1.Ischemic Reperfusion Injury and Anesthetics.
Korean Journal of Anesthesiology 2007;52(5):499-510
No Abstract available.
Anesthetics*
;
Reperfusion Injury*
;
Reperfusion*
2.Volatile anesthetics and ischemia-reperfusion injury.
Korean Journal of Anesthesiology 2015;68(3):211-212
No abstract available.
Anesthetics*
;
Reperfusion Injury*
4.Intracellular Calcium Alterations Induced during Reperfusion Injury are Altered by Local Anesthetics and Hypothermia.
Myung Hee KIM ; Yu Hong KIM ; Taehyung HAN ; Baekhyo SHIN
Korean Journal of Anesthesiology 1996;30(4):392-400
No abstract available.
Anesthetics, Local*
;
Calcium*
;
Hypothermia*
;
Reperfusion Injury*
;
Reperfusion*
5.Effects of preconditioning against reperfusion injury in rat skeletal muscle.
Kyu Jeong HAN ; Chang Sub UHM ; Dong Ho KIM ; Young Suk SUH
Korean Journal of Anatomy 1993;26(2):199-213
No abstract available.
Animals
;
Muscle, Skeletal*
;
Rats*
;
Reperfusion Injury*
;
Reperfusion*
7.The Influence of Propofol Concentration on the Generation of Reactive Oxygen Species after Reoxygenation in Rat Embryonic Heart H9c2 Cell.
Yun Hong KIM ; Hyun Soo KIM ; Young Jae YI ; Won Joon CHOI ; Jun Kyu SONG ; Seon Min LEE ; Won Chae CHOE ; Sung Soo KIM
Korean Journal of Anesthesiology 2006;51(1):89-93
BACKGROUND: Reoxygenation of an ischemic heart causes a decrease in the cardiac function, which is known as reperfusion injury that is associated with an increase in the concentration of reactive oxygen species (ROS). This study examined the effect of the propofol concentration on the generation of ROS during reoxygenation in rat embryonic heart H9c2 cells. METHODS: Cultured H9c2 cells were examined in the following sequences: Prehypoxic, Hypoxic and Reoxygenation period. Each period required 60 minutes. The cells were exposed to propofol at the beginning of the prehypoxic period. Thirty minutes later, DCFH-DA (dichlorofluorescin diacetate) 10 micrometer was added to detect the ROS. The propofol concentrations used were 0, 5, 25, 50, 250 micrometer in the first experiment and 0, 1, 2, 3, 4, 5 micrometer in the second experiment. The ROS level was estimated using a fluorometer at 5-minute intervals from 5 to 60 minutes after reoxygenation. RESULTS: When the propofol concentrations was > 5 micrometer, the ROS levels were significantly lower than those of the untreated group (P0) (P < 0.05). At propofol concentrations < 5 micrometer, the ROS levels 35 to 60 minutes after reoxygenation were significantly lower that in the untreated group (P < 0.05). Between 5 and 30 minutes after reoxygenation, the cells exposed to 1, 4 and 5 micrometer propofol also showed lower ROS levels than the untreated group P0. However, 2 and 3 micrometer propofol did not show any significant difference in ROS values to those observed in the untreated group except for 2 micrometer at 25 minutes after reoxygenation. CONCLUSIONS: During the reoxygenation period in H9c2 cells, propofol concentrations > 5 micrometer inhibited ROS production over the whole period, and even 1micrometer showed some inhibition of ROS.
Animals
;
Heart*
;
Propofol*
;
Rats*
;
Reactive Oxygen Species*
;
Reperfusion
;
Reperfusion Injury
8.Reperfusion Injury after Autologous Cranioplasty in a Patient with Sinking Skin Flap Syndrome.
Sae Min KWON ; Jin Hwan CHEONG ; Jae Min KIM ; Choong Hyun KIM
Journal of Korean Neurosurgical Society 2012;51(2):117-119
The sinking skin flap syndrome is a rare complication after a large craniectomy. It consists of a sunken skin above the bone defect with neurological symptoms such as severe headache, mental changes, focal deficits, or seizures. In patient with sinking skin flap syndrome, cerebral blood flow and cerebral metabolism are decreased by sinking skin flap syndrome, and it may cause the deterioration of autoregulation of brain. We report a case of a patient with sinking skin flap syndrome who suffered from reperfusion injury after cranioplasty with review of pertinent literature.
Brain
;
Headache
;
Homeostasis
;
Humans
;
Reperfusion
;
Reperfusion Injury
;
Seizures
;
Skin
9.Effects of verapamil and allopurinol on ischemia-reperfusion injury in rat liver.
Young Hyun LEE ; Jae Dong CHEON ; Joo Seop KIM ; Gu KANG ; Soo Tong PAI
Journal of the Korean Surgical Society 1993;45(3):307-313
No abstract available.
Allopurinol*
;
Animals
;
Liver*
;
Rats*
;
Reperfusion Injury*
;
Verapamil*