1.Nerestoxin poisoning
Journal of Vietnamese Medicine 2004;298(5):5-9
In 2002-2004 year period, at Bach Mai Hospital, 6 intoxicated victims aged 14-41 were admitted (3 males, 3 females). They attempted to suicide and hospitalized 15-60 min. after use the toxic with the symptoms of gastro-enteritis (vomit even vomit with blood), abdomen pain, diarrhoea, purpurae and systenic edema, circulation volume reduced, high blood pressure, tachycardia, 3 victims who consumed 7-10g recovered but 3 who consumed > 20g died 24 hours after hospitalization
Poisoning
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Insecticides
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Suicide
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Gastroenteritis
3.Fatal Brain Injury in Pyrethroid Poisoned Patient: Case Report.
Woo Jin JUNG ; Yong Sung CHA ; Dong Keon LEE ; Hyun KIM
Journal of The Korean Society of Clinical Toxicology 2014;12(2):88-91
Pyrethroids have been widely using insecticides. Although generally regarded as less toxic to mammals including humans, we report one fatal case of pyrethroid poisoning with severe brain injury.
Brain Injuries*
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Humans
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Insecticides
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Mammals
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Poisoning
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Pyrethrins
4.Three cases of acute chlorfenapyr poisoning.
Ji Lai QU ; Hai Yan YAN ; Xue Chuang ZHU ; Yu Gui HAO
Chinese Journal of Industrial Hygiene and Occupational Diseases 2023;41(6):461-462
This paper reported 3 cases of poisoning caused by chlorfenagyr. Chlorfenapyr poisoning has gradually increased in clinical practice. The early stage after poisoning is digestive tract symptoms, followed by sweating, high fever, changes in consciousness, changes in myocardial enzymology, etc. Its main mechanism of intoxication is uncoupling oxidative phosphorylation. Since there is no specific antidote after poisoning, the fatality rate of chlorfenapyr poisoning remains high. The therapeutic measures are early gastrointestinal decontamination, symptomatic and supportive treatments, and early blood purification may be an effective treatment.
Humans
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Pyrethrins
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Gastrointestinal Tract
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Insecticides
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Poisoning/diagnosis*
5.A case of hemolytic anemia induced by dipterex.
Chong-hai LIU ; Xing-wei CHEN ; Gang LI ; Chun WU ; Bin ZHONG ; Yu-shu WEI
Chinese Journal of Pediatrics 2003;41(10):765-765
Anemia, Hemolytic
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etiology
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Child
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Female
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Humans
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Insecticides
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poisoning
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Trichlorfon
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poisoning
6.Avermectin poisoning in a child.
Yi-Nan YANG ; Yu-Ying TANG ; Bao-Quan ZHU
Chinese Journal of Contemporary Pediatrics 2010;12(4):310-310
9.A Case Report of a First Sulfoximine Class of Insecticide, Sulfoxaflor Poisoning.
Jaehoon OH ; Hyunggoo KANG ; Tae Ho LIM ; Sanghyun LEE ; Chiwon AHN
Journal of The Korean Society of Clinical Toxicology 2015;13(1):43-45
Sulfoxaflor is the first insecticide belonging to the sulfoximine class and is efficient against sap-feeding insects that are resistant to other insecticides. Sulfoxaflor acts as a neurotoxin to the central nervous system of insects compared with very low toxicity to mammalian. We report on a case of a 67-year-old male who ingested insecticide and received conservative treatment for mild metabolic acidosis and gastrointestinal symptoms.
Acidosis
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Aged
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Central Nervous System
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Humans
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Insecticides
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Insects
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Male
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Poisoning*
10.A Study on the Distributions of Paraoxonase Activity and the Factors affecting Paraoxonase Activity of a Rural opulation.
Korean Journal of Preventive Medicine 1990;23(2):194-200
The paraoxonase (E. C. 3.1.1.2.) is a major enzyme to detoxicate the organophosphorus and carbamate which are the most widely used as the agricultural spraying insecticides. To investigate the distributions of plasma paraoxonase activity and the fators affecting the enzyme activity, the plasmas of 945 Korean rural population were analyzed with the modified Krisch's direct sphectro;hotometry method. Three indices of the enzyme activity-basal activity, stimulated activity (by NaCl), % stimulation -were obtained from the analysis. Three indicies suggested unimodal distributions, so we couldn't identify the low activity group; risk group to organophosphorus & carbamate insecticides poisoning. There is no significant relation between 3 activity and the stimulated activity have significant relationship and high coefficient of determination with the activities of their parents (r2=0.30, 0.24; p<0.05), but the % stimulation does not (r2=0.02; p<0.05). These results suggest that the activity of paraoxonase is determined mainly by the genetic factor.
Aryldialkylphosphatase*
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Humans
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Insecticides
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Parents
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Plasma
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Poisoning
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Rural Population