Traditionally, the Rhododendron species has been used in gastrointestinal disorder or hypertension. Grayanotoxin exists in honey, flowers, pollen, and the nectar of the Rhododendron species. We experienced 3 cases of Grayanotoxin intoxication. The symptoms of intoxication were nausea, vomiting, hypotension, bradycardia, diplopia, dizziness, and chest discomfort. Generally, the treatment for Grayanotoxin intoxication is fluid resuscitation and injection of atropine sulfate. The patients who were intoxicated with Grayanotoxin were discharged without complication after supportive care.
Atropine ; Bradycardia ; Diplopia ; Dizziness ; Flowers ; Honey ; Humans ; Hypertension ; Hypotension ; Nausea ; Plant Nectar ; Pollen ; Resuscitation ; Rhododendron ; Thorax ; Vomiting

Jeollabuk-do ; Osteotomy ; Recurrence

Orthognathic Surgery ; Politics ; Prognosis ; Social Change

PURPOSE: To compare the sedation outcome according to the dose of per os chloral hydrate in children who underwent laceration repair in the emergency department (ED). METHODS: This retrospective study was performed to the children who underwent sedation using chloral hydrate for laceration repair in the ED from January 2015 through November 2015. A total of 370 children aged younger than 6 years underwent the sedation. We compared the induction time, duration of sedation, and ED length of stay (EDLOS) between the single dose (50 mg/kg) and additional dose (plus 25 mg/kg) groups. RESULTS: Of 370 children, 335 (90.5%) were sedated successfully, 284 (76.8%) were sedated with initial dose (the single dose group), and 51 (13.8%) were sedated with additional dose (the additional dose group). The induction time and EDLOS were longer in the additional dose group (induction time: 31.0 ± 17.2 minutes vs. 96.2 ± 25.4 minutes, P < 0.001; EDLOS: 137.2 ± 35.5 minutes vs. 193.0 ± 36.0 minutes, P < 0.001). The duration of sedation showed no difference between the 2 groups (44.4 ± 24.0 minutes vs. 42.0 ± 20.8 minutes; P = 0.500). No one had serious adverse reactions. CONCLUSION: Additional dose of chloral hydrate can increase the induction time and EDLOS without increasing the duration of sedation and causing serious adverse reactions. This information may improve the efficiency of ED workflow when shared with parents of the children.
Child* ; Chloral Hydrate* ; Conscious Sedation ; Emergency Service, Hospital ; Humans ; Lacerations* ; Length of Stay ; Parents ; Retrospective Studies

Cicatrix ; Esthetics ; Facial Nerve Injuries ; Hand ; Mandibular Condyle ; Temporomandibular Joint

Diplopia ; Ecchymosis ; Edema ; Enophthalmos ; Epistaxis ; Frontal Bone ; Humans ; Orbit ; Subcutaneous Emphysema ; Zygoma

In Korea, the dependence on living donations is high due to the shortage of organs available for donation compared to the number of people waiting for transplants and the number of living organ donations continues to increase. In particular, the number of living-donor transplantations is high worldwide, highlighting the importance of pre-transplant psychosocial evaluation of living kidney donors. According to previous studies, when evaluating living organ donors before transplantation, it is crucial to determine whether the donor can give informed consent and be aware of the risks after surgery. Pre-transplant evaluation tests such as ELPAT living organ donor Psychological Assessment Tool (EPAT), Live Donor Assessment Tool (LDAT), Living Donation Expectancies Questionnaire (LDEQ), Minnesota Multiphasic Personality Inventory-2 questionnaire (MMPI-2) and Temperament and Character Inventory (TCI) are conducted for donors. After reviewing the literature on these pre-transplant psychosocial assessment tools, we will also look at legal considerations for living kidney donors in Korea and suggest an effective and essential pre-transplant screening evaluation method for living kidney transplant donors.

Penetrating facial wounds are uncommon and are usually life threatening because of the possibility of brain damage. There are three possible pathways for penetrating the cranium through the orbit: via the orbital roof, via the superior orbital fissure, or between the optic canal and lateral wall of the orbit. Brain injuries resulting from the penetrating wounds show extensive parenchymal damage, hemorrhage, and brain edema. Transorbital penetrating wounds can lead to diverse lesions of the optical apparatus, including the eye globe, the optical nerve, and the chiasm. Moreover, intracerebral structures may be hurt, and bleeding and infection may occur. Early diagnosis and prompt debridement are the fundamental factors affecting the outcome of a penetrating facial wound. An 87-year-old man was admitted to the emergency department with a grinder impacted into the medial aspect of the right eye. On presentation, the man was fully conscious with a Glasgow Coma Scale score of 15 and complained of a visual disturbance of the right eye. Computed tomography demonstrated a right orbital medial and inferior wall fracture, a frontal bone fracture, and a contusional hemorrhage in frontal lobe of the brain. A craniotomy with hematoma removal and repair of the orbital floor was done. He showed no neurological deficits except right visual loss. This appears to be the first report of a man with a penetrating facial wound caused by a grinder, who presented with a potentially disastrous craniocerebral injury that did not lead to any serious neurological seguelae.
Aged, 80 and over ; Brain ; Brain Edema ; Brain Injuries ; Contusions ; Craniocerebral Trauma ; Craniotomy ; Debridement ; Early Diagnosis ; Emergency Service, Hospital ; Frontal Bone ; Frontal Lobe ; Glasgow Coma Scale ; Hematoma ; Hemorrhage ; Humans ; Orbit ; Skull ; Wounds and Injuries* ; Wounds, Penetrating

PURPOSE: We analyzed the characteristics and outcome of pediatric out-of-hospital cardiac arrest. METHODS: Pediatric out-of-hospital cardiac arrest from January 2000 to December 2005 at two tertiary hospitals were described and evaluated using the Utstein style. We reviewed the records retrospectively and analyzed the outcome variables which were any return of spontaneous circulation (ROSC), sustained ROSC, survived event, and survival to hospital discharge. Neurologic outcome was assessed by the Pediatric Cerebral Performance Category (PCPC) scale. RESULTS: The study included 62 children with out-of-hospital cardiac arrest. Any ROSC was achieved in twenty patients (32.3%). Sustained ROSC of any ROSC group was achieved in sixteen patients (80.0%). Of the sustained ROSC group, fourteen patients (87.5%) were admitted to hospital, and only four patients (28.6%) of survived event group survived to hospital discharge. The prevalent etiology were injuries. Although 35 children (56.5%) of the arrests occurred at home with family members present, only 1 patients received bystander CPR. Nonshockable rhythm (96.8%) were showed more than shockable rhythm (3.2%). In any ROSC group, time to initiation of CPR was 9.3 minutes,duration of total CPR was 20.4 minutes. CONCLUSION: Mortality of pediatric out-of-hospital cardiac arrest was high and neurologic outcome was poor. Factors that increased survival rate were prevention of injuries, enhanced education programs of bystander CPR, rapid initiation of CPR.
Cardiopulmonary Resuscitation ; Child ; Education ; Humans ; Mortality ; Out-of-Hospital Cardiac Arrest* ; Resuscitation ; Retrospective Studies ; Survival Rate ; Tertiary Care Centers

Antiphospholipid antibody (APLA) syndrome, which is presented by seizure, is uncommon. Most seizures in APLA syndrome are associated with systemic lupus erythematosus (SLE) or other autoimmune diseases. Its pathophysiology is not clear; however, many authorities suggest that autoantibodies related to APLA syndrome are related to an ischemic change in the brain. We experienced a man with APLA syndrome, who presented with a seizure, but he had no evidence of an ischemic change in the brain. He underwent conservative management for 2 weeks and was discharged. He was admitted twice due to seizure. One year later, he had a cerebrovascular attack and was hospitalized.
Antibodies, Antiphospholipid* ; Antiphospholipid Syndrome* ; Autoantibodies ; Autoimmune Diseases ; Brain ; Lupus Erythematosus, Systemic ; Seizures