A study on 60 patients with the cerebral lacunar infarction, ages of 20-85 and 60 patients with cerebral infarction (control), ages of 20 -87 has shown that the syndromes of cerebral lacunar infarction comprised hemiplegia type of merely moving, speaking disorder hemiplegia type of combination of sense and moving, merely sense accident and other lacunar syndromes. The risk factors of diseases were hypertension, diabetes and smoking.
Cerebral Infarction ; Brain Infarction ; diagnosis

Humans ; Myocardial Infarction ; diagnosis

Herein, we report on a case of primary torsion of the greater omentum with segmental infarction, which should provide useful information for the preoperative diagnosis of future such cases. Primary torsion of the greater omentum with omental infarction is a rare condition. There are only a few radiological reports of secondary torsion of the greater omentum caused by the hernial sac. During surgical exploration, infarction of the greater omentum was identified, due to the observation of omental torsion without any underlying cause. We describe a patient with characteristic computed tomography (CT) findings of primary omental torsion with segmental infarction, which correlated with the operative and pathologic results.
Diagnosis ; Humans ; Infarction* ; Omentum*

This paper introduces the troponin, a new gold standard for diagnosis of myocardial infarction and role of troponin in other diseases. The authors introduced also some new standards for diagnosis of the myocardial infarction such as cut-off value, isoenzyme CK-MB, GOT, myoglobin and LDH
Troponin ; Cardiomyopathies ; Infarction ; diagnosis ; Myocardial Infarction

101 male patient with diagnosis of acute myocardial infarction were involved in study. The participants have ages ranged from 42 to 85 (mean 62.13+/-8.17). Out of these patients, 4.9% had not a chest pain. 20% of patients had a severe angina and 40.6% of these had a mild angina. Time delay in admission of patients with severe chest pain was shortest (4.55 hours) and that of the patients with mild chest pain was longest (75.61 hours)
Myocardial Infarction ; Chest Pain ; diagnosis

101 men were diagnosed acute myocardial infarction. Of these patients 4.9% hadn't a chest pain, 20% had a severe angina, 40.6% a mild angina. The time delay of the patients with severe angina was the shortest (4.55h) while that of the patients with mild angina was the longest (75.61h)
Chest Pain ; Myocardial Infarction ; diagnosis

Humans ; Myocardial Infarction ; diagnosis ; Takotsubo Cardiomyopathy ; diagnosis

Contrary to ventricular mural thrombi, left atrial appendage thrombi are extremely rare in cerebral infarction correlated with acute myocardial infarction but they can be easily detected by transesophageal echocardiography(TEE). We expierienced a case of cerebral infarction which was suspected to be caused from the thrombi in left atrial appendage in a patient with acute myocardial infarction. The cerebral infarction was developed 2 days after myocardial infarction had been occurred and any source of the thrombi could not be detected except in left atrial appendage. The diagnosis was established by TEE and also aided by transthoracic echocardiography, brain computed tomography.
Atrial Appendage* ; Brain ; Cerebral Infarction* ; Diagnosis ; Echocardiography ; Humans ; Myocardial Infarction*

Perforation of the interventricular septum complicating acute myocardial infarction is uncommon. The condition was first described anatomically by Latham in 1845 and the first antemortem diagnosis was made by Brunn in 1923. In both cases, bedside catheterization utilizing a flow directed catheter detected a step up of O2 saturation at the ventricular level, compatible with ventricular septal defect, and two dimensional echocardiogram allowed direct visualization and localization of the postmyocardial infarction VSD. Finally cineventriculogram confirmed them.
Catheterization ; Catheters ; Diagnosis ; Heart Septal Defects, Ventricular ; Infarction* ; Myocardial Infarction

The usual imaging findings of common splenic infarction are well known, while the findings for splenic infarctions in the accessory spleen or polysplenia are rare; these unusual imaging findings may make the diagnosis difficult. We report here on two patients who have complained of abdominal pain, and they were diagnosed as splenic infarction that developed in either the accessory spleen or as has having polysplenia. We can diagnose splenic infarction that unusually develops in the accessory spleen or polysplenia when we identify a round, hyperechoic, avascular solid mass on US, or when we identify a round, rim-enhancing, hypodense solid mass with adjacent inflammatory changes and a small amount of ascites on CT that is adjacent to the normal spleen or in one of splenules of polysplenia in the clinical settings of acute abdominal pain.
Abdominal Pain ; Ascites ; Child* ; Diagnosis ; Humans ; Infarction* ; Spleen* ; Splenic Infarction