 g/m2), in control group hasn’t any patients. In VT Ia diabetic group, VTIe/VTIa rate reduce, DT time lengthen significantly compare with control group
Diabetes Mellitus, Type 2 ; Ventricular Dysfunction, Left ; Ventricular Function, Left

Investigate the correlation between the left ventricular diastolic functions and risk factor hypertension of 121 male type 2 diabetics admitted Hospital 103 from 2000 December to 2002 November. All patients were divided into 2 groups: 66 diabetics without hypertension, 55 diabetics with hypertension, and 103 healthy controls matched in age, gender, and BMI index. Results: getting diabetes will affect to left ventricular diastolic dysfunction. Combination of diabetes and hypertension will exacerbate left ventricular diastolic dysfunction
Diabetes Mellitus, Type 2 ; Ventricular Dysfunction ; Ventricular Dysfunction, Left

Electrocardiography ; Humans ; Takotsubo Cardiomyopathy ; Ventricular Dysfunction, Left

The volume-time curve change in patients with normal left ventricular (LV) diastolic function and diastolic dysfunction was evaluated by real-time three-dimensional echocardiography (RT3DE). LV diastolic dysfunction was defined by E'ventricular septal (IVS) of mitral annulus. In 24 patients with LV diastolic dysfunction, including 12 patients with delayed relaxation (delayed relaxation group) and 12 patients with pseudo-normal function (pseudo-normal group) and 24 normal volunteers (control group), data of full-volume image were acquired by real-time three-dimensional echocardiography and subjected to volume-time curve analysis. EDV (end-diastolic volume), ESV (end-systolic volume), LVEF (left ventricular ejection fraction), PER (peak ejection rate), PFR (peak filling rate) from RT3DE were examined in the three groups. Compared to the control group, PFR (diastolic filling index of RT3DE) was significantly reduced in the delayed relaxation group and pseudo-normal group (P<0.05). There were no significant differences in EDV, ESV, LVEF, PER (P>0.05). It is concluded that PFR, as a diastolic filling index of RT3DE, can reflect the early diastolic function and serve as a new non-invasive, quick and accurate tool for clinical assessment of LV diastolic function.
Diastole/physiology ; Echocardiography, Three-Dimensional/*methods ; Ventricular Dysfunction, Left/physiopathology ; Ventricular Dysfunction, Left/*ultrasonography ; Ventricular Function, Left/*physiology

Physicians prefer traditional right ventricular apical pacing to right ventricular outflow tract pacing because of easy accessibility, short procedure time, stable lead function and a low incidence of complications. However, right ventricular apical pacing produce an abnormal pattern of ventricular activation compared with right ventricular outflow tract pacing. There is growing evidence that right apical pacing may have long-term detrimental effects on left ventricular function. The report of Kim et al. in this issue showed that ventricular dyssynchrony was commonly seen in patients with permanent pacemaker implantation. They suggested the role of paced QRS duration to predict ventricular dyssynchrony during the follow-up period in patients with permanent pacemaker. However, a large scaled prospective observational study is needed to assess the factors influencing ventricular dyssynchrony in Korean patients with permanent pacemaker.
Follow-Up Studies ; Humans ; Incidence ; Ventricular Dysfunction ; Ventricular Function, Left

BACKGROUND: Circulating levels of N-terminal pro-brain natriuretic peptide (NT-proBNP) have been used to identify and monitor myocardial dysfunction in patients with various cardiac diseases. However, its clinical significance in patients with chronic renal failure (CRF) is uncertain because NT-proBNP clearance may be affected by renal function. METHODS: We studied 331 patients with CRF (eGFR <60 mL/min/1.73m2) admitted to Samsung Medical Center between March 2003 and March 2005, in whom both NT-proBNP measurement and echocardiography were performed within a week. RESULTS: Mean value of eGFR was 29+/-15 mL/ min/1.73m2 and median value of NT-proBNP was 6,971 pg/mL. NT-proBNP levels increased with declining eGFR (p<0.05). NT-proBNP levels were also elevated in patients with left ventricular hypertrophy (LVH), systolic dysfunction and diastolic dysfunction. In multiple regression analysis, ejection fraction (beta=-0.373, p>0.01) was strong independent correlate of NT-proBNP, eGFR (beta=-0.358, p<0.01), left ventricular mass index (beta=0.126, p>0.01) and diastolic dysfunction 2 or higher (beta=0.171, p< 0.05) were also independent correlates of NT-proBNP. Receiver-operating characteristic (ROC) analyses demonstrated NT-proBNP to be 75% sensitive and 76% specific for the detection of left ventricular systolic dysfunction, as indicated by area under the ROC curve of 0.78 (p<0.05), with NT-proBNP cutoff concentration of 25,000 pg/mL. CONCLUSION: Circulating NT-proBNP levels increased with declining renal function. However, its level were significantly correlated with LVH, systolic and diastolic dysfunction in patients with CRF. The measurement of NT-proBNP levels might be useful to predict left ventricular dysfunction in patients with CRF.
Echocardiography ; Heart Diseases ; Humans ; Hypertrophy, Left Ventricular ; Kidney Failure, Chronic* ; Plasma* ; ROC Curve ; Ventricular Dysfunction, Left ; Ventricular Function, Left*

A recently reported cardiac syndrome of transient left ventricular dysfunction, clinically resembles acute myocardial infarction and presents with chest pain, ECG changes and minimal elevation of cardiac enzymes in absence of myocardial ischemia or injury. The clinical presentation includes a wide range of symptoms and left ventricular function is normalized completely within days to weeks. This syndrome is likely a non-ischemic, metabolic-dependent syndrome caused by stress-induced activation of the cardiac adrenoreceptors. We report three cases of stress-induced transient LV dysfuction.
Cardiomyopathies* ; Chest Pain ; Electrocardiography ; Myocardial Infarction ; Myocardial Ischemia ; Ventricular Dysfunction, Left ; Ventricular Function, Left