We report a patient with a left anterior cerebral artery (ACA) infarct showing representational neglect. Representational neglect is some form of neglect to areas of imagined space. We believe that the description of our case gives further information about neglect syndrome and the functional asymmetry of the cerebral hemispheres because these observations are rare.
Anterior Cerebral Artery* ; Cerebrum ; Humans ; Infarction, Anterior Cerebral Artery*

No abstract available.
Anterior Cerebral Artery ; Cerebral Infarction ; Headache Disorders, Primary ; Infarction, Anterior Cerebral Artery ; Vasoconstriction

The accessory middle cerebral artery (MCA) is an anomalous vessel which arises from the anterior cerebral artery (ACA) and runs through the Sylvian fissure along with the normal MCA. Here we present a case of acute cerebral infarction in a patient with stenosis of the accessory MCA. The accessory MCA, which originated from the proximal A1 segment of the ACA, had severe focal stenosis in its proximal part and the ischemic lesions were in the frontal subcortical white matter. This case illustrates the anomalous vessel and its territory, the atheromatous vascular change, and the related ischemic insults.
Anterior Cerebral Artery ; Cerebral Infarction ; Constriction, Pathologic ; Glycosaminoglycans ; Humans ; Middle Cerebral Artery

Subfalcial herniation secondary to a unilateral supratentorial space occupying lesion is frequently seen on the brain CT scan, however, the development of distal anterior cerebral artery infarction following compression of the terminal branches of the pericallosal artery against the falx during the period of subfalcial herniation is uncommon. For the last three years, we have experienced three cases of distal anterior cerebral artery infarction caused by subfalcial herniation on the brain CT scan. All patitnts had acute subdural hematomas resulting in subfalcial herniation on the first CT scan and subsequently developed focal infarctions in the ipsilateral paracentral lobule. The patients did not improve clinically and died several days after operation. The development of such infarction may reflect severe cerebral injury and portend a poor clinical outcome.
Anterior Cerebral Artery* ; Arteries ; Brain ; Hematoma, Subdural, Acute ; Humans ; Infarction* ; Infarction, Anterior Cerebral Artery ; Tomography, X-Ray Computed

Anterior cerebral artery(ACA) dissecting aneurysms are extremely rare and optimal treatment remains unclear. The majority of cases manifest as cerebral infarction or as intracranial bleeding. The authors report two cases of ACA dissecting aneurysm, one with a large partially thrombosed gradually growing aneurysm and one with a cerebral infarction in the ACA territory. The patient with a large aneurysm was treated by trapping the aneurysm, and the patient with infarction by conservative management. We report on two cases of dissecting aneurysm of the ACA and include a review of pertinent literature.
Aneurysm ; Aneurysm, Dissecting* ; Anterior Cerebral Artery* ; Cerebral Infarction ; Hemorrhage ; Humans ; Infarction

The spontaneous dissecting aneurysm of the anterior cerebral artery is very rare and shows difference in nature and clinical presentation. The authors present 2 cases of spontaneous dissecting aneurysms localized to the anterior cerebral artery. Both patients experienced severe headache and ischemic symptoms. The lesions were repaired successfully with wrapping and clipping.
Aneurysm, Dissecting* ; Anterior Cerebral Artery* ; Cerebral Angiography ; Cerebral Infarction ; Headache ; Humans

Previous reports on the lesions causing stroke-related periodic limb movement in sleep (PLMS) have involved subcortical structures such as the basal ganglia/corona radiata or pons. We report a case of an 81-year-old female patient who presented with bilateral, right-side-predominant PLMS that developed after a left cortical infarction. The right-side PLMS may be attributable to the loss of cortical inhibition following a pyramidal tract lesion, while the left-side PLMS may be caused by activation of the contralateral motor cortex due to dysfunction of interhemispheric inhibition.
Aged, 80 and over ; Anterior Cerebral Artery ; Cerebral Infarction ; Extremities ; Female ; Humans ; Infarction ; Infarction, Anterior Cerebral Artery ; Motor Cortex ; Nocturnal Myoclonus Syndrome ; Pons ; Pyramidal Tracts

Akinetic mutism is a clinical syndrome in which the patient is unable to speak (mutism) or move (akinesia). Various brain lesions can induce akinetic mutism. We attended a 71-year-old woman who presented with akinetic mutism caused by bilateral anterior cerebral artery infarction. The patient improved after the administration of levodopa com-bined with carbidopa, in response to visual and verbal stimuli. Increased verbal output and spontaneous motor activities were also noted. Levodopa may be helpful to the treatment of akinetic mutism.
Aged ; Akinetic Mutism* ; Anterior Cerebral Artery* ; Brain ; Carbidopa ; Female ; Humans ; Infarction, Anterior Cerebral Artery* ; Levodopa* ; Motor Activity

An unanesthetized cat model of acute focal cerebral ischemia has been establishes by the technique of transorbital snare ligature for middle cerebral artery (MCA) occlusion, The model was used to investigate patterns of changes of regional cerebral blood flow (rCBF) for up to 16 hours following MCA occlusion by the hydrogen clearance technique and to explore the correlation among microregional blood flow changes, neurological deficit, and pathological changes including size of infarct and severity of brain edema. The animals were divided into 2 groups according to size of infarct that was identified by 2% triphenyl tetrazolium chloride solution stain. The results were as follows : 1) Infarct larger than 10% of coronal section surface of the ipsilateral cerebral hemisphere was found in 18 cats( Group A), and smaller infarct was found in the remaining 7 cats (Group B). Between these 2 groups, there was a statistically significant difference in the average rCBF value of ipsilateral MCA territory during 16 hours of ischemia (Group A:6.5ml/100g/min, Group B:32.6ml/100g/min) (P<0.01). 2) Increasing grade of contralateral paralysis correlated well with decreasing rCBF in MCA territory of occlusion side and all animals showing complete paralysis belonged to Group A with their average rCBF from ipsilateral MCA territory below 10.0ml/100g/min. 3) Increasing grade of cerebral hemispheric swelling was directly proportional to increasing grade of paralysis and there was also a significant difference in hemispheric swelling between Group A and B, Correlation between grades of infarct size and severity of paralysis was not evident. 4) There patterns of charges of rCBF were observed :In 15 cats of Pattern I, MCA occlusion caused persistent severe ischemia, measuring less than 16-17ml/100g/min(average rCBF:6.28ml/100g/min), to produce large infarct, pronounced paralysis, and severe histological damage(Group A). 5) In 7 cats of Pattern I, persistent mild to moderate ischemia, maintaining more than 23-24ml/100g/min(average rCBF:32.6ml/100g/min), was noted in association with much smaller infarct and milder hemiparesis(Group B). 6) In the remaining 3 cats of Pattern I, MCA occlusion caused immediate severe ischemia followed by early postischemic hyperemia and death during 8 to 14 hours after MCA occlusion owing to marked brain swelling and transtentorial herniation (Group A). 7) During the initial stage of ischemia, significant decrease in rCBF of the contralateral hemisphere was observed in both groups, however, in Group A, rCBF gradually increased to preocclusion level. Only Group B presented further decrease in rCBF suggesting the presence of interhemispheric diaschisis. Thus Group B appeared to take advantage of diaschisis during the late phase of infarct development as well as substantial collateral flow from the surrounding posterior cerebral and anterior cerebral artery territories.
Animals ; Anterior Cerebral Artery ; Brain Edema ; Brain Ischemia ; Cats ; Cerebral Infarction* ; Cerebrum ; Hydrogen ; Hyperemia ; Infarction, Middle Cerebral Artery* ; Ischemia ; Ligation ; Middle Cerebral Artery* ; Paralysis ; SNARE Proteins

Anterior cerebral artery (ACA) dissection with simultaneous subarachnoid hemorrhage (SAH) and cerebral infarction is very rare and its treatment continues to be debates. We present a case of simultaneous SAH and cerebral infarction caused by dissection of the ACA that was successfully treated by bypass surgery and an endovascular procedure.
Aneurysm, Dissecting ; Anterior Cerebral Artery ; Cerebral Infarction ; Endovascular Procedures ; Subarachnoid Hemorrhage