No abstract available.
Adenocarcinoma* ; Cardia* ; Stomach*

No abstract available.
Burns* ; Humans

Massive ovarian deema is a tate conditian. It is a benign enlargement of the ovary caused by accumulation of fluid, which is thought to result from intermittent partial toraion of the ovarian pedicle. Histologically, the ovaries were characterized by diffuse edema of medulla and inner cortex. Two cases of massive ovanan edema are reported with brief review of the literatures.
Edema* ; Female ; Ovary

No abstract available.
Polydactyly*

No abstract available.
Animals ; Aorta, Thoracic* ; Rats*

No abstract available.

Craniodiaphyseal dysplasia is a rare hereditary bone dysplasia, transmitted as autosomal recessive trait. This disorder is defined by Gorlin in 1969 as one of a family of severe bone disorders called "Craniotubular bone dysplasia", which is charaterized by massive and generalized hyperostosis and sclerosis, especially involving the skull facial bones. The major clinical features include marked craniofacial changes associated with bony overgrowth such as an enlarged head circumference, cranial nerve palsies and severe facial distortion. We experienced a case of craniodiaphyseal dysplasia in 2 year-9 month-old female patient who presented with typical clinical manifestation and radiologic findings nearly identical to those described by Gorlin. This is the first description of this rare disease in the Korean literature. Thus, we report a case of craniodiaphyseal dysplasia with brief related literature.
Bone Diseases, Developmental ; Cranial Nerve Diseases ; Facial Bones ; Female ; Head ; Humans ; Hyperostosis ; Rare Diseases ; Sclerosis ; Skull

Craniodiaphyseal dysplasia is a rare hereditary bone dysplasia, transmitted as autosomal recessive trait. This disorder is defined by Gorlin in 1969 as one of a family of severe bone disorders called "Craniotubular bone dysplasia", which is charaterized by massive and generalized hyperostosis and sclerosis, especially involving the skull facial bones. The major clinical features include marked craniofacial changes associated with bony overgrowth such as an enlarged head circumference, cranial nerve palsies and severe facial distortion. We experienced a case of craniodiaphyseal dysplasia in 2 year-9 month-old female patient who presented with typical clinical manifestation and radiologic findings nearly identical to those described by Gorlin. This is the first description of this rare disease in the Korean literature. Thus, we report a case of craniodiaphyseal dysplasia with brief related literature.
Bone Diseases, Developmental ; Cranial Nerve Diseases ; Facial Bones ; Female ; Head ; Humans ; Hyperostosis ; Rare Diseases ; Sclerosis ; Skull

Thenar motor unit number estimation(MUNE) was performed in 49 normal subjects without known neuromuscular diseases using statistical method. The purposes of this study were to compare MUNE parameters between the dominant and non-dominant hands and to evaluate the changes according to age. Reproducibility was assessed in 20 subjects also. The results showed that thenar motor units number, maximum negative peak compound muscle action potential (CMAP) area and mean area of single motor unit potentials(SMUP) were not different between dominant and non-dominant sides, but maximum negative peak CMAP amplitude was larger in dominant than non-dominant side. With aging, the motor unit number, maximum negative peak CMAP amplitude and area decreased and the mean area of single motor unit potentials increased. There was a high reproducibility between the test and the retest values for motor unit number, maximum negative peak CMAP amplitude and area as well as mean area of single motor unit potentials. The statistical estimate of the motor unit number is a reliable method and easily available in clinical settings. The results of this study are expected to be used as a baseline data for the future.
Action Potentials ; Aging ; Hand ; Muscles* ; Neuromuscular Diseases

Around the time of recovery from general anesthesia we usually use atropine and neostigmine to counteract the effect of muscle relaxant that is remaining in the body. We used various kinds of combinations of atropine and neostigmine to see the effect of them on heart rate. The combinations were as follows: I) atropine 0.02mg/kg+neostigmine 0. 02mg/kg. 2) atropine 0. 2mg /kg+neostigmine 0.03mg/kg. 3) atropine 0. 02mg/kg+neostigmine 0.04mg/kg. 4) atropine 0.02mg/kg+neostigmine 0.05mg/kg. 5) atropine 0.02mg/kg+neostigmine 0.06mg/kg. 6) atropine 0.02mg/kg+neostigmine 0.07mg/kg. Ten subjects(ps.I.ASA) were taken for each combination. Thus total sample size of the six combinations were sixty, Each study has been performed in the state of ASA P.S.1, and anesthetized for b but two hours. N2O-O2-Halothane technique was used for anesthesia. The dosage of pancuronium was 0. 08mg/kg. The age distribution of the sample was 2 to 60, and male to female ratio was 4 to 6. We came to the following conclusions from this study. 1) Types of dosage combinations those caused little change in heart rate were; atrop!ne 0.02mg/kg+neostigmine 0.02mg/kg, and atropine 0.02mg/kg+ neostigmine 0.03mg/kg. 2) Just after the simultaneous injection of the drugs, there appeared mild tachycardia, but fifteen minutes after the administration severe bradycardia came on and, after that the heart rate returned to normal. 3) The combinations which included neostigmine, more than 0. 04mg/kg, caused severe braycardia, and the most severe bradycardia was seen with a combination of atropine 0.02mg/kg and neostigmine 0.06mg/kg.
Age Distribution ; Anesthesia ; Anesthesia, General ; Atropine* ; Bradycardia ; Female ; Heart Rate* ; Heart* ; Humans ; Male ; Neostigmine* ; Neuromuscular Blockade* ; Pancuronium ; Sample Size ; Tachycardia