1.Methods and clinical applications of targeted temperature management
Neurology Asia 2015;20(4):325-333
Hypoxic/ischemic brain damage is well-known catastrophic injury. The specific treatment, socalled
neuroprotective therapy, aims to prevent or diminish this havoc damage. However, approved
neuroprotective therapy in clinical practice is limited. Targeted temperature management (TTM) shows
the most promising neuroprotective therapy. Moreover, TTM is also useful for intracranial pressure
(ICP) control. Many methods of TTM have been reported. TTM can apply to several clinical conditions
associated with hypoxic/ischemic brain injury or elevated intracranial pressure.
Hypoxia-Ischemia, Brain
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Hypoxia, Brain
2.Perinatal brain damage caused by cerebral hypoxia-ischemia.
Journal of the Korean Pediatric Society 1993;36(1):1-8
No abstract available.
Brain*
;
Hypoxia-Ischemia, Brain*
3.Management of Hypoxic-Ischemic Encephalopathy: Present and Future.
Korean Journal of Perinatology 2003;14(4):393-399
No abstract available.
Hypoxia-Ischemia, Brain*
4.Hypothermia Therapy in Neonatal Hypoxic Ischemic Encephalopathy.
Korean Journal of Perinatology 1999;10(4):447-452
No abstract available.
Hypothermia*
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Hypoxia-Ischemia, Brain*
5.Predictors of neurologic handicap in hypoxic ischemic encephalopathy.
Seung Tae KIM ; Gui Ran KIM ; Byung Hak LIM ; Sang Geel LEE ; Im Ju KANG
Journal of the Korean Pediatric Society 1991;34(4):473-479
No abstract available.
Hypoxia-Ischemia, Brain*
6.Hypoxic-Ischemic Encephalopathy Following Carotid Endarterectomy.
Journal of the Korean Neurological Association 2013;31(2):136-137
No abstract available.
Carotid Stenosis
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Endarterectomy, Carotid
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Hypoxia-Ischemia, Brain
7.Language development in first 3 years of life and early language screening scale.
Journal of the Korean Pediatric Society 1991;34(4):465-472
No abstract available.
Hypoxia-Ischemia, Brain
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Language Development*
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Mass Screening*
8.Diffusion-weighted MR Imaging of Hypoxic-Ischemic Encephalopathy.
Hye Young CHOI ; Dae Seob CHOI ; Jae Wook RYOO ; Jae Min CHO ; Eun Sook KO ; Tae Beom SHIN ; Jae Beom NA ; Nak Cheon CHOI
Journal of the Korean Society of Magnetic Resonance in Medicine 2008;12(1):49-54
PURPOSE: The purpose of this study was to determine the characteristics of hypoxic-ischemic encephalopathy (HIE) on diffusion-weighted imaging (DWI) and the role of DWI for the diagnosis of HIE. MATERIALS AND METHODS: Six patients with HIE underwent MRI including DWI. MR examinations were performed within 4 - 32 days (mean, 11.8 days) after hypoxic brain insult. We assessed the distribution of the lesions and compared the DWI and T2, FLAIR images for the subjective conspicuity of the lesions. RESULTS: In all patients, symmetrical hyperintense lesions were demonstrated in the bilateral basal ganglia on T2, FLAIR, and DWI. On ADC map image, the lesions were hypointense in four of six patients and isointense in other two patients. Lesion conspicuity on DWI was higher than on T2 and FLAIR images in four of six patients and similar in other two patients. For the involvement of the cortex and subcortical white matter, in five of six patients, bilateral symmetric hyperintense lesions were seen on T2, FLAIR, and DWI. Lesion conspicuity on DWI was higher than on T2 and FLAIR images in three of them and similar in other two patients. On ADC map image, the lesions showed hypointensity in three of five patients and isointensity in other two patients. For the involvement of the deep cerebral white matter, T2, FLAIR, and DWI showed bilateral symmetric hyperintense lesions in four of six patients. Among them, Lesion conspicuity on DWI was higher than on T2 and FLAIR images in only one patient. CONCLUSION: HIE is characterized by symmetrical hyperintense lesions in the bilateral basal ganglia, cerebral cortex, and white matter on DWI and the lesions are more conspicuously demonstrated on DWI than on T2 and FLAIR images.
Anoxia
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Basal Ganglia
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Brain
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Cerebral Cortex
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Humans
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Hypoxia-Ischemia, Brain