1.Two Cases of Segmantal Dilatation of the Intestine in Newborn Infants.
Tai Sung JUNG ; Eun Sil LEE ; Son Moon SHIN ; Young Soo HUH
Korean Journal of Perinatology 1997;8(3):315-323
Segmental dilatation of small intestine or colon can induce signs of intestinal obstruction, such as abdominal distension, vomiting and constipation. There are no anatomical gross obstructive lesions, and moreover, ganglion cells are observed in both dilated and undilated distal segments of the intestine. It often accompanied by other congenital anomalies. We reported two cases of segmental dilatation of the intestine in the newborn infants, one in small intestine and the other in colon, with brief review of the related literatures.
Colon
;
Constipation
;
Dilatation*
;
Ganglion Cysts
;
Humans
;
Infant, Newborn*
;
Intestinal Obstruction
;
Intestine, Small
;
Intestines*
;
Vomiting
2.Photodynamic Therapy with Methyl Aminolevulinate for Disseminated Superficial Porokeratosis.
Joon Won HUH ; Young In JEONG ; Geon KIM ; Mihn Sook JUE ; Eun Jung KIM ; Hyangjoon PARK
Korean Journal of Dermatology 2014;52(10):757-758
No abstract available.
Photochemotherapy*
;
Porokeratosis*
3.A Case of Adult Colloid Milium on the Scalp.
Joon Won HUH ; Young In JEONG ; Geon KIM ; Mihn Sook JUE ; Hyangjoon PARK ; Eun Jung KIM
Korean Journal of Dermatology 2015;53(9):740-741
4.Merkel Cell Carcinoma Coexistent with Epidermal Cyst in an Immunosuppressed Man.
Young In JEONG ; Joon Won HUH ; Geon KIM ; Eun Jung KIM ; Hyang Joon PARK ; Mihn Sook JUE
Korean Journal of Dermatology 2014;52(5):368-370
No abstract available.
Carcinoma, Merkel Cell*
;
Epidermal Cyst*
;
Immunosuppression
5.Pilomatricoma on the Ear Helix.
Young In JEONG ; Joon Won HUH ; Geon KIM ; Mihn Sook JUE ; Hyang Joon PARK ; Eun Jung KIM
Korean Journal of Dermatology 2015;53(3):261-262
No abstract available.
Ear*
;
Pilomatrixoma*
6.A quick measurement of stress in outpatient clinic setting.
Jung Myon BAE ; Eun Kyeong JEONG ; Tai Woo YOO ; Bong Yul HUH ; Cheol Hwan KIM
Journal of the Korean Academy of Family Medicine 1992;13(10):809-820
No abstract available.
Ambulatory Care Facilities*
;
Humans
;
Outpatients*
8.Central Nervous System Involvement in a Patient with Multiple Myeloma Manifesting as an Intraventricular Mass with Leptomeningeal Spread
Jung Eun LEE ; Eun Ja LEE ; Hee Jin HUH ; Jae Woo CHUNG ; Eun Kyoung LEE ; Hyun Jung LEE
Journal of the Korean Radiological Society 2018;79(1):50-55
Central nervous system involvement in multiple myeloma (CNS-MM) is a rare condition. Various manifestations of CNS-MM have been reported, including dural, parenchymal, and leptomeningeal involvement. Among them, leptomeningeal involvement is less common and intraventricular involvement is exceptional, with only one case reported in the literature. Herein, we report the first case of CNS-MM manifesting as an intraventricular mass with leptomeningeal involvement combined with perineural spread. We also describe characteristic computed tomography and magnetic resonance imaging findings of intraventricular multiple myeloma.
9.Computerized Tomography Findings Suggesting Non-aneurysmal Spontaneous Subarachnoid Hemorrhage.
Yong Eun CHO ; Seung Kon HUH ; Jung Ho SUH ; Kyu Chang LEE
Journal of Korean Neurosurgical Society 1988;17(5):995-1002
Fourty-seven patients with non-aneurysmal spontaneous subarachnoid hemorrhage were reviewed retrospectively. Attention was directed to the distribution and amount of subarachnoid hemorrhage on computerized tomography scans. Though the hemorrhage could be distributed in all cisterns, the frequency and amount of hemorrhage were higher in infratentorial cisterns than in supratentorial cisterns. Among infratentorial cisterns, the hemorrhage was distributed to cisterns around the brain-stem mainly and it had a predilection for interpeduncular cistern. Also the telangiectasia of thalamoperforating artery might be one of the causes of non-aneurysmal spontaneous subarachnoid hemorrhage.
Arteries
;
Hemorrhage
;
Humans
;
Retrospective Studies
;
Subarachnoid Hemorrhage*
;
Telangiectasis
10.The Overxpression of p53 in gestational Trophoblastic Disease and Normal Human Placenta.
Sung Ook WHANG ; Jong Hyeok KIM ; Jooryung HUH ; Yong Man KIM ; Young Tak KIM ; Joo Hyun NAM ; Jung Eun MOK
Korean Journal of Gynecologic Oncology and Colposcopy 1997;8(3):300-314
Mutations in the tumor suppressor p53 gene are the most frequently observed genetic lesions in human cancers. It seems that wild type p53 does significant role on growth and differentiation of normal cells, Mutations and allelic loss of the p53 gene are thought to be a cause of tumor development and to be correlated with the prognostic factors in various human cancers such as breast, ovary and lung cancer. Mutant p53 proteins have a prolonged half-life and can be detected by immunohistochemistry. In case of GTD(gestational trophoblastic disease), although the mutation of p53 gene mutation was revealed to be very rare, the overexpression of p53 in immunohistochemical staining has been reported in wide range of discrepancy and its role or prognostic significance in GTD is uncertain. This study is performed to define the status of p53 overexpression in GTD and to evaluate the correlations between p53 overexpression and prognostic factors of GTD. THE RESULTS WERE AS FOLLOWS: 1. p53 overexpression was detected in none of normal placental tissue, in 58.3%(14/24) of hydatidiform mole, in 15%(6/8) of invasive mole, in 75%(3/4) of choriocarcinoma, and in 100%(1/1) of placental site trophoblastic tumor, and showed significant difference between normal placenta and GTD. We could not find any difference of the p53 overexpression between benign group(H-mole) of GTD and malignant one(invasive mole, choriocarcinoma, and placental site trophoblastic tumor) 2. In H-mole, low-risk group showed significantly higher prevalence of p53 overexpression than high-risk group did. In malignant group, there is no difference in the prevalence of p53 overexpression between early(FIGO stage I) and late(II- IV)stage-diseases, but the prevalence of p53 overexpression of low-risk group is slightly higher than that of high-risk group although we failed to find statistical significance. In conclusion, the high prevalence of p53 overexpression in GTD suggests that p53 may have a certain role in the pathogenesis of GTD or at least represent generalized DNA damage or genetic instability of GTD. And the higher prevalence of p53 overexpression in low-risk group suggests that accumulation of wild-type p53 may be related with favorable prognosis in GTD.
Breast
;
Choriocarcinoma
;
DNA Damage
;
Female
;
Genes, p53
;
Gestational Trophoblastic Disease*
;
Half-Life
;
Humans*
;
Hydatidiform Mole
;
Hydatidiform Mole, Invasive
;
Immunohistochemistry
;
Loss of Heterozygosity
;
Lung Neoplasms
;
Ovary
;
Placenta*
;
Pregnancy
;
Prevalence
;
Prognosis
;
Trophoblastic Tumor, Placental Site
;
Trophoblasts