No abstract available.
Child* ; Humans ; Status Epilepticus*

No abstract available.
Macrophages*

No abstract available.
Macrophages*

No abstract available.

No abstract available.
Colitis, Ulcerative* ; Ulcer*

This study was undertaken to determine whether verapamil protects renal function in rabbits with ischemic acute renal failure. Renal ischemia was induced by clamping bilateral renal arteries for 60 min. One group received intravenously an infusion of verapamil (lmg/kg) for 30 min prior to initiation of renal artery clamping and the other group received equal volume of saline. Renal blood flow was measured with flowmeter before (basal) and 24 hr after ischemia. Serum creatinine level increased 24 hr after ischemia and remained high to 72 hr. When verapamil was pretreated, the level 48 and 72 hr after ischemia was significantly decreased compared with saline insusion. Urine flow was markedly decreased 24 hr after ischemia and remained depressed to 72 hr, but it was significantly increased 72 hr after ischemia in verapa- mil-pretreatment animals as compared with the saline-infusion animals. GFR were markedly reduced 24 hr after ischemia and remained depressed to 72 hr, which was significantly prevented by verapamil pretreatment. Ischemia caused a significant increase in FEVa and a reduction in Uosm, and TcH2O, indicating impairment in urine concentrating ability of tubules, and the impairment was significantly attenuated by verapamil. The uptake of p-aminohippurate in cortical slices was depressed by ischemia, which was significantly prevented by verapamil pretreatment. In salineinfusion animals, renal blood flow was not significantly different between the basal value and that after 24 hr of reflow. Renal blood flow was not significantly altered by verapamil pretreatment. Anoxia/reoxygenation injury in the control renal slices was not significantly prevented by Ca channel blockers. These results suggest that verapamil exerts a protective effect in ichemic acute renal failure, and the beneficial effects may be attributed to effects other than vasodilation. These data also indicate that a reduction in GFR following ischemia does not result from change in renal blood flow.
Acute Kidney Injury* ; Animals ; Constriction ; Creatinine ; Flowmeters ; Ischemia ; Kidney Concentrating Ability ; p-Aminohippuric Acid ; Rabbits* ; Renal Artery ; Renal Circulation ; Vasodilation ; Verapamil*

No abstract available.
Drainage* ; Lung Abscess* ; Lung* ; Pneumothorax* ; Suppuration*

Anti-Bacterial Agents ; Copper ; Disinfection ; Ethylene Oxide ; Fibroblasts ; Freezing ; Micropore Filters ; Sterilization ; Succinate Dehydrogenase ; Transplants

Branchio-oto-renal(BOR) syndrome is an autosomal dominant disorder. The pathophysiology of this syndrome is unknown. BOR syndrome has a wide range of clinical manifestations affecting the branchial, auditory and renal systerns. Associated abnormalities of the face, lacrimal duct, palate and ureters have also been described. However, the major clinical findings associated and/ or ear pits, and renal anormaly. We experienced a case of a 15-day-old rnale newborn who had visited our hospital for deformed auricle and atresia of external auditory canal found at birth. We report this case with a review of the related literatures.
Branchial Region ; Branchio-Oto-Renal Syndrome* ; Ear ; Ear Canal ; Humans ; Infant, Newborn ; Palate ; Parturition ; Ureter

Induction of general anesthesia with tracheal intubation has several advantages but also causes some cardiovascular side effects. Several approaches have been tried to reduce these side effects. As an attempt to reduce elevated blood pressure and increased heart rate during tracheal intubation, we used thiopental 3 mg/kg with Thalamonal 0.05 mg/kg (experimental group II, 30 cases) as an induction of general anesthesia inatead of the conventional use of thiopental 6 mg/kg(control group I, 30 caaes). The results are as follows:1. Blood pressure and heart rate changes between the 2 gruops at postintubation 30 sec. were statistically significant; 194+/-24 torr, 111+/-15/min. in control gruop and 166+/-29 torr (p<0.01), 102+/-14/min. (p<0.05) in experimental group. 2. There were no significant changes in blood pressure and heart rate at pre-intubation and post-extubation period. 3. The incidence of tachycardia and arrhythmia were more frequent in the control group, while hypotension and severe respiratory depression in experimental group.
Anesthesia, General ; Arrhythmias, Cardiac ; Blood Pressure ; Heart Rate ; Hypotension ; Incidence ; Intubation* ; Respiratory Insufficiency ; Tachycardia ; Thiopental