1.Two Cases of Severe Hyperkalemia with Atypical Electrocardiographic Manifestations.
Sik LEE ; Min Hee LEE ; Kyung Pyo KANG ; Won KIM ; Sung Kwang PARK ; Sung Kyew KANG
Electrolytes & Blood Pressure 2005;3(1):52-54
No abstract available.
Electrocardiography*
;
Hyperkalemia*
2.Acute hyperkalemia induced by hyperglycemia in non-diabetic patient.
Sun Young PARK ; Tae Joon KIM ; Min Jung KIM
Korean Journal of Anesthesiology 2011;61(2):175-176
No abstract available.
Humans
;
Hyperglycemia
;
Hyperkalemia
3.Secondary Hyperkalemic Paralysis Mimicking Demyelinating Sensorimotor Polyneuropathy.
Tae Eun KIM ; Yeo Jeong KANG ; Jeong Ho PARK ; Sun Ah PARK ; Ki Bum SUNG ; Tae Kyeong LEE
Journal of the Korean Neurological Association 2012;30(4):355-357
No abstract available.
Hyperkalemia
;
Paralysis
;
Polyneuropathies
;
Spironolactone
4.2 Cases of Fatal Neurological Complications Following D.P.T. Vaccination.
Chul Ho KIM ; Kyung Hee YOO ; Myung Hi SHIN ; Jong Young LEE
Journal of the Korean Pediatric Society 1978;21(12):1154-1158
2 cases of fatal neurological complications following D.P.T. vaccination were presented with a brief review of literature. One case was a 6 mo. Old baby who developed symptoms 5 hrs. after his. 3rd D.P.T. inoculation and expired about 29 hrs. after vaccination. The other case was 5 mo. Old male who developed symptom 3 2/1 days after his 2nd D.P.T. incoulation and expired about 4 2/1 days after vaccination. The interesting findings in these 2 cases were hyperglycemia, severe acidosis, and hyperkalemia.
Acidosis
;
Humans
;
Hyperglycemia
;
Hyperkalemia
;
Male
;
Vaccination*
5.Hypokalemia and hyperkalemia.
Korean Journal of Pediatrics 2006;49(5):470-474
Hypokalemia and hyperkalemia are the most commonly encountered electrolyte abnormalities in hospitalized patients. Because untreated hypokalemia or hyperkalemia is associated with high morbidity and mortality, it is important to recognize and treat them immediately. Hypokalemia and hyperkalemia can result from disruptions in transcellular homeostasis or in the renal regulation of K+ excretion. Although the recognition is simple, appropriate management requires an understanding of normal K+ homeostasis and pathophysiology. In this article, normal K+ homeostasis, pathophysiology, diagnosis and management of hypokalemia and hypokalemia are discussed.
Diagnosis
;
Homeostasis
;
Humans
;
Hyperkalemia*
;
Hypokalemia*
;
Mortality
;
Potassium
7.Cardiac arrest from acute hyperkalemia during liver surgery: A case report.
Su Hyun LEE ; Ki Jun KIM ; Chul Ho CHANG ; Sang Baeg HEO
Korean Journal of Anesthesiology 2010;59(Suppl):S124-S127
We experienced a case of sudden onset of hyperkalemia during liver lobectomy and this was followed by ventricular tachycardia and cardiac arrest. The main cause of this fatality is assumed to be the wide range of surgical manipulation that induced reduced hepatic blood flow and ischemic necrosis of the hepatic cells. We report here on this case and we review the relevant medical literature.
Heart Arrest
;
Hepatocytes
;
Hyperkalemia
;
Liver
;
Necrosis
;
Tachycardia, Ventricular
8.Differance in Serum K+ Concentration after Injection of Succinylcholine in Non-burned Area in Electric Barn Patients.
Soon Ok SEONG ; Youn Sil KIM ; Chang Jae KWON
Korean Journal of Anesthesiology 1984;17(4):278-280
Transient hyperkalemia has been reported to occur in eletric burn patients following intravenous administration of succinylcholine. We have studied the origin of the elevated serum K+ concentration after injection of succinylcholine from both the non-burned area and from the burned area in electirc burn patients. Blood samples for serum K+ measurement were drawn 1,2,3,4,5 and 10 minutes after administration of succinylcholine from both the burned are and the non-burned area in 6 electric burn patients. The following results were obtained: 1) Serum K+ levels increased in the burned group in 3 minutes after injection of succinylcholine. 2) The serum K+ concentration of venous samples from the electric burn area were more increased than from the non-burned area injection of succinylcholine.
Administration, Intravenous
;
Burns
;
Burns, Electric
;
Humans
;
Hyperkalemia
;
Succinylcholine*
9.Change of Serum K+ Concentration after Injection of Succinylcholine in the Electric Burn Patient .
Sung Woo LEE ; Yeo Song CHO ; Chang Jae KWON
Korean Journal of Anesthesiology 1982;15(2):156-160
Transient hyperkalemia is well known to occur in man following intravenous administration of succinylcholine chloride. We studied the change of the serum potassium concentration after injection of succinylcholine in nonburn patients and electricburn patients, physical status 1 or 2 adopted by the American Society of Anesthesiologists. We compared two groups: Non-burn patient group(Group 1) as control, Electric burn patient group (Group 2) as experimental. The following results were obtained: 1) In non-burn patient group(Group 1) serum potassium concentration was slightly decreased in the 1st, 2nd, 3rd, 4th, 5th, 6th and 10th minute after succinylcholine injection compared with control, but nostatistical significance was noticed in this group. 2) In electric burn patient group (Group 2) serum potassium concentration was maximally increased compared with the controls. Statistical significance was noticed at the 2nd and 3rd minute after succinylcholine injection in this group.
Administration, Intravenous
;
Burns, Electric*
;
Humans
;
Hyperkalemia
;
Potassium
;
Succinylcholine*
10.Hyperkalemia from preoperative non-steroidal anti-inflammatory drugs and angiotensin II receptor blockers in patients with nephropathy.
Ho Kyung SONG ; Yeon JANG ; Jin Woo NAM ; Ju Hyun YOU
Korean Journal of Anesthesiology 2011;61(6):533-534
No abstract available.
Angiotensin II
;
Angiotensin Receptor Antagonists
;
Angiotensins
;
Hyperkalemia
;
Receptors, Angiotensin