Objective: To explore the emotional experience of abnormal fetal pregnant women after induced labor. Methods: Phenomenological methods were used to conduct in-depth interviews with 20 pregnant women with abnormal fetal induced labor from November 2018 to January 2019. Colaizzi analysis method was used to sort, code, classify, summarize and extract the subjects. Results: Two themes were extracted: Post-traumatic growth experiences and promotive factors of PTG. Post-traumatic growth experiences included traumatic reaction, new life attitude, exploring potential advantages, cherishing the relationship between relatives and friends, and actively seeking knowledge;Promotive factors of PTG included providing social support, mobilizing internal positive cognition, diverting attention, and planning for the future. Conclusions Medical staff should provide opportunities for pregnant women with abnormal fetal induced labor to express their emotions, fully integrate social and emotional support, mobilize internal positive cognition, and then promote their post-traumatic growth.

Gastric cancer is one of the most common malignancies worldwide; however, the molecular mechanism in tumorigenesis still needs exploration. BCL2L11 belongs to the BCL-2 family, and acts as a central regulator of the intrinsic apoptotic cascade and mediates cell apoptosis. Although miRNAs have been reported to be involved in each stage of cancer development, the role of miR-24 in GC has not been reported yet. In the present study, miR-24 was found to be up-regulated while the expression of BCL2L11 was inhibited in tumor tissues of GC. Studies from both in vitro and in vivo shown that miR-24 regulates BCL2L11 expression by directly binding with 3'UTR of mRNA, thus promoting cell growth, migration while inhibiting cell apoptosis. Therefore, miR-24 is a novel onco-miRNA that can be potential drug targets for future clinical use.
Animals ; Apoptosis ; genetics ; Apoptosis Regulatory Proteins ; deficiency ; genetics ; Base Sequence ; Bcl-2-Like Protein 11 ; Cell Line, Tumor ; Cell Movement ; genetics ; Cell Proliferation ; genetics ; Down-Regulation ; genetics ; Gene Silencing ; Male ; Membrane Proteins ; deficiency ; genetics ; Mice ; MicroRNAs ; genetics ; Proto-Oncogene Proteins ; deficiency ; genetics ; Rats ; Stomach Neoplasms ; genetics ; pathology