No abstract available.
Animals ; Dopamine* ; Nucleus Accumbens* ; Panax* ; Rats* ; Saponins*

No abstract available.
Brain* ; Encephalitis, Herpes Simplex* ; Herpes Simplex* ; Technetium Tc 99m Exametazime* ; Tomography, Emission-Computed, Single-Photon*

No abstract available.
Brain* ; Encephalitis, Herpes Simplex* ; Herpes Simplex* ; Technetium Tc 99m Exametazime* ; Tomography, Emission-Computed, Single-Photon*

No abstract available.
Brain* ; Technetium Tc 99m Exametazime*

No abstract available.
Brain* ; Technetium Tc 99m Exametazime*

No abstract available.
Mucormycosis* ; Technetium Tc 99m Exametazime*

No abstract available.
Immunoglobulin G* ; Inflammation*

No abstract available.
Thyroid Gland* ; Thyroid Neoplasms*

Purpose: The purpose of this study was to evaluate whether three-dimensional (3D)-printed flexible denture resin has suitable mechanical properties for use as a thermoplastic denture base resin material. Materials and Methods: A total of 96 specimens were prepared using the 3D printed flexible denture resin (Flexible Denture). Specimens were designed in CAD software (Tinkercad) and printed through a digital light-processing 3D printer (Asiga MAX UV). Post-polymerization process was conducted according to air exposure or glycerin immersion at 35°C or 60°C and for 30 or 60 minutes. The maximum flexural strength, elastic modulus, 0.2% offset yield strength, and Vickers hardness of 3D-printed flexible denture resin were assessed.Result: The maximum flexural strength ranged from 64.46±2.03 to 84.25±4.32 MPa, the 0.2% offset yield strength ranged from 35.28±1.05 to 46.13±2.33 MPa, the elastic modulus ranged from 1,764.70±64.66 to 2,179.16±140.01 MPa, and the Vickers hardness ranged from 7.01±0.40 to 11.45±0.69 kg/mm2 . Conclusion Within the limits of the present study, the maximum flexural strength, 0.2% offset yield strength, elastic modulus, and Vickers hardness are sufficient for clinical use under the post-polymerization conditions of 60°C at 60 minutes with or without glycerin precipitation.

PURPOSE: This study examined the histological changes and apoptosis in contralateral rat testes after testicular torsion or the repair of torsion, and investigated the functional role of clusterin in the pathogenesis for the injury of the contralateral testes after testicular torsion. MATERIALS AND METHODS: H&E staining and TUNEL assay were performed in contralateral testes following torsion or repair of torsion. The mean numbers of spermatids per tubule and apoptotic germ cells per tubule were calculated in each group with respect to time. The expression of clusterin after torsion or the repair of torsion was observed by RT-PCR and Western blot analysis. RESULTS: Following the repair of torsion, significant germ cell injuries and abnormal spermatogenesis were observed in the contralateral testes compared to those without repair. The expressions of clusterin mRNA and protein after testicular torsion were no different from those in the control testes. However, in the contralateral testes, the clusterin mRNA and clusterin protein expressions in the contralateral testes were increased 8 hours after the repair of torsion. The appearance of apoptosis in the contralateral testes after the repair of torsion appeared to correlate with the clusterin protein expression. CONCLUSIONS: The injury to contralateral testes during testicular torsion seems to be caused by the apoptosis induced by reperfusion of ischemic torsioned testis. Clusterin appears to play an antiapoptotic role in the apoptotic process in the injury to the contralateral testis due to the repair of testicular torsion.
Animals ; Apoptosis* ; Blotting, Western ; Clusterin* ; Germ Cells ; In Situ Nick-End Labeling ; Rats ; Reperfusion ; RNA, Messenger ; Spermatic Cord Torsion* ; Spermatids ; Spermatogenesis ; Testis