1.BC Gitis.
Kwang Wook KO ; Je Keun JI ; Kwi Won PARK
Journal of the Korean Pediatric Society 1982;25(12):1295-1300
No abstract available.
2.Pancreatic pseudocyst associated with severe adhesive ileus.
Kwang Wook KO ; Je Geun CHI ; Kwi Won PARK
Journal of the Korean Pediatric Society 1983;26(10):1044-1047
No abstract available.
Adhesives*
;
Ileus*
;
Pancreatic Pseudocyst*
3.Congenital biliary atresia.
Kwang Wook KO ; Je Geun CHI ; Kwi Won PARK
Journal of the Korean Pediatric Society 1983;26(1):106-
No abstract available.
Biliary Atresia*
4.Jejunal atresia with meconium peritonitis and sepsis.
Kwang Wook KO ; Je Geun CHI ; Kwi Won PARK
Journal of the Korean Pediatric Society 1982;25(9):972-976
No abstract available.
Intestinal Atresia*
;
Meconium*
;
Peritonitis*
;
Sepsis*
5.Tracheal Foreign Body Associated with Esophageal Duplication Cyst.
Kwang Wook KO ; Je Geun CHI ; Kwi Won PARK
Journal of the Korean Pediatric Society 1985;28(10):1052-
No abstract available.
Foreign Bodies*
6.Intracranial Encephalocele: an autopsy case of anterior basal type.
Hyun Wook KANG ; Je G CHI ; Tae Dong PARK ; Hum Rae PARK
Korean Journal of Pathology 1988;22(4):500-504
Encephalocele is a relatively rare congenital anomaly which is classified into occipital, parietal, anterior syncipital and anterior basal type regarding to the protrusion site through the bony defect of the skull. Anterior basal type of encephalocele is important in view of it's pathogenesis as well as diagnostic difficulty because of invisibility on external appearance. We have experienced a case which could be best fit to anterior basal encephalocele. This type of encephalocele is extremely rare. This report deals with a case of deadborn of 34 weeks of gestation with body weight of 2400gm and head circumference of 32 cm. There was no evidence of protrusion of brain on external examination. On autopsy the normal brain structure was compressed by abnormal mass of brain with normal consistency which was found in the petrous portion of the parietal area and covered partly by the dura. In this case, there were another associated anomalies, such as atrophy of the left optic nerve, hemihypoplasia of the left mandible, patent ductus arteriosus, bilateral hydrocele, and Meckel's diverticulum.
7.Continuous culture of recombinant mammalian cells producing hepatitis B virus surface antigen in stirred tank reactor equipped with a cell sedimentation column.
Suk Hoon HA ; Tae Wook HAHN ; Ree Ann YOO ; Wan Je PARK ; Hyun Su KIM
Journal of the Korean Society of Virology 1992;22(1):77-80
No abstract available.
Hepatitis B virus*
;
Hepatitis B*
;
Hepatitis*
8.Immunogenicity from polio/hepatitis B chimeric virus.
Tae Wook HAN ; Ree Ann YOO ; Suk Hoon HA ; Wan Je PARK ; Hyun Su KIM
Journal of the Korean Society of Virology 1992;22(2):111-117
No abstract available.
9.Hepatoblastoma with Rupture and Hemorrhage.
Kwang Wook KO ; Hyung Ro MOON ; Je Geun CHI ; Kwi Won PARK
Journal of the Korean Pediatric Society 1985;28(12):1261-
No abstract available.
Hemorrhage*
;
Hepatoblastoma*
;
Rupture*
10.Pyrin Domain (PYD)-containing Inflammasome in Innate Immunity.
Sujeong HONG ; Sangjun PARK ; Je Wook YU
Journal of Bacteriology and Virology 2011;41(3):133-146
Inflammasome is a cytosolic multiprotein complex to activate caspase-1 leading to the subsequent processing of inactive pro-interleukin-1-beta (Pro-IL-1beta) into its active interleukin-1 beta (IL-1beta) in response to pathogen- or danger-associated molecular pattern. In recent years, a huge progress has been made to identify inflammasome component as a molecular platform to recruit and activate caspase-1. Nucleotide-binding oligomerization domain-like receptor (NLR) family proteins such as NLRP1, NLRP3 or interleukin-1beta-converting enzyme (ICE)-protease activating factor (IPAF) have been first characterized to form inflammasome complex to induce caspase-1 activation. More recently, non-NLR type, pyrin-domain (PYD)-containing proteins such as pyrin or absent in melanoma2 (AIM2) were also proposed to form caspase-1-activating inflammasome machinery with apoptosis-associated speck-like protein containing a CARD (ASC), an essential adaptor molecule. Inflammasome pathways were shown to be crucial for protecting host organisms against diverse pathogen infections, but accumulating evidences also suggest that excessive activation of inflammasome/caspase-1 might be related to the pathogenesis of inflammation-related diseases. Indeed, mutations in NLRP3 or pyrin are closely associated with autoinflammatory diseases such as familial Mediterranean fever (FMF) syndrome or Muckle-Wells syndrome (MWS), indicating that the regulation of caspase-1 activity by inflammasome is a central process in these hereditary inflammatory disorders. Here, recent advances on the molecular mechanism of caspase-1 activation by PYD-containing inflammasomes are summarized and discussed.
Cryopyrin-Associated Periodic Syndromes
;
Cytoskeletal Proteins
;
Cytosol
;
Familial Mediterranean Fever
;
Humans
;
Immunity, Innate
;
Inflammasomes
;
Interleukin-1beta
;
Proteins