No abstract available.
Islets of Langerhans* ; Pheochromocytoma*

No abstract available.
Child* ; Humans ; Meningitis*

No abstract available.
Gwangju* ; Humans

No abstract available.
De Lange Syndrome*

No abstract available.
Precursor Cell Lymphoblastic Leukemia-Lymphoma*

No abstract available.
Epidermal Growth Factor* ; Humans* ; Receptor, Epidermal Growth Factor*

PURPOSE: To assess the results of laser in situ keratomileusis (LASIK) enhancement by lifting the original flap and reablation for the treatment of myopic regression and undercorrection after initial LASIK. METHODS: We analyzed LASIK enhancement results in 19 patients (27 of 1026 eyes) which had myopic regression or undercorrection after initial LASIK. We evaluated the changes of refractive error, uncorrected visual acuity, best corrected visual acuity, corneal topography, and corneal thickness before and six months after enhancement. We also analyzed enhancement rate according to the original refractive error. RESULTS: Overall, the mean spherical equivalent (SE) was -7.10+/-1.78 diopters (D) before LASIK, -2.80+/-0.79D prior to enhancement and -0.31+/-0.46D at 6 months after enhancement (p<0.001). The uncorrected visual acuity at 6 months after enhancement was 1.0 or better in 10 eyes (37.0%) and 0.5 or better in 27 eyes (100%). Best corrected visual acuity (BCVA) did not change in 12 eyes (44.4%) and improved 1 line or more in 9 eyes (33.3%) at 6 months after enhancement. The final refractive error of one group which underwent enhancement within postoperative 3 month was closer to emmetropia than that of the other which had enhancement after postoperative 3 month, but there was no statistically significant difference (p=0.589). CONCLUSIONS: LASIK enhancement by using the original flap was an effective procedure, if the changes of refractive error were stable on refraction or corneal topography.
Corneal Topography ; Emmetropia ; Humans ; Keratomileusis, Laser In Situ* ; Lifting ; Refractive Errors ; Visual Acuity

PURPOSE: The platelet number alone does not give a complete picture of platelet maturity and function, therefore, the platelet indices have been the subject of intensive study in recent years, but they have not been firmly established in the neonates. METHODS: Blood samples collected in K2 EDTA bottle from femoral vein of 176 neonates (54 healthy fullterm, 96 sick fullterm, 10 sick preterm, 9 healthy preterm and 7 healthy small for gestational age) were analyzed with CELL-DYN 1600. RESULTS: 1) Platelet counts (PC) and platelet indices of 70 healthy neonates correlated neither with gestational age, nor with birth weight. 2) Significant correlations were found between PC and plateletcrit and between PC and mean platelet volume with the correlation coefficients being 0.937 and -0.284 respectively. Other indices did not show any significant correlation. 3) There were no significant differences of PC and platelet indices between healthy neonates and sick neonates, between healthy fullterm and sick fullterm, between healthy preterm and sick preterm, between healthy fullterm and healthy preterm, and between healthy fullterm and healthy small for gestational age. 4) Sick fullterm neonates suffering certain diseases showed altered indices, compared with healthy fullterm neonates. In diarrhea, PC, plateletcrit and platelet distribution width were higher than those in healthy fullterm neonates. In hyperbilirubinemia and urinary tract infection, PC and plateletcrit were higher, whereas in sepsis, platelet distribution width was lower. CONCLUSIONS: These data indicate that PC and platelet indices of healthy neonates did not correlate with gestational age, nor with birth weight, and show some significant correlations between PC and platelet indicies. Further studies will be needed in clinical application of platelet indices to each neonatal disease.
Birth Weight ; Blood Platelets* ; Diarrhea ; Edetic Acid ; Femoral Vein ; Gestational Age ; Humans ; Hyperbilirubinemia ; Infant, Newborn* ; Mean Platelet Volume ; Platelet Count* ; Sepsis ; Urinary Tract Infections

Ectopic pancreas is commonly located at stomach, duodenum and jejunum. It is a relatively rare entity and is usually of no clinical importance which especially is a very rare source of massive upper gastrointestinal bleeding. We have recently experienced a case of an ectopic pancreas in the second portion of the duodenum which was proven to be a source of massive gastrointestinal bleeding by duodenoscopy and was confirmed by surgical exploration.
Duodenoscopy ; Duodenum* ; Esophagus* ; Hemorrhage* ; Intestines* ; Jejunum ; Pancreas* ; Stomach*

No abstract available.
Hyperlipoproteinemias*