1.Alterations of Neuropeptides and Neutrotrophic Factors in Kindled Seizures.
Journal of Korean Epilepsy Society 2000;4(2):147-149
No abstract available.
Neuropeptides*
;
Seizures*
2.The Role of Neuropeptides in the Pathogenesis of Asthma.
Pediatric Allergy and Respiratory Disease 1999;9(3):243-251
No Abstracts Available.
Asthma*
;
Neuropeptides*
3.Neuropeptide Effects of Hematopoiesis.
Korean Journal of Pediatric Hematology-Oncology 1998;5(2):235-239
No abstract available.
Hematopoiesis*
;
Neuropeptides*
4.Neuropeptide S Receptor Induces Neuropeptide Expression and Associates With Intermediate Phenotypes of Functional Gastrointestinal Disorders (Gastroenterology 2010;138:98-107).
Journal of Neurogastroenterology and Motility 2010;16(2):218-220
No abstract available.
Gastrointestinal Diseases
;
Neuropeptides
;
Phenotype
5.Effects of neuropeptide Y on the motility of rabbit uterine strip.
Hee Sug RYU ; Kyung Eun LEE ; Young Soo AHN
Korean Journal of Obstetrics and Gynecology 1991;34(4):476-487
No abstract available.
Neuropeptide Y*
;
Neuropeptides*
6.Pituitary Hormones and Neuropeptides.
Korean Journal of Medicine 1998;55(4):503-511
No abstract available.
Neuropeptides*
;
Pituitary Hormones*
7.Pituitary Hormones and Neuropeptides.
Korean Journal of Medicine 1998;55(4):503-511
No abstract available.
Neuropeptides*
;
Pituitary Hormones*
8.Neuropeptide Y(NPY)-immunoreactive neuronal changes in senile dementia of alzheimer type(SDAT).
Korean Journal of Physical Anthropology 1993;6(2):249-261
No abstract available.
Alzheimer Disease*
;
Neurons*
;
Neuropeptides*
9.Distribution of sensory and autonomic neuropeptides in nasal mucosaof the hamsters.
Jeung Gweon LEE ; Joo Heon YOON ; In Yong PARK ; Kee Hyun PARK
Korean Journal of Otolaryngology - Head and Neck Surgery 1992;35(4):505-511
No abstract available.
Animals
;
Cricetinae*
;
Neuropeptides*
10.Changes in neuromedin S and its receptor after traumatic brain injury in cycling rats
Mohammad Khaksari ; Fatemeh Maghool ; Gholamreza Asadikaram ; Vida Naderi
Neurology Asia 2015;20(4):375-384
Animal studies indicate that gonadal steroids have prominent neuroprotective effects in several models
of experimental traumatic brain injury (TBI). Neuromedin U (NMU) and neuromedin S (NMS) are
regulatory peptides involved in inflammatory and stress responses, and modulation of the gonadotropic
axis. Since steroid hormone levels change during the estrous cycle, we sought to determine whether
variations in ovarian hormones would affect blood-brain barrier (BBB) permeability and brain levels
of NMS, NMU, and neuromedin S receptor 2 in experimental TBI. Two groups (proestrus and nonproestrus)
of female rats underwent diffuse TBI. At 24 hrs after TBI, results showed a significantly
decrease in BBB permeability in traumatic-proestrus animals (TBI-P) in comparison to traumatic nonproestrus
(TBI-NP) rats. Western blot analyzes demonstrated an enhanced expression of prepro-NMS
in TBI-P compared with that in the TBI-NP group. Likewise, TBI-P rats exhibited significantly higher
NMUR2 gene expression compared with those of TBI-NP, whereas no significant difference in brain
NMU content was seen between sham and traumatic animals. Our findings indicate that diffuse TBI
induces an increase in prepro-NMS and neuromedin S receptor 2 expression in traumatic-proestrus
rats which may mediate the anti-edematous effects of gonadal hormones in proestrus rats following
trauma.
Neuropeptides
;
Receptors, Neuropeptide
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