Renal hypertension is found to be the cause in 5-15ft of patients with hypertension, and may be vascular or parenchymal in nature. Renal studies in diagnostic technique and vascular reconstructive techniques now enable successful management in many patients with renal hypertension. We experienced one case of renovascular hypertension due to renal vein hypoplasia. This rare renal venous hypertension has been diagnosed preoperatively and can be cured by nephrectomy, so we reported this case with brief review of the literature.
Humans ; Hypertension ; Hypertension, Renal ; Hypertension, Renovascular* ; Nephrectomy ; Renal Veins*

Renal artery embolism is an uncommon event, but a correct diagnosis and appropriate treatment are often delayed. We report a case in which viability was sustained by surgical intervention 18 hours after an episode of embolism that produced hypertension.
Diagnosis ; Embolism* ; Hypertension ; Hypertension, Renovascular* ; Renal Artery*

No abstract available.
Aneurysm* ; Arteriovenous Fistula* ; Hypertension, Renovascular* ; Renal Artery*

Atherosclerotic renovascular hypertension is a form of secondary hypertension due to renal artery stenosis. After the introduction of medical therapy such as with statins and angiotensin blocking agents, it has been considered a very slowly progressive disease. In the 1990s, surgical methods were compared to radiological intervention and showed no additional benefits. Recent clinical data also demonstrate that in cases of relatively stable atherosclerotic renovascular disease, medical therapy is as effective as other interventions with regard to patient outcomes. In this paper the recent clinical outcomes are reviewed.
Angiotensins ; Atherosclerosis ; Humans ; Hypertension ; Hypertension, Renovascular ; Renal Artery Obstruction

Renal artery stenosis is a cause of secondary hypertension which can be cured by surgical or radiological intervention such as percutaneous transluminal renal artery stent placement. In this case we present a subcapsular hematoma of the kidney, a complication following percutaneous transluminal stent placement in the renal artery. Reperfusion injury to the kidney may be a possible mechanism of subcapsular hematoma of the kidney. Long standing severe renal artery stenosis and high pre- and post- procedure pressure gradient might contribute to the complication.
Angioplasty ; Hematoma* ; Hypertension ; Hypertension, Renovascular ; Kidney ; Renal Artery Obstruction* ; Renal Artery* ; Reperfusion Injury* ; Reperfusion* ; Stents

Renal artery stenosis is a major cause of renovascular hypertension and the most common cause of treatable secondary hypertension. There are several methods to treat renal artery stenosis, including surgery, percutaneous transluminal renal angioplasty(PTRA), and renal artery stenting(RAS). But, renal artery embolization can be tried in atherosclerotic stenosis, multiple stenosis, microaneurysm, and stenosis difficult to try PTRA or RAS. We report a case of renovascular hypertension in a 14-year-old female who had multiple segmental renal artery stenosis. Hypertension was controlled by renal ablation therapy with renal artery embolization.
Adolescent ; Constriction, Pathologic ; Female ; Humans ; Hypertension ; Hypertension, Renovascular* ; Renal Artery Obstruction ; Renal Artery*

Renovascular hypertension caused by renal artery stenosis is an uncommon but curative cause of hypertension in children. We report a case of recurrent severe hypertension caused by renovascular hypertension. After recurrence of hypertension after redo percutaneous transluminal renal angioplasty, the blood pressure was finally controlled by renal autotransplantation. This case demonstrates the importance of considering renovascular hypertension as a cause of severe hypertension in children. Also, renal autotransplantation should be considered as a viable treatment option for treatment of renovascular hypertension that is recurrent after renal angioplasty.
Angioplasty ; Blood Pressure ; Child ; Humans ; Hypertension ; Hypertension, Renovascular* ; Recurrence ; Renal Artery ; Renal Artery Obstruction ; Transplantation*

In hypertensive patients it is very important to detect renal artery stenosis or pheochromocytoma, since both diseases are curable causes of hypertension. However, renal artery stenosis can be induced by pheochromocytoma, when the diagnosis of the two simultaneous disease is very difficult. We experienced two cases of pheochromocytoma presented as renal artery stenosis. Pheochromcytoma was overlooked when renovascular hypertension was diagnosed. Pheochromocytoma was found during surgery in one patient and after angioplasty in the other. In both cases, BP returned to normal after surgical removal of pheochromocytoma without repair of the stenosis. Prevention of ineffective and unnecessary renal artery angioplasty of surgery requires knowledge of this unusal association between pheochromocytoma and renal artery stenosis and a high degree of clinical alertness for pheochromocytoma.
Angioplasty ; Constriction, Pathologic ; Diagnosis ; Humans ; Hypertension ; Hypertension, Renovascular ; Pheochromocytoma* ; Renal Artery Obstruction* ; Renal Artery*

Renovascular hypertension is the leading cause of surgically curable arterial hypertension. With the marked advancement in the technique of vascular surgery, renal revascularization is preferred to nephrectomy. It is used for the recovery of impaired function or the prevention of renal failure and the control of hypertension. We report a case of renovascular hypertension with the nonfunction of the left kidney due to complete renal artery obstruction, treated successfully with an aortorenal bypass graft.
Hypertension ; Hypertension, Renovascular* ; Kidney ; Nephrectomy ; Renal Artery Obstruction ; Renal Insufficiency ; Saphenous Vein* ; Transplants*

It has been suggested that arginine vasopressin is involved in the acute, but not chronic, regulation of blood pressure. It is still debatable on the role of arginine vasopressin in the development and maintenance of high blood pressure, especially in renal hypertension. This study was performed to examine antidiuretic and renal hemodynamic effects of extremely low doses of arginine vasopressin and the modification of the effects in the early phase of two-kidney one clip Goldblatt hypertensive rabbits. Very low doses(up to 10-20mole/kg/min) of intrarenal arginine vasopressin induced decreases in urine volume, free water clearance, glomerular filteration rate and renal plasma flow(CPAH). The renal effects of arginine vasopressin were dose-dependent. These data indicate that the sensitivity of the kidney to decrease urine volume in response to arginine vasopressin is at least three orders of magnitude higher than previously reported. The renal effects of arginine vasopressin was significantly attenuated by the pretreatment of V2-receptor antagonist. The antidiuretic and renal hemodynamic effects elicited by very low doses of intrarenal arginine vasopressin were suppressed in the contralateral unclipped kidney of two-kidney, one clip Goldblatt hypertensive rabbits. These data suggest that the renal functions can be regulated normally by an extremely low concentration of plasma arginine vasopressin and the regulatory mechanism controlled by the ultralow plasma levels of arginine vasopressin is suppressed in the early phase of two-kidney, one clip Goldblatt hypertension.
Arginine Vasopressin* ; Arginine* ; Blood Pressure ; Hemodynamics ; Hypertension ; Hypertension, Renal ; Hypertension, Renovascular ; Kidney ; Plasma ; Rabbits ; Water