7.Five cases of abdominal sacral colpopexy for the vaginal vault prolapse after total hysterectomy.
Sang Joon CHOI ; Kyung LEE ; Young Gyul KIM ; Hyuk JUNG
Korean Journal of Obstetrics and Gynecology 1999;42(10):2377-2381
A rate com;lication of hysterectomy is complete prolapse and inversion of vagina. This can occur whether the hysterectomy was abdominal or vaginal and cystocele and/or enterocele is often associated with this condition. Traditionally, prolapse has been treated by surgery, the types of operation for prolapse are generally but not always, carried out through the vaginal rather than through the abdominal surgical route. We experienced five cases who had repair of posthysterectomy vaginal vault prolapse by transabdominal sacral colpopexy and report with brief review of literatures.
Cystocele
;
Hernia
;
Hysterectomy*
;
Pelvic Organ Prolapse*
;
Prolapse
;
Vagina
8.Letter form Editor.
Hoon HUR ; Hyo Jung PARK ; Hyuk Joon LEE
Journal of Clinical Nutrition 2017;9(2):37-37
No abstract available.
9.A clinical study of gallstone disease in elderly patients.
Young Joon KIM ; Min Hyuk LEE ; Kyung Bal HUR
Journal of the Korean Surgical Society 1992;43(1):67-77
No abstract available.
Aged*
;
Gallstones*
;
Humans
10.Fine structure and detoxification kinetics in kupffer cells after injection of endotoxin in rats.
Joon Hyuk CHOI ; Won Hee CHOI ; Tae Sook LEE
Yeungnam University Journal of Medicine 1993;10(2):313-337
The aim of this study was to clarify the role of Kupffer cells in the mechanism of endotoxin-induced liver injury. The study on fine structure of Kupffer cells was performed after the injection of endotoxin. The endotoxin(Escherichia soli lipopolysaccharide 026: B6, 1.5mg/100 g of body weight) was intraperitoneally injected in Sprague-Dewley rats. Animals were sacrificed at 1/4, 1/2, 1, 2, 4, 8, 16, 24, 72 and 120 hours after the injection of endotoxin. Livers were extirpated and processed to be examined by light and electron microscopy. The results obtained were summerized as follows: Early changes observed in liver after endotoxin injection included the increased number and hypertrophy of Kupffer cells, infiltration of neutrophils and presence of fibrin thrombi within the sinusoids. The coritinuous increase of the Kupffer cells in number with hypertrophy, congestion and infiltration of inflammatory cells within the sinusoids were observed. Hepatocytes showed* fatty change and occasional necrosis. At 72 hours the congestion decreased. At 120 hours the number of Kupffer cells was increased, but the morphology of Kupffer cells became similar to that of the control group. The numbers and sizes of primary and secondary lysosomes and amount of euchromatin of Kupffer cells increased. Swellings and increase in number of mitochondria, Golgi complex, smooth endoplasmic reticulum, rough endoplasmic reticulum were evident. Microthrombi were present within the sinusoids. The swelling of rough endoplasmic reticulum and mitochondria, decrease of glycogen particles, fatty change, hypoxic vacuoles, pyknotic nuclei and occasional necrosis were observed in hepatocytes. At 72 hours the number of secondary lysosomes in Kupffer cells decreased. At 120 hours the morphology of Kupffer cells became similar to that of the control group. According to these results, it was postulated that the endotoxin was initially taken up by pinocytosis into Kupffer cells and degraded in secondary lysosomes of activated Kupffer cells. Kupffer cells may play an important role in the defense mechanism of liver during endotoxemia. The dysfunction of Kupffer cells and ischemia by sinusoidal microthrombi may cause liver injury.
Animals
;
Endoplasmic Reticulum, Rough
;
Endoplasmic Reticulum, Smooth
;
Endotoxemia
;
Estrogens, Conjugated (USP)
;
Euchromatin
;
Fibrin
;
Glycogen
;
Golgi Apparatus
;
Hepatocytes
;
Hypertrophy
;
Ischemia
;
Kinetics*
;
Kupffer Cells*
;
Liver
;
Lysosomes
;
Microscopy, Electron
;
Mitochondria
;
Necrosis
;
Neutrophils
;
Pinocytosis
;
Rats*
;
Vacuoles