No abstract available.
Polyhydramnios*

No abstract available.
Hemangioma* ; Polyhydramnios*

No abstract available.
Indomethacin* ; Polyhydramnios*

No abstract available.
Humans ; Infant* ; Mothers* ; Polyhydramnios

Pena-Shokeir syndrome is a rare, often lethal disease, characterized by intrauterine growth retardation, craniofacial anomalies, limb ankylosis, polyhydramnios and pulmonary hypoplasia. This autosomal recessive disease should be differentiated from trisomy 18, which the second most common multiple congenital malformation syndrome. It is therefore clear that the two syndromes have certain features in common, the most consistent being craniofacial and limb abnormalities and intrathoracic pathology. Therefore, final diagnosis should be based on chromosome study. The case that we experienced had typical Pena-Shokeir phenotype, but chromosomal study show 47, XY, +18.
Ankylosis ; Diagnosis ; Extremities ; Fetal Growth Retardation ; Pathology ; Phenotype* ; Polyhydramnios ; Trisomy*

A case of esophageal atresia associated with malrotation and segmental dilatation of the ileum is described. To our knowledge, this is the first such report of this combination of features. The prenatal sonogram showed polyhydramnios. Esophageal atresia was diagnosed 3 days after birth. The diagnosis of malrotation and segmental dilatation of the ileum was made using a barium enema and an explolaparotomy 10 days after the first operation. The facts indicate that abnormal findings on a prenatal sonography and esophageal atresia may be associated with many congenital anomalies should be considered.
Barium ; Diagnosis ; Dilatation* ; Enema ; Esophageal Atresia* ; Ileum* ; Parturition ; Polyhydramnios

Amniotic fluid plays an important role in fetal development and growth. Amniotic fluid volume should be maintained adequately throughout gestational periods. Polyhydramnios is a relatively common obstetrical problem, but is associated with poor perinatal outcome both in mother and fetus. For the proper management of polyhydramnios, it is important to find out accurate causes, but it is not easy even if obstetricians perform thorough prenatal assessments. It is also important to understand the regulation of amniotic fluid volume. Based on these backgrounds, this article will briefly review mechanisms of the regulation of amniotic fluid, and will also review diagnosis, etiologies, managements and prognosis of polyhydramnios.
Amniotic Fluid ; Female ; Fetal Development ; Fetus ; Humans ; Mothers ; Polyhydramnios ; Prognosis

We report a case of a premature newborn baby who presented with hyponatremia, hyperkalemia, and metabolic acidosis accompanied by severe polyhydramnios in utero. The baby was diagnosed with pseudohypoaldosteronism on the basis of normal 17-hydroxyprogesterone levels, elevated aldosterone, and clinical symptoms. His serum electrolyte levels were corrected with sodium chloride supplementation. Sodium supplementation was reduced gradually and discontinued at 5 months of age. At 5 months, the child was able to maintain normal serum electrolyte levels without oral sodium chloride supplementation, and showed normal physical and neurological development. This case illustrates that pseudohypoaldosteronism must be considered if a newborn infant with an antenatal history of severe polyhydramnios shows excessive salt loss with normal levels of 17-hydroxyprogesterone.
17-alpha-Hydroxyprogesterone ; Acidosis ; Aldosterone ; Child ; Humans ; Hyperkalemia ; Hyponatremia ; Infant, Newborn ; Polyhydramnios ; Pseudohypoaldosteronism ; Sodium ; Sodium Chloride

Twin-twin transfusion syndrome is attributed to an unbalanced blood flow between the donor and the recipient twin, but the exact pathophysiology remains incompletely understood. Despite active prenatal management, fetal twin-twin transfusion syndrome is still associated with a substantial residual perinatal mortality and morbidity. The donor twin progressively becomes anemic, growth restricted, oliguric and has oligohydramnios, where as the recipient becomes plethoric, polyuric and has polyhydramnios and in the most severe cases develops cardiomegaly, congestive heart failure and hypertension. We report a case of severe systemic hypertension developed in recipient twin.
Cardiomegaly ; Female ; Heart Failure ; Humans ; Hypertension* ; Oligohydramnios ; Perinatal Mortality ; Polyhydramnios ; Pregnancy ; Tissue Donors

Acardiac twinning affects 1 in 100 monozygotic twin pregnancies and 1 in 35,000 pregnancies overall. This condition is characterized by the absence or rudimentary development of fetal heart, and associated with various anomaly. The presence of an acardiac twin requires the normal (or "pump") twin to provide circulation for itself, as well as the acardiac sibling. The acardiac malformations are uniformly fatal in the affected twin, and mortality in the co-twin is as high as 55%. The principal perinatal problems associated with acardiac twinning are pump-twin congestive heart failure, maternal hydramnios, and preterm delivery. We recently experienced a case of acardius anceps associated with a normal male infant, so present with a brief review of the literature.
Fetal Heart ; Heart Failure ; Humans ; Infant ; Male ; Mortality ; Polyhydramnios ; Pregnancy ; Siblings ; Twins, Monozygotic