We experienced three young patients with diffuse lymphoid hyperplasia of the gastric antrum. The peculiar nodularity of the stomach in tliese patients is another cause of etat mammelanne in addition to hypertrophic gastritis and prominence of the areae gastricae. The etiology of the diffuse lymphoid hyperplasia of the gastric antrum is not known. The symptqm is not specific and not related to hitologic and gastroscopic finding but rather to psychological disturbance. We propose longstanding observation for the change of the nodularity.
Gastritis, Hypertrophic ; Humans ; Hyperplasia ; Pyloric Antrum* ; Stomach

Gastritis cystica profunda is an uncommon benign mass that usually occurs on the gastric side of the site of a gastroenterostomy, but has also been known to develop in which has not been operated on. We report the case of stomach a 51-years-old man with pathologically proven gastritis cystica profunda. This patient had not undergone gastric surgery and CT showed a well-defined, 3 cm sized, cystic mass at the gastric antrum.
Gastritis* ; Gastroenterostomy ; Humans ; Pyloric Antrum ; Stomach

Gastric antral vascular ectasia ("watermelon stomach") is characterized by a prominent longitudinalerythematous fold of gastric antrum. Because it has usually been diagnosed by gastroscopy and biopsy, itsradiologic findings have not been well described. We report a case of gastric antral vascular ectasia, anddescribe its findings, as seen on UGIS, ultrasonography, and computed tomography.
Biopsy ; Gastric Antral Vascular Ectasia* ; Gastroscopy ; Pyloric Antrum ; Stomach ; Ultrasonography

Gastroduodenal intussusception is a rare condition and an invagination of a part of the gastric wall through the pyloric canal and into the duodenum. Gastroduodenal intussusception is always caused by a mobile gastric tumor, usually benign. However, gastroduodenal intussusception which was caused by gastric submucosal hemangiomatosis is not documented. We have maneged a case of gastric submucosal tumor leading to gastroduodenal intussusception in 2 years and 10 months old boy. He had a 10 x 5 x 3 cm sized submucosal tumor in posterior wall of gastric antrum. Laparotomy, manual reduction of intussusecption, and wedge resection of posterior gastric wall including the submucosal tumor were performed. Pathologic diagnosis showed a submucosal hemangiomatosis. We report a case of gastric submucosal tumor leading to gastroduodenal intussusception, and review the literature.
Diagnosis ; Duodenum ; Humans ; Infant ; Intussusception* ; Laparotomy ; Male ; Pyloric Antrum

Gastroduodenal fistula or double pylorus is a very rare condition. It is a fistulous communication between gastric antrum and duodenal bulb. It can be either congenital or acquired. In most cases it is thought to be a complication of gastric ulcer. We recently experienced a case of gastroduodenal fistula in 70 year-old man presenting as epigastric pain. He was diagnosed with non ST elevation myocardial infarction previously, and was taking aspirin. Gastroduodenal communication was revealed by endoscopy. We report a case of gastroduodenal fistula that developed in man who was taking aspirin, with review of the literature.
Aspirin ; Endoscopy ; Fistula ; Myocardial Infarction ; Pyloric Antrum ; Pylorus ; Stomach Ulcer

Double pylorus is a rare disease. It is described by a double communication between the gastric antrum and the duodenal bulb. The cause of double pylorus is either a congenital abnormality or an acquired condition. It is believed to be mostly a complication of peptic ulcer disease. Most reports revealed conditions only for the gastric antrum. However, case reports describing the involvement of the gastric body are extremely rare. Herein, we report a case of a double pylorus that developed on the gastric body and we present a review of the literature.
Congenital Abnormalities ; Peptic Ulcer ; Pyloric Antrum ; Pylorus ; Rare Diseases

Since the first case of gastric serrated adenoma found in 2001, 35 additional cases have been reported. Among these cases, 26 cases were associated with invasive adenocarcinoma within the serrated adenoma. Gastric serrated adenoma when compared with traditional adenoma has close correlation with invasive carcinoma. Serrated colorectal polyps are classified as hyperplastic polyps, sessile serrated adenoma/polyps, and tranditional serrated adenoma (TSA) depending on histological features. Two distinct phenotypes of TSA in the colon and rectum are reported. Those are unlocked serrated crypts (US-TSA) and ectopic crypt formations (ECFs). All gastric serrated adenoma are TSA in historical aspect and ECFs on phenotype. Whereas gastric adenomas are reported with high frequency in the antrum, gastric serrated adenomas are founded in the body and cardia. We report a case of a 60-year-old woman receiving endoscopic submucosal dissection for gastric serrated adenoma with adenocarcinoma discovered during routine screening.
Adenocarcinoma* ; Adenoma* ; Cardia ; Colon ; Female ; Humans ; Mass Screening ; Middle Aged ; Phenotype ; Polyps ; Pyloric Antrum ; Rectum ; Stomach*

Gastric lipoma is a typical benign submucosal tumor that is usually asymptomatic and is generally detected incidentally when performing gastroscopy. However, depending on its size and location, an atypical gastrointestinal lipoma can cause abdominal pain, diarrhea, constipation, intestinal obstruction, intussuception and life-threatening gastrointestinal bleeding. We report herein a case of gastric lipoma with bleeding in a 43-year-old man. The gastroscopy showed a 4x4 cm ulcero-fungating submucosal mass at the anterior wall of the gastric antrum. Laparoscopic gastric wedge resection was performed and the lesion was diagnosed as gastric lipoma.
Abdominal Pain ; Adult ; Constipation ; Diarrhea ; Gastrointestinal Hemorrhage ; Gastroscopy ; Hemorrhage* ; Humans ; Intestinal Obstruction ; Lipoma* ; Pyloric Antrum

PURPOSE: To demonstrate the two-phase spiral CT features of pseudo-wall thickening and tumor in the gastricantrum, and to evaluate the possibility of differential diagnosis by analyzing two-phase spiral CT scans. MATERIALS AND METHODS: We retrospectively reviewed 120 cases in which two-phase spiral CT scans showed focal wallthickening in the prepyloric antrum of the stomach. Our series included 60 cases of gastric cancer (includingseven of early gastric cancer) and 60 cases of normal prepyloric antrum. All patients underwent two-phase spiralCT and upper gastrointestinal series (n=83) and/or gastric endoscopy (n=80). All cancer cases were confirmed bybiopsy (n=60). We evaluated the differential points between gastric cancer and pseudo-wall thickening of thegastric antrum. RESULTS: The mean thickness of the antral wall was 19.0mm in the cancer group and 12.5mm in thenormal group. Thirty-one cases (51.7%) in the cancer group and 51 (85.0%) in the normal group showed concentricwall thickening, while in each group, the remainder showed eccentric wall thickening. The common enhancementpatterns of thickened wall in the cancer group were 1) a thick enhanced mucosal layer during the arterial phase,with diffusely enhanced whole wall thickness during the venous phase (n=21); 2) a thick enhanced mucosal layerduring the arterial phase, with thicker and more intense enhancement of the same area during the venous phase(n=18). In the normal group, the common enhancement pattern was a thin enhanced mucosal layer during both thearterial and venous phase (n=34). In the cancer group, the common associated findings were regionallymphadenopathy (n=43) and food remnants in the stomach (n=15), and in the normal group, intraluminal normalmucosal folds in the thickened segment (n=50). The findings of food remnants despite overnight fasting andintraluminal normal folds occurred only in the cancer and normal group, respectively. CONCLUSION: Pseudo-wallthickening frequently showed thin enhancement of the mucosal layer on both the arterial and venous phases oftwo-phase spiral CT scan; a tumor frequently showed a thick enhanced mucosal layer during the arterial phase, withdiffusely enhanced whole-wall thickness during the venous phase or a thick enhanced mucosal layer during thearterial phase, with thicker and more intense enhancement of the same area during the venous phase. The finding ofthin and homogeneously enhanced intraluminal normal mucosal folds in the thickened segment strongly suggested thatthe lesion was a pseudo-lesion.
Diagnosis, Differential ; Endoscopy ; Fasting ; Humans ; Pyloric Antrum* ; Retrospective Studies ; Stomach ; Stomach Neoplasms ; Tomography, Spiral Computed

BACKGROUND/AIMS: Gastric cancer is one of the most common malignant disease worldwide, particularly in Korean populations, but its etiology is not well-established. The infection with Helicobacter pylori (H. pylori) has been implicated in the pathogenesis of gastroduodenal diseases and recent studies have focused on whether specific H. pylori strains are associated with gastric carcinogenesis. The aim of this study was to investigate whether VacA seropositivity is associated with increased risk of gastric cancer in Korean populations. METHODS: Gastroduodenoscopy was done in patients with upper gastrointestinal complaints. During the examination, rapid urease test for the presence of H. pylori infection was done using gastric biopsy specimens taken from normal gastric antrum. Sera were collected from 20 chronic gastritis and 23 gastric cancer patients with H. pylori infection. Western blotting was carried out using a commercially prepared kit-Helicoblot 2.0. RESULTS: VacA seropositivity was 35% (8/23) in patients with gastric cancer and 55% (11/20) in patients with chronic gastritis (p=0.15). According to Lauren classification of gastric cancer, the numbers of patients with the intestinal and diffuse type were 12 and 11, respectively. VacA seropositivity was 25% (3/12) in patients with the intestinal type and 45.4% (5/11) in patients with the diffuse type (p=0.30). CONCLUSIONS: The infection with VacA positive H. pylori strain is not associated with increasing risk of gastric cancer in Korean populations.
Biopsy ; Blotting, Western ; Carcinogenesis ; Classification ; Gastritis ; Helicobacter pylori* ; Helicobacter* ; Humans ; Pyloric Antrum ; Stomach Neoplasms* ; Urease