1.What is the Prion?.
Journal of the Korean Medical Association 2001;44(5):464-474
No abstract available.
2.No title in English
Journal of the Korean Medical Association 1997;40(6):747-756
No abstract available.
3.Prevalence of Toxocara canis, Toxascaris leonina and Dirofilaria immitis in dogs in Chuncheon, Korea (2004) .
The Korean Journal of Parasitology 2005;43(2):65-67
The intestines and hearts of dogs were examined for Toxocara canis, Toxascaris leonina, and Dirofilaria immitis, after necropsy between June 26 and September 29, 2004 in Chuncheon, Korea. Of the 662 dogs examined, 6 were infected with T. canis (0.9%), 86 with T. leonina (13.0%). Fifty dogs were infected with D. immitis among 500 dogs examined (10.0%). Five were co-infected with T. canis and T. leonina, and three were co-infected with T. leonina and D. immitis. The cumulative positive infection rate for three species was 134/662 (20.2%). Considering previously reported seropositive rates of T. canis excretory-secretory antigen, i.e., 5% in the adult population in Korea, the possibility of toxocariasis caused by T. leonina should be reevaluated.
Animals
;
Dirofilaria immitis/*isolation & purification
;
Dirofilariasis/*epidemiology
;
Dog Diseases/*epidemiology
;
Dogs
;
Feces/parasitology
;
Heart/parasitology
;
Intestines/parasitology
;
Korea/epidemiology
;
Prevalence
;
Toxocara/*isolation & purification
;
Toxocara canis/isolation & purification
;
Toxocariasis/*epidemiology
4.Mad Cow Disease and New Variant Creutzfeldt Jacob Disease.
Journal of the Korean Academy of Family Medicine 2004;25(7):509-518
No abstract available.
Animals
;
Cattle
;
Creutzfeldt-Jakob Syndrome*
;
Encephalopathy, Bovine Spongiform*
5.Obstructive Colitis: A Clinicopathologic Analysis of 7 Cases.
Korean Journal of Pathology 1996;30(12):1116-1122
Obstructive colitis is an ulceroinflammatory lesion of the large intestine at a distance proximal to the obstructing or potentially obstructing lesion of any etiology. It has been suggested that a rise in the intraluminal pressure related to the obstruction results in a fall in the intramural blood flow with subsequent ischemic necrosis. We examined 7 cases of surgically resected obstructive colitis associated with distal constrictive adenocarcinoma of the large intestine. Two cases of the obstructive colitis were radiologically suspected prior to surgery either as a separate tumor mass or diverticulosis, and another case was suggested to be a tumor seeding by colonoscopy. Grossly, five cases presented with single or multiple, shallow, well-demarcated ulcerative lesions, and the remaining two were featured with a segmental ulcerative lesion the with pseudopolypoid area. The ulceroinflammatory lesions were separated from the distal obstructive carcinoma by a skipped zone of relatively normal mucosa measuring 4-43 cm. The proximal colon to the obstructive lesions were obviously dilated except in one case. Microscopically, mucosa and submucosa were replaced by granulation tissue, and showed inflammatory cell infiltration and fibrin exudates. Muscle coat often accompanied ischemic contraction. Fissuring was noted in one case. The recognition of these lesions prior to or during surgery is stressed based on the morphological features corresponding to an ischemic change in the proximal mucosa to the primary obstructive lesion.
Adenocarcinoma
6.Morphologic Comparisons of Focal Segmental Glomerulo-sclerosis between Human and 5/6 Nephrectomy Rat Model.
Yong Jin KIM ; Yong Sun KIM ; Chae Hong SUH
Korean Journal of Pathology 1997;31(2):100-111
This study was conducted to set up a common mechanism for varying phases of focal segmental glomerulosclerosis(FSGS) by comparing the morphological differences between human FSGS and changes in 5/6 renal ablation animal model, which has been accepted as experimental prototype for hyperfiltration theory as pathogenesis of FSGS. Both the human and the experimental rats showed very similar changes such as segmental glomerulosclerosis, vacuole formations or inclusion of small granules of podocytes, appearance of foamy cells in the capillary lumina, eosinophilic deposits along the mesangial area, and focal atrophy of tubules with associated interstitial fibrosis. The halo, frequently seen in human FSGS, is due to detachment of visceral epithelium from basement membrane, however, did not appear in the experimental rat specimen. On the other hand, the foamy cells and hyalinization were more frequently noted in the rat series and even involved the arterioles. The mesangial proliferation never appeared in the rat series occasionally found in human FSGS. In conclusion, the pathogenesis of FSGS cannot depend solely on the hyperfiltration theory of hemodynamic derangement, but has complex impairment of visceral epithelium and cells forming the constituents of basement membrane.
Animals
;
Arterioles
;
Atrophy
;
Basement Membrane
;
Biopsy
;
Capillaries
;
Eosinophils
;
Epithelium
;
Fibrosis
;
Hand
;
Hemodynamics
;
Humans*
;
Hyalin
;
Models, Animal*
;
Nephrectomy*
;
Podocytes
;
Rats*
;
Vacuoles
7.Morphologic Comparisons of Focal Segmental Glomerulo-sclerosis between Human and 5/6 Nephrectomy Rat Model.
Yong Jin KIM ; Yong Sun KIM ; Chae Hong SUH
Korean Journal of Pathology 1997;31(2):100-111
This study was conducted to set up a common mechanism for varying phases of focal segmental glomerulosclerosis(FSGS) by comparing the morphological differences between human FSGS and changes in 5/6 renal ablation animal model, which has been accepted as experimental prototype for hyperfiltration theory as pathogenesis of FSGS. Both the human and the experimental rats showed very similar changes such as segmental glomerulosclerosis, vacuole formations or inclusion of small granules of podocytes, appearance of foamy cells in the capillary lumina, eosinophilic deposits along the mesangial area, and focal atrophy of tubules with associated interstitial fibrosis. The halo, frequently seen in human FSGS, is due to detachment of visceral epithelium from basement membrane, however, did not appear in the experimental rat specimen. On the other hand, the foamy cells and hyalinization were more frequently noted in the rat series and even involved the arterioles. The mesangial proliferation never appeared in the rat series occasionally found in human FSGS. In conclusion, the pathogenesis of FSGS cannot depend solely on the hyperfiltration theory of hemodynamic derangement, but has complex impairment of visceral epithelium and cells forming the constituents of basement membrane.
Animals
;
Arterioles
;
Atrophy
;
Basement Membrane
;
Biopsy
;
Capillaries
;
Eosinophils
;
Epithelium
;
Fibrosis
;
Hand
;
Hemodynamics
;
Humans*
;
Hyalin
;
Models, Animal*
;
Nephrectomy*
;
Podocytes
;
Rats*
;
Vacuoles
8.Percutaneous transhepatic variceal obliteration
Yong Sun KIM ; Yong Joo KIM ; Duk Sik KANG
Journal of the Korean Radiological Society 1986;22(5):672-682
Pecutaneous transhepatic portogram with selective catheterizatio of the portal vein and its tributaries notonly provide information about the status of portal circulation and but can also be used in the treatment ofbleeding esophageal varices by selective embolization with various embolic materials. The authors describe easierand safer modified method of conventional percutaneous transhepatic portogram. We wish to describe our experiencewith the technical aspects and portosystemic collateral patterns in 26 patients with variceal bleeding, from July1985 to July 1986 at Kyungpook National University Hospital. 1. To overcome the difficulties of catheter passageand superselection of variceal supplying vein, we used 7F sheath directly introduced over the ,018″ guide wire.We used coaxial system using 25cm 18G needle within 7F vessel dilator to make the tip of dilator more rigid. 2.Variceal obliteration attempted in 23 patients who showed variceal supplying veins on the protogram, Successfulobilteration was obtained in 20 patients. We used absolute ethanol, stainless steel coil, and Gelfoam cubes withsclerosing and embolica agent of variceal vein. 3. Portosystemic collaterals of 24 patients; Coronary vein;21cases, Inferior mesenteric vein:9 cases, Short gastric vein:7 cases, Umbilical & paraumbilical vein; 6 cases,Gastrorenal: 3 cases, Splenorenal: 2 cases, Splenoretoperitoneal: 1 case. 4. The number of coronay vein were 18cases of single and 4 cases of two. The locations of coronary vein: Splenic vein: 13 cases, Main portal vein: 7cases, Junctional area: 6 cases. 5. Transhepatic obliteration of the gastroesophageal veins is a relatively simpleand usually successful form of palliative treatment for actively bleeding and stable gastroesophageal varices.
Catheters
;
Coronary Vessels
;
Esophageal and Gastric Varices
;
Ethanol
;
Gelatin Sponge, Absorbable
;
Gyeongsangbuk-do
;
Hemorrhage
;
Humans
;
Methods
;
Needles
;
Palliative Care
;
Portal Vein
;
Splenic Vein
;
Stainless Steel
;
Varicose Veins
;
Veins
9.Rasmussen Syndrome.
Journal of the Korean Child Neurology Society 2004;12(1):1-11
No abstract available.
Encephalitis*
10.Rasmussen Syndrome.
Journal of the Korean Child Neurology Society 2004;12(1):1-11
No abstract available.
Encephalitis*