1.Spontaneous Intracranial Hypotension.
Doo Sik KONG ; Jong Soo KIM ; Kwan PARK ; Do Hyun NAM ; Whan EOH ; Hyung Jin SHIN ; Seung Chyul HONG ; Jong Hyun KIM
Journal of Korean Neurosurgical Society 2000;29(2):240-248
No abstract available.
Intracranial Hypotension*
2.A Case of Intracranial Hypotension after Horse Riding.
Sibel KARSIDAG ; Nilgun CINAR ; Sevki SAHIN ; Miruna Florentina ATES ; Necati Alp TABAK
Journal of Clinical Neurology 2019;15(1):130-131
No abstract available.
Horses*
;
Intracranial Hypotension*
3.Spontaneous Intracranial Hypotension Followed by Subdural Hemorrhage
Hye Won HWANG ; Byung Nam YOON
Journal of the Korean Neurological Association 2018;36(1):59-60
No abstract available.
Hematoma, Subdural
;
Intracranial Hypotension
4.Unintended Complication of Intracranial Subdural Hematoma after Percutaneous Epidural Neuroplasty.
Sung Bum KIM ; Min Ki KIM ; Kee D KIM ; Young Jin LIM
Journal of Korean Neurosurgical Society 2014;55(3):170-172
Percutaneous epidural neuroplasty (PEN) is a known interventional technique for the management of spinal pain. As with any procedures, PEN is associated with complications ranging from mild to more serious ones. We present a case of intracranial subdural hematoma after PEN requiring surgical evacuation. We review the relevant literature and discuss possible complications of PEN and patholophysiology of intracranial subdural hematoma after PEN.
Hematoma, Subdural
;
Hematoma, Subdural, Intracranial*
;
Intracranial Hypotension
5.Bilateral Subdural Hematoma due to an Unnoticed Dural Tear during Spine Surgery.
Youn Young JUNG ; Chng Il JU ; Seok Won KIM
Journal of Korean Neurosurgical Society 2010;47(4):316-318
We report a rare case of intracranial hypotension that was complicated by a subdural hematoma following spine surgery. Intraoperatively, we did not notice any breach of the dura. However, the patient continued to have fluid leakage from the inferior edge of the lumbar incision. During revision surgery, a small dural tear was identified and repaired. It is likely that a small dural tear was overlooked or the dura was weakened during the initial operation and caused a subdural hematoma associated with intracranial hypotension.
Hematoma, Subdural
;
Humans
;
Intracranial Hypotension
;
Spine
6.Intracranial Hypertension Following Epidural Blood Patch in a Patient With Spontaneous Intracranial Hypotension.
Seol Hee BAEK ; Yeon Sun WOO ; Jin Woo PARK ; Jae Gyum KIM ; Kyung Hee CHO
Journal of the Korean Neurological Association 2014;32(1):30-33
Epidural blood patch (EBP) is one of the treatments for spontaneous intracranial hypotension (SIH), and its complications have rarely been reported. We report a patient with SIH and developed intracranial hypertension after EBP. The mechanism of rebound intracranial hypertension was uncertain. If the patient presented with a different type of headache or newly developed neurological symptoms after treatment of SIH, rebound intracranial hypertension should be considered and the proper treatment needed quickly.
Blood Patch, Epidural*
;
Headache
;
Humans
;
Intracranial Hypertension*
;
Intracranial Hypotension*
7.Treatment with Epidural Blood Patch for Iatrogenic Intracranial Hypotension after Spine Surgery.
Jaekook KIM ; Sunyeul LEE ; Youngkwon KO ; Wonhyung LEE
Journal of Korean Neurosurgical Society 2012;52(3):254-256
Intracranial hypotension syndrome typically occurs spontaneously or iatrogenically. It can be associated with headache, drowsy mentality and intracranial heamorrhage. Iatrogenic intracranial hypotension can occur due to dural pucture, trauma and spine surgery. Treatment may include conservative therapy and operation. We report a case of a 54-year-old man who was successfully treated with epidural blood patches for intracranial hypotension due to cerebrospinal fluid (CSF) leakage into the lumbosacral area after spine surgery.
Blood Patch, Epidural
;
Headache
;
Humans
;
Intracranial Hypotension
;
Middle Aged
;
Spine
8.A Case of Spontaneous Intracranial Hypotension: Detection of Cerebrospinal Fluid Leakage by Early Dynamic Radionuclide Cisternography.
Seok Gun PARK ; Jae Soo KIM ; Dae Woong YANG
Korean Journal of Nuclear Medicine 1999;33(2):184-188
Although cerebrospinal fluid leakage is suggested as one of the causes of spontaneous intracranial hypotension, on]y a few cases with direct evidence of cerebrospinal fluid leakage on radionuclide cisternography have been reported in the literature Indirect evidences of cerebrospinal fluid leakage such as early visualization of the soft tissue and bladder or delayed migration of radiotracer have been observed in most patients with spontaneous intracranial hypotension. We report a case of spontaneous intracranial hypotension in which cerebrospinal fluid leakage was directly demonstrated by early dynamic imaging of spine on radionuclide cisternography. We suggest that early dynamic imaging of spine is an important adjunctive procedure in detecting cerebrospinal fluid leakage in patients with spontaneous intracranial hypotension.
Cerebrospinal Fluid*
;
Humans
;
Intracranial Hypotension*
;
Spine
;
Urinary Bladder
9.Evaluation of Spontaneous Intracranial Hypotension Using Radionuclide Cisternography.
Chan H PARK ; Seok Nam YOON ; Moonsun PAI ; Suzy KIM ; Yunmin OH ; Jangsung KIM
Korean Journal of Nuclear Medicine 1999;33(2):178-183
We report four cases of spontaneous intracranial hypotension that were investigated by radionuclide cisternography Tc-99m-diethylenetriamine pentaacetic acid radionuclide cisternography of all our patients showed direct sign of cerebrospinal fluid leakage as well as indirect signs of less activity than expected over the cerebral convexities and rapid appearance of bladder activity. The headache of all patients was eventually controlled with bed rest and hydration.
Bed Rest
;
Cerebrospinal Fluid
;
Headache
;
Humans
;
Intracranial Hypotension*
;
Urinary Bladder
10.A Fatal Adverse Effect of Barbiturate Coma Therapy: Dyskalemia.
Hyun Mook KWON ; Jin Wook BAEK ; Sang Pyung LEE ; Jae Ik CHO
Korean Journal of Neurotrauma 2016;12(2):156-158
The management guideline for traumatic brain injury (TBI) recommends high-dose barbiturate therapy to control increased intracranial pressure refractory to other therapeutic options. High-dose barbiturate therapy, however, may cause many severe side effects; the commonly recognized ones include hypotension, immunosuppression, hepatic dysfunction, renal dysfunction, and prolonged decrease of cortical activity. Meanwhile, dyskalemia remains relatively uncommon. In this study, we report the case of a hypokalemic patient with severe rebound hyperkalemia, which occurred as a result of barbiturate coma therapy administered for TBI treatment.
Brain Injuries
;
Coma*
;
Humans
;
Hyperkalemia
;
Hypokalemia
;
Hypotension
;
Immunosuppression
;
Intracranial Pressure