1.A Case of Oil Granuloma Developed after Application of Squalene on Both Eyelids.
Jong Seo LEE ; Kwang Hoon LEE ; Soo Il CHUN
Korean Journal of Dermatology 1995;33(1):135-139
Oil granuloma develops mainly after injection of oil substances such as paraffin, silicone, etc, but there have been reports of oil granuloma after topical application of lipid substance to the body. A 22-year-old woman developed oil granuloma after the application of squalene on both lower eye-lids. Histopathologic findings ahowed acanthosis and dyskeratosis of the hair follicles. Lipid suketances staining with oil-red-O stain were found mainly on the hair follicles. and partly on the epidermis. We report a case of oil grinulorna caused by squalerie that may have penetrated through the hair follicles and partly through the epidermis resulting in chronic granulous changes in the dermis.
Dermis
;
Epidermis
;
Eyelids*
;
Female
;
Granuloma*
;
Hair Follicle
;
Humans
;
Paraffin
;
Silicones
;
Squalene*
;
Young Adult
2.V-Y advanced hamstring myocutaneous flap for the treatment of ischial pressure sores.
Jong Ryang LEE ; Jae Sung HA ; You Jin LEE ; Chung Oh SEO ; Kun Soo CHUN
Journal of the Korean Society of Plastic and Reconstructive Surgeons 1992;19(5):871-877
No abstract available.
Myocutaneous Flap*
;
Pressure Ulcer*
3.The effect of ultraviolet-B irradiation on the cardiac allograft in mice.
Jong Seo LEE ; Hae Myung CHUN ; Eung Kook KIM ; Sang Yong CHOO
Journal of the Korean Surgical Society 1991;41(1):1-8
No abstract available.
Allografts*
;
Animals
;
Mice*
4.A clinical analysis and follow up study of stomach cancer.
Jong Ryul LEE ; Kyoung Ho SEO ; Il Dong JUNG ; Bong Kweon CHUN ; Doo Bok PARK
Journal of the Korean Surgical Society 1993;44(2):196-207
No abstract available.
Follow-Up Studies*
;
Stomach Neoplasms*
;
Stomach*
5.A study of the cause of metal failure in treatment of femur shaft fracture: Fractographical and clinical analysis of metal failure.
Chun Bae JEON ; Jae Sung SEO ; Jong Chul AHN ; Myun Whan AHN ; Joo Chyl IHN
Yeungnam University Journal of Medicine 1990;7(1):81-93
The author fractographically analyzed the cause of metal failure (the first time this procedure has been used for this metal failure) and also analyzed in clinically. In this study, I selected eight cases which have been analyzed fractographically. In all these cases, the analysis was done after treatment of metal failure of implants internally fixed to femur shaft fractures at the Department of Orthopedic Surgery, Yeung-Nam University Hospital during the six year period from May 1983 to September 1989. 1. Metal failure occurred in five dynamic-compression plates, one Jewett nail, one screw in Rowe plate, and one interlocking nail. 2. The clinical cause of metal failure was deficiency of medial buttes in five cases, incorrect position of implant in one case, and incorrect selection of implant in two cases. 3. The time interval between internal fixation and metal failure was four months in one case, between five months to twelve months in six cases, three years in one case. 4. The fractographically analytical cause of metal failure was ; first, impact failure, one case, second, fatigue failure, six cases, machining mark (stress riser), four case type: low consistent cyclic fatigue failure irregular cyclic fatigue failure third, stress corrosion crack, one case. 5. 316 L Stainless Steel has good resistance to corrosion. However, when its peculiar surface film is destroyed by fretting, it shows pitting corrosion. This is, perhaps, the main cause of metal failure. 6. It is possible that mechanical injury occurred in implants during the manufacturing of implants or that making a screw hole in the main cause of metal failure.
Corrosion
;
Fatigue
;
Femur*
;
Orthopedics
;
Stainless Steel
6.Correction of bilateral cleft lip with black method.
Jong Ryang LEE ; Jae Sung HA ; You Jin LEE ; Chung Oh SEO ; Kun Soo CHUN
Journal of the Korean Society of Plastic and Reconstructive Surgeons 1992;19(5):747-753
No abstract available.
Cleft Lip*
7.Keratoacanthoma in Co-existence with Nevus Sebaceus.
Seung Won AHN ; Tae Jong CHUN ; Seong Joon SEO ; Chang Kwun HONG
Annals of Dermatology 2000;12(2):138-140
Nevus sebaceus of Jadassohn has been referred to as a organoid nevus that classically evolves through three stages and may be associated with a range of skin tumors, and of these, syringocystadenoma papilliferum and basal cell carcinoma have been commonly reported to occur. The occurrence of keratoacanthoma is a very rare event in nevus sebaceus. Herein we report on a 23-year-old man with nevus sebaceus of the cheek. He represents a keratoacanthoma arising within nevus sebaceus.
Carcinoma, Basal Cell
;
Cheek
;
Humans
;
Keratoacanthoma*
;
Nevus*
;
Nevus, Sebaceous of Jadassohn
;
Organoids
;
Skin
;
Young Adult
8.The Study of Tenascin Expression in Vitiligo.
Jong Seong AHN ; Kyung Chan PARK ; Young Gull KIM ; Kwang Hyun CHO ; Jung Wook SEO ; Duk Kyu CHUN
Korean Journal of Dermatology 1999;37(4):495-499
BACKGROUND: Defective adhesion and migration of melanocyte may be involved in pathogenesis of vitiligo. Tenascin, a glycoprotein of the extracellular matrix, has a role in cell adhesion and migration. It has been reported that abundant expression of tenascin in vitiligo lesion may inhibit melanocyte adhesion and migration. OBJECTIVE: To investigate the tenascin expression in vitiligo skin lesions and to compare with clinical findings. METHODS: We studied 9 patients with vitiligo. The expressions of tenascin were studied by immunahistochemieal techniques.
Cell Adhesion
;
Extracellular Matrix
;
Glycoproteins
;
Humans
;
Melanocytes
;
Skin
;
Tenascin*
;
Vitiligo*
9.Activation of NF-kappa B in the cisplatin-induced apoptosis of oral squamous cell carcinoma.
Jong Chun SEO ; Iel Yong SUNG ; Jong Roul KIM
Journal of the Korean Association of Oral and Maxillofacial Surgeons 2006;32(2):94-100
Purpose: This study was done to confirm the role of NF-kappa B in cisplatin-induced apoptosis of oral squamous cell carcinoma. Materials and Methods: Five cell lines originated from different oral cancer patients were tested for the apoptosis by the treatment of cisplatin. These cells showed different degree of cisplatin-resistance and the order is OSCC-2> OSCC-3> OSCC-5> OSCC-1> OSCC-4. OSCC-2 and OSCC-4 cells were assayed for the apoptosis by measuring DNA fragmentation and TUNEL staining after cisplatin treatment. While OSCC-4 cells showed apoptosis, OSCC-2 cells showed no or very slight apoptosis by cisplatin treatment. Next, It was determined whether NF-kappa B activation is required in mediating cisplatin-induced apoptosis of OSCC-4 . Result: The result was that elevated NF-kappa B activity mediated cisplatin-induced apoptosis. Conclusion: In conclusion, these findings suggest that NF-kappa B activation is essential to cisplatin-induced apoptosis and it may be involved in cisplatin resistance in OSCC cells.
Apoptosis*
;
Carcinoma, Squamous Cell*
;
Cell Line
;
Cisplatin
;
DNA Fragmentation
;
Humans
;
In Situ Nick-End Labeling
;
Mouth Neoplasms
;
Negotiating
;
NF-kappa B*
10.Activation of NF-kappa B in the cisplatin-induced apoptosis of oral squamous cell carcinoma.
Jong Chun SEO ; Iel Yong SUNG ; Jong Roul KIM
Journal of the Korean Association of Oral and Maxillofacial Surgeons 2006;32(2):94-100
Purpose: This study was done to confirm the role of NF-kappa B in cisplatin-induced apoptosis of oral squamous cell carcinoma. Materials and Methods: Five cell lines originated from different oral cancer patients were tested for the apoptosis by the treatment of cisplatin. These cells showed different degree of cisplatin-resistance and the order is OSCC-2> OSCC-3> OSCC-5> OSCC-1> OSCC-4. OSCC-2 and OSCC-4 cells were assayed for the apoptosis by measuring DNA fragmentation and TUNEL staining after cisplatin treatment. While OSCC-4 cells showed apoptosis, OSCC-2 cells showed no or very slight apoptosis by cisplatin treatment. Next, It was determined whether NF-kappa B activation is required in mediating cisplatin-induced apoptosis of OSCC-4 . Result: The result was that elevated NF-kappa B activity mediated cisplatin-induced apoptosis. Conclusion: In conclusion, these findings suggest that NF-kappa B activation is essential to cisplatin-induced apoptosis and it may be involved in cisplatin resistance in OSCC cells.
Apoptosis*
;
Carcinoma, Squamous Cell*
;
Cell Line
;
Cisplatin
;
DNA Fragmentation
;
Humans
;
In Situ Nick-End Labeling
;
Mouth Neoplasms
;
Negotiating
;
NF-kappa B*