1.Toward Unity of the Mind and Body.
Journal of Korean Neuropsychiatric Association 2010;49(3):287-290
No abstract available.
2.Somatiaztion disorder.
Journal of the Korean Academy of Family Medicine 2001;22(8):1153-1162
No abstract available.
3.Psychiatric Disorders in Primary Care Setting.
Journal of the Korean Medical Association 1997;40(12):1663-1671
No abstract available.
Primary Health Care*
5.The Future of Research on Extracorporeal Membrane Oxygenation (ECMO).
Korean Journal of Critical Care Medicine 2016;31(2):73-75
No abstract available.
Extracorporeal Membrane Oxygenation*
6.Effects of Alcohol-Related Factors on Drinking Behaviors and Problematic Sexual Behaviors in College Students.
Korean Journal of Health Promotion 2011;11(2):91-99
BACKGROUND: The purpose of this study was to explore the relationships between psychosocial stress, alcohol expectancy, drinking refusal self-efficacy, and drinking behaviors with problematic sexual behaviors in college students. METHODS: Participants were college students (129 males, 67 females) with a mean age of 22.6 years old. Data analysis was done with SPSS 13.0 for descriptive statistics and LISREL 8.53 program for path analysis. RESULTS: The path model showed a good fit to the empirical data (chi2=10.14, P=0.71, GFI=0.98, AGFI=0.94, CFI=0.96, RMSEA=0.07). Our analysis showed that psychosocial stress, drinking refusal self-efficacy, and alcohol expectancy had a significant direct effect on drinking behaviors and that drinking behaviors had a significant direct effect on problematic sexual behaviors. CONCLUSIONS: These findings suggest that alcohol prevention or education programs should emphasize stress management and refusal skills training to prevent college students from drinking behaviors and problematic sexual behaviors at the highest risk for various stressors and heavy drinking.
Alcohols
;
Disulfiram
;
Drinking
;
Drinking Behavior
;
Humans
;
Male
;
Sexual Behavior
;
Statistics as Topic
7.Prurigo Pigmentosa after Injection Lypolysis with Phosphatidylcholine and Deoxycholate.
Korean Journal of Dermatology 2016;54(8):673-674
No abstract available.
Deoxycholic Acid*
;
Phosphatidylcholines*
;
Prurigo*
8.Clinical analysis of choledochal cyst.
Journal of the Korean Surgical Society 1992;43(1):60-66
No abstract available.
Choledochal Cyst*
9.The Clinical Efficacy of BTA Stat Test in the Diagnosis for Bladder Cancer.
Korean Journal of Urology 2000;41(1):23-28
No abstract available.
Diagnosis*
;
Urinary Bladder Neoplasms*
;
Urinary Bladder*
10.In Vitro Perfusion Studies on Coronary Function of Cardiac Ischemia-Reperfusion in Spontaneously Hypertensive Rat Heart.
Korean Circulation Journal 1995;25(1):59-67
BACKGROUND: Myocardial ischemia in human hypertension and in various animal models of hypertension may be due to abnormal maximal coronary vasodilator reserve and disturbaces of coronary vasomotion. The vascular reactivity defects in hypertension have been associated with the defective endothelium and sympathetic neural activation. However, such abnormalities in hypertension need to be elucidated. In the present study the effectsof cardiac ischemia reperfusion on coronary circulation, intramyocytic adenylates and purine nucleosides were examined in Langendorff-perfused Sprague Dawley rat (SD) and spontaneously hypertensive rat (SHR) hearts. Coronary venous and cardiac lactate and cardiac pyruvate were also measured. It should be noted that in the regulation of coronary flow the intrinsic flow autoregulation is highly variable due to coexisting metabolic flow control, and that natural coronary flow and cardiomyocytic energy state are normally reciprocally related in perfused heart. METHODS: For the Langendorff heart perfusion, bicarbonate perfusion buffer (pH 7.40+/-0.02,37degrees C) was equilibrated with 95% O2 : 5% CO2 and contained 5mM glucose (+5U/1 insulin) and 2mM pyruvate as energy-yielding substrates. Global hypoperfusion ischemia was induced by lowering coronary perfusion pressure of 100 to 40 cmH2O, followed by 20 min reperfusion. RESULTS: During the ischemia and reperfusion, metabolic acidosis and enhanced venous lactate output in SHR were observed with increases in coronary vascular resistance and myocardial oxygen consumption.In addition, coronary reactive hyperemia during reperfusion was depressed. Although ischemia-induced increase in combined adenosine plus inosine were abolished during prolonged reperfusion, SD still exhibited coronary vasodilation. The depressed reactive hyperemia in SHR was associated with decreases in cardiac adenosine triphosphate (ATP) pool and creatine phosphate/inorganic phosphate (CrP/Pi) ratio and an increase in cardiac lactate/pyruvate ratio. CONCLUSION: This abnormal vascular reactivity during ischemia and reperfusion in SHR may be in part due to an alteration in the cardiac energy state and hence to a mismatch between myocardial metabolic demand and supply.
Acidosis
;
Adenosine
;
Adenosine Triphosphate
;
Animals
;
Coronary Circulation
;
Creatine
;
Endothelium
;
Glucose
;
Heart*
;
Homeostasis
;
Humans
;
Hyperemia
;
Hypertension
;
Inosine
;
Ischemia
;
Lactic Acid
;
Models, Animal
;
Myocardial Ischemia
;
Oxygen
;
Perfusion*
;
Purine Nucleosides
;
Pyruvic Acid
;
Rats
;
Rats, Inbred SHR*
;
Reperfusion
;
Vascular Resistance
;
Vasodilation