We measured apparent accommodation in 40 contact lens wearing monocular aphakic eyes (40 patients). The mean apparent accommodation was 1.74 +/- 0.73 diopters. The apparent accommodation was not influenced by the target size. There was no primary correlation between apparent accommodation and reciprocal of pupillary diameter, but significant difference between apparent accommodation of small pupil group(or=7.0mm). It suggests that apparent accommodation is influenced by the pupil size in aphakic eyes. Accommodation of phakic eyes was significantly greater than apparent accommodation of contralateral aphakic eyes in the group of age between 45 and 55.
Miosis ; Pupil

BACKGROUND: Horner syndrome (HS), also known as Claude-Bernard-Horner syndrome or oculosympathetic palsy, comprises ipsilateral ptosis, miosis, and facial anhidrosis. CASE REPORT: We report herein the case of a 67-year-old man who presented with congenital HS associated with ipsilateral hypoplasia of the internal carotid artery (ICA), as revealed by heterochromia iridis and confirmed by computed tomography (CT). CONCLUSIONS: CT evaluation of the skull base is essential to establish this diagnosis and distinguish aplasia from agenesis/hypoplasia (by the absence or hypoplasia of the carotid canal) or from acquired ICA obstruction as demonstrated by angiographic CT.
Aged ; Carotid Artery, Internal* ; Diagnosis ; Horner Syndrome* ; Humans ; Hypohidrosis ; Miosis ; Paralysis ; Skull Base

Raeder's syndrome, or Raeder's paratrigeminal syndrome, is a painful Horner's syndrome characterized by unilateral head pain, oculosympathetic paralysis (miosis, ptosis) and anhydrosis over the forehead with otherwise normal facial sweating. We report two cases of Raeder's syndrome whose cause had not been found despite of intensive investigation and one case associated with nasopharyngeal tumor. The first case had a headache, miosis, ptosis but had not cranial nerve palsy. The second case had a unilateral facial headache, oculosympathetic paralysis, aoydrosis of forehead and sensory change in the ipsilateral ophthalmic division of the trigeminal nerve. The third case had a headache, miosis, ptosis, anhydrosis of forehead and sensory change in the whole territory of the trigeminal nerve.
Cranial Nerve Diseases ; Forehead ; Headache ; Horner Syndrome ; Miosis ; Paralysis ; Sweat ; Sweating ; Trigeminal Nerve

Pericarotid syndrome is the combination of a postganglionic Horner's syndrome and ipsilateral head and facial pain, which is caused by diverse pathologic processes in and around the internal carotid artery. We report a case of peri-carotid syndrome which presented Horner's syndrome and ipsilateral periodic severe hemicrania associated with malig-nant lymphma lapping internal carotid artery. After surgical removal of the mass and chemotherapy, miosis, ptosis, and ipsilateral hemicrania improved.
Carotid Artery, Internal ; Drug Therapy ; Facial Pain ; Head ; Headache ; Horner Syndrome ; Lymphoma* ; Miosis ; Pathologic Processes

BACKGROUND: Horner's syndrome, including ptosis and miosis, is a sign of success following stellate ganglion block (SGB). However, it is not sufficient to ensure adequate sympathetic block to the upper extremity. There are numerous recommended local anesthetic volumes for SGB. The aim of this study was to evaluate the effects of differing volumes used in SGB on the elevation of skin temperature of ipsilateral upper extremity, and to find the volume required to elevate skin temperature most frequently. METHODS: Patients with sensory neural hearing loss underwent SGB, repeated daily in the paratracheal approach using different volumes (6, 8, 10, 12 and 16 ml) of 0.2% bupivacaine. Skin temperatures were checked in the hypothenar area before SGB, and 1, 2, 3, 4, 5, 10 and 15 minutes following SGB. Time intervals required for the elevation of skin temperature 1oC above the preblock level, and for the appearance of Horner's syndrome were recorded. RESULTS: Twenty-three patients repeatedly received SGB over 4 times at different volumes. The total SGB was 100 times, and all displayed Horner's syndrome. The total incidence of skin temperature elevation was 48%. Horner's syndrome appeared prior to the elevation of skin temperature (2.0 2.1 vs 5.3 3.8 min). There were significant differences in the incidence of skin temperature elevation, depending on the volume of the local anesthetics; 6 ml (17.6%), 8 ml (34.8%), 10 ml (52.2%), 12 ml (73.9%) and 16 ml (57.1%); greater volumes resulted in high incidences (pearson chi-square test, P = 0.005). The volume of twelve ml resulted in the highest incidence. Each patient needed different minimal volume to lead to the skin temperature elevation; 6 ml (13.1%), 8 ml (21.7%), 10 ml (17.4%), 12 ml (26.1%) and 16 ml (4.4%), and several patients (17.4%) did not have elevated skin temperature at any volumes. CONCLUSIONS: This study reveals that a large volume of local anesthetic (e.g. 12 ml) is needed in SGB for adequate sympathetic blockade to the upper extremity, and that each patient needs a different minimal volume for the skin temperature elevation to occur.
Anesthetics, Local ; Bupivacaine ; Hearing Loss ; Horner Syndrome ; Humans ; Incidence ; Miosis ; Skin Temperature* ; Skin* ; Stellate Ganglion* ; Upper Extremity*

BACKGROUND: Horner's syndrome, including ptosis and miosis, is a sign of success following stellate ganglion block (SGB). However, it is not sufficient to ensure adequate sympathetic block to the upper extremity. There are numerous recommended local anesthetic volumes for SGB. The aim of this study was to evaluate the effects of differing volumes used in SGB on the elevation of skin temperature of ipsilateral upper extremity, and to find the volume required to elevate skin temperature most frequently. METHODS: Patients with sensory neural hearing loss underwent SGB, repeated daily in the paratracheal approach using different volumes (6, 8, 10, 12 and 16 ml) of 0.2% bupivacaine. Skin temperatures were checked in the hypothenar area before SGB, and 1, 2, 3, 4, 5, 10 and 15 minutes following SGB. Time intervals required for the elevation of skin temperature 1oC above the preblock level, and for the appearance of Horner's syndrome were recorded. RESULTS: Twenty-three patients repeatedly received SGB over 4 times at different volumes. The total SGB was 100 times, and all displayed Horner's syndrome. The total incidence of skin temperature elevation was 48%. Horner's syndrome appeared prior to the elevation of skin temperature (2.0 2.1 vs 5.3 3.8 min). There were significant differences in the incidence of skin temperature elevation, depending on the volume of the local anesthetics; 6 ml (17.6%), 8 ml (34.8%), 10 ml (52.2%), 12 ml (73.9%) and 16 ml (57.1%); greater volumes resulted in high incidences (pearson chi-square test, P = 0.005). The volume of twelve ml resulted in the highest incidence. Each patient needed different minimal volume to lead to the skin temperature elevation; 6 ml (13.1%), 8 ml (21.7%), 10 ml (17.4%), 12 ml (26.1%) and 16 ml (4.4%), and several patients (17.4%) did not have elevated skin temperature at any volumes. CONCLUSIONS: This study reveals that a large volume of local anesthetic (e.g. 12 ml) is needed in SGB for adequate sympathetic blockade to the upper extremity, and that each patient needs a different minimal volume for the skin temperature elevation to occur.
Anesthetics, Local ; Bupivacaine ; Hearing Loss ; Horner Syndrome ; Humans ; Incidence ; Miosis ; Skin Temperature* ; Skin* ; Stellate Ganglion* ; Upper Extremity*

Horner syndrome (HS) occurs when there is interruption of the oculosympathetic pathway. The causes of HS are various, but HS originated from herniated cervical disc is very few. HS attributable to the lesion of the first-order neuron of cervical spinal cord is extremely rare. A 41-year old male was admitted for sudden onset of left ptosis and right side numbness. Neurological examination revealed ptosis, miosis and facial anhidrosis on the left side. MRI and CT scans demonstrated large left paramedian disc herniation with cord compression at the C4-5 level. The herniated disc was removed through anterior approach and his symptoms were improved after the operation.
Horner Syndrome ; Humans ; Hypesthesia ; Hypohidrosis ; Intervertebral Disc ; Intervertebral Disc Displacement ; Male ; Miosis ; Neurologic Examination ; Neurons ; Spinal Cord

Horner syndrome (HS) occurs when there is interruption of the oculosympathetic pathway. The causes of HS are various, but HS originated from herniated cervical disc is very few. HS attributable to the lesion of the first-order neuron of cervical spinal cord is extremely rare. A 41-year old male was admitted for sudden onset of left ptosis and right side numbness. Neurological examination revealed ptosis, miosis and facial anhidrosis on the left side. MRI and CT scans demonstrated large left paramedian disc herniation with cord compression at the C4-5 level. The herniated disc was removed through anterior approach and his symptoms were improved after the operation.
Horner Syndrome ; Humans ; Hypesthesia ; Hypohidrosis ; Intervertebral Disc ; Intervertebral Disc Displacement ; Male ; Miosis ; Neurologic Examination ; Neurons ; Spinal Cord

Horner syndrome is characterized by miosis, partial blepharoptosis and anhidrosis on the affected side of the face. This syndrome develops when the oculosympathetic nerve pathways to the eye and face are interrupted by various causes such as tumor in the brain, intrathoracic region or neck, surgery, drugs, trauma, carotid artery dissection, and others. It is referred to as painful Horner syndrome when Horner syndrome is accompanied by hemifacial pain. Pain is probably related to trigeminal nerve injury. Horner syndrome is a rare complication of thyroidectomy. Here, we report the case of a patient who experienced ipsilateral painful Horner syndrome after total thyroidectomy and unilateral neck dissection for thyroid cancer.
Blepharoptosis ; Brain ; Carotid Artery Injuries ; Horner Syndrome* ; Humans ; Hypohidrosis ; Miosis ; Neck ; Neck Dissection ; Thyroid Neoplasms ; Thyroidectomy* ; Trigeminal Nerve Injuries

Neurilemmoma arising from the cervical sympathetic chain is not common. It is most often manifested as a solitary, benign, and slow-growing mass, and its malignant degeneration is rare. Recently, we experienced a case of schwannoma arising from cervical sympathetic chain. A 43-year-old woman presented a left neck mass that was found incidentally. Measuring approximately 3x3 cm, the mass was located medial to the left sternocleidomastoid muscle at the level of the carotid bifurcation. Fine needle aspiration biopsy was performed, but no diagnostic information was given. A computed tomography (CT) scan showed a heterogenous, poorly-demarcated mass in the left poststyloid parapharyngeal space, displacing the carotid artery anteriorly and the internal jugular vein laterally. Tumor was excised via transcervical approach. Postoperatively, the patient exhibited mild miosis with ptosis in the left eye. We report this case with a brief review of literature.
Adult ; Biopsy ; Biopsy, Fine-Needle ; Carotid Arteries ; Female ; Horner Syndrome* ; Humans ; Jugular Veins ; Miosis ; Neck ; Neurilemmoma* ; Sympathetic Nervous System