Background: Fecal incontinence (FI) results from damage to the external anal sphincter (EAS), significantly affecting quality of life. This clinical trial evaluated the impact of low-level laser (LLL) therapy on EAS repair and the treatment of FI. Methods: Thirty FI patients with EAS deficiency were divided into two groups (n = 15): a control group receiving sphincteroplasty alone and a laser group undergoing sphincteroplasty plus laser therapy. Following surgery, the laser group received daily laser therapy for 2 weeks. Outcomes were assessed using Wexner scores, electromyography (EMG), and endorectal sonography. Results: The laser group exhibited a significant increase in muscle bulk (P = 0.008) and a lower Wexner index (P < 0.0001) compared to the control group. EMG confirmed muscle contractility in the laser group. Conclusions Two weeks of LLL therapy effectively increased muscle at the EAS injury site, leading to significant, lasting improvements in FI.

Background: Fecal incontinence (FI) results from damage to the external anal sphincter (EAS), significantly affecting quality of life. This clinical trial evaluated the impact of low-level laser (LLL) therapy on EAS repair and the treatment of FI. Methods: Thirty FI patients with EAS deficiency were divided into two groups (n = 15): a control group receiving sphincteroplasty alone and a laser group undergoing sphincteroplasty plus laser therapy. Following surgery, the laser group received daily laser therapy for 2 weeks. Outcomes were assessed using Wexner scores, electromyography (EMG), and endorectal sonography. Results: The laser group exhibited a significant increase in muscle bulk (P = 0.008) and a lower Wexner index (P < 0.0001) compared to the control group. EMG confirmed muscle contractility in the laser group. Conclusions Two weeks of LLL therapy effectively increased muscle at the EAS injury site, leading to significant, lasting improvements in FI.

Background: Fecal incontinence (FI) results from damage to the external anal sphincter (EAS), significantly affecting quality of life. This clinical trial evaluated the impact of low-level laser (LLL) therapy on EAS repair and the treatment of FI. Methods: Thirty FI patients with EAS deficiency were divided into two groups (n = 15): a control group receiving sphincteroplasty alone and a laser group undergoing sphincteroplasty plus laser therapy. Following surgery, the laser group received daily laser therapy for 2 weeks. Outcomes were assessed using Wexner scores, electromyography (EMG), and endorectal sonography. Results: The laser group exhibited a significant increase in muscle bulk (P = 0.008) and a lower Wexner index (P < 0.0001) compared to the control group. EMG confirmed muscle contractility in the laser group. Conclusions Two weeks of LLL therapy effectively increased muscle at the EAS injury site, leading to significant, lasting improvements in FI.