1.Two Cases of Severe Hyperkalemia with Atypical Electrocardiographic Manifestations.
Sik LEE ; Min Hee LEE ; Kyung Pyo KANG ; Won KIM ; Sung Kwang PARK ; Sung Kyew KANG
Electrolytes & Blood Pressure 2005;3(1):52-54
No abstract available.
Electrocardiography*
;
Hyperkalemia*
2.Secondary Hyperkalemic Paralysis Mimicking Demyelinating Sensorimotor Polyneuropathy.
Tae Eun KIM ; Yeo Jeong KANG ; Jeong Ho PARK ; Sun Ah PARK ; Ki Bum SUNG ; Tae Kyeong LEE
Journal of the Korean Neurological Association 2012;30(4):355-357
No abstract available.
Hyperkalemia
;
Paralysis
;
Polyneuropathies
;
Spironolactone
3.Acute hyperkalemia induced by hyperglycemia in non-diabetic patient.
Sun Young PARK ; Tae Joon KIM ; Min Jung KIM
Korean Journal of Anesthesiology 2011;61(2):175-176
No abstract available.
Humans
;
Hyperglycemia
;
Hyperkalemia
4.2 Cases of Fatal Neurological Complications Following D.P.T. Vaccination.
Chul Ho KIM ; Kyung Hee YOO ; Myung Hi SHIN ; Jong Young LEE
Journal of the Korean Pediatric Society 1978;21(12):1154-1158
2 cases of fatal neurological complications following D.P.T. vaccination were presented with a brief review of literature. One case was a 6 mo. Old baby who developed symptoms 5 hrs. after his. 3rd D.P.T. inoculation and expired about 29 hrs. after vaccination. The other case was 5 mo. Old male who developed symptom 3 2/1 days after his 2nd D.P.T. incoulation and expired about 4 2/1 days after vaccination. The interesting findings in these 2 cases were hyperglycemia, severe acidosis, and hyperkalemia.
Acidosis
;
Humans
;
Hyperglycemia
;
Hyperkalemia
;
Male
;
Vaccination*
5.Hypokalemia and hyperkalemia.
Korean Journal of Pediatrics 2006;49(5):470-474
Hypokalemia and hyperkalemia are the most commonly encountered electrolyte abnormalities in hospitalized patients. Because untreated hypokalemia or hyperkalemia is associated with high morbidity and mortality, it is important to recognize and treat them immediately. Hypokalemia and hyperkalemia can result from disruptions in transcellular homeostasis or in the renal regulation of K+ excretion. Although the recognition is simple, appropriate management requires an understanding of normal K+ homeostasis and pathophysiology. In this article, normal K+ homeostasis, pathophysiology, diagnosis and management of hypokalemia and hypokalemia are discussed.
Diagnosis
;
Homeostasis
;
Humans
;
Hyperkalemia*
;
Hypokalemia*
;
Mortality
;
Potassium
6.Spontaneous Sinus Conversion of Permanent Atrial Fibrillation During Treatment of Hyperkalemia.
Ji Hyun YOON ; Da Hyun JUNG ; Seung Kyo PARK ; Ji Soo PARK ; Jong Youn KIM ; Pil Ki MIN ; Byung Kwon LEE ; Young Won YOON ; Bum Kee HONG ; Hyuck Moon KWON ; Se Joong RIM
Korean Circulation Journal 2012;42(1):65-68
Hyperkalemia is a common adverse effect of treatment for heart failure and is associated with high mortality and morbidity. The cardiac manifestations of hyperkalemia include various electrocardiogram changes. We describe a case of a 74-year-old woman with heart failure and permanent atrial fibrillation who reverted to normal sinus rhythm during recovery from hyperkalemia.
Aged
;
Atrial Fibrillation
;
Electrocardiography
;
Female
;
Heart Failure
;
Humans
;
Hyperkalemia
7.Hemolysis of irradiated leukoreduced red blood cells during rapid warming: An in vitro experimental study.
Sukyung LEE ; Sooho LEE ; Jong Eun OH ; Won Jung SHIN ; Won Ki MIN ; Mijeung GWAK
Journal of Dental Anesthesia and Pain Medicine 2015;15(4):229-233
BACKGROUND: Although water chambers are often used as surrogate blood-warming devices to facilitate rapid warming of red blood cells (RBCs), these cells may be damaged if overheated. Moreover, filtered and irradiated RBCs may be damaged during the warming process, resulting in excessive hemolysis and extracellular potassium release. METHODS: Using hand-held syringes, each unit of irradiated and leukocyte-filtered RBCs was rapidly passed through a water chamber set to different temperatures (baseline before blood warming, 50℃, 60℃, and 70℃). The resulting plasma potassium and free hemoglobin levels were then measured. RESULTS: Warming RBCs to 60℃ and 70℃ induced significant increases in free hemoglobin (median [interquartile ranges] = 60.5 mg/dl [34.9-101.4] and 570.2 mg/dl [115.6-2289.7], respectively). Potassium levels after warming to 70℃ (31.4 ± 7.6 mEq/L) were significantly higher compared with baseline (29.7 ± 7.1 mEq/L; P = 0.029). Potassium levels were significantly correlated with storage duration after warming to 50℃ and 60℃ (r = 0.450 and P = 0.001; r = 0.351 and P = 0.015, respectively). CONCLUSIONS: Rapid warming of irradiated leukoreduced RBCs to 50℃ may not further increase the extracellular release of hemoglobin or potassium. However, irradiated leukoreduced RBCs that have been in storage for long periods of time and contain higher levels of potassium should be infused with caution.
Erythrocytes*
;
Hemolysis*
;
Hyperkalemia
;
In Vitro Techniques*
;
Plasma
;
Potassium
;
Syringes
;
Water
8.Effects of Interdialytic Interval and Potassium(K) Removal via Dialvsis on the Plasma Potassium Concentration in Maintenance Hemodialysis.
Jin Yeong KIM ; Kwang Ho ROH ; Joon Ho RYU ; Joong Don MOON ; Kyung Won LEE ; Il Gyu PARK ; Ho Jung KIM
Korean Journal of Nephrology 2000;19(1):106-111
To evaluate potassium(K) homeostasis during in-terdialytic and dialytic phases in chronic hemodialysis patients, we analyzed pre- and post- dialysis plasma K concentration(n=28) over n week with an interdialytic interval of 7Zhrs, 48hrs(l), and 48hrs(II), respectively, and the quantity of total dialytic K removal via dialysate. The predialysis plasma K at 72h interval(prePK72h: 4.89+/-0.17mEq/L) was significantly higher than those at 48h interval(prePK48h-I: 4.57+/-0.15mEq/L, and prePK48h-II: 4.40+/-15mEq/L) (p=0.000, p=0.000). 10.7% in prePK72h were categorized into severe hyperkalemia more than 6.0mEq/L, but none in prePK48h-I, II(p=0.000, p=0.000). In contrast no difference between 72-h and 42-h intervals was found in the postdialysis plasma K(postPK72h: 3.59+/-0.07 vs postPK48h-I : 3.530+/-08mEq/L, p>0.05) and in the quantity of total dialytic K removal via dialysate(delta Ktota172h : 74+/-2.6 vs delta Ktota148h-I:71+/-2.2mEq, p>0.05). On approach to this with two-compartment model, there was significant difference in dialytic K removal from ECF(delta Kecf72h:22.2+/-1.6 vs delta Kecf48h-I:17.7+/-1.6mEq, p<0.01), but not in that from ICF(delta Kicf72h:51.6+/-3.1 vs delta Kicf48h-I: 53.5+/-2.7mEq, p>0.05). In all 28 patients, age, sex and body weight were not correlated with either pre- and post- plasma K levels or total K removal per kg body weight. In conclusion, the majority of dialytic K removal is from the replenishment of the ICF potassium and it has rather constant feature in that there was no autoregulatory increment even with the higher predialysis plasma K concentration. So the plasma K concentration on chronic maintenace hemodialysis is more dependent on the potassium gain during interdialytic phase than the potassium removal during dialytic phase. Also it is reasonable to restrict dietary K intake and apply K-exalate orientating to the interdialytic phase of 72hrs because severe hyperkalemia is rare in that of 48hrs.
Body Weight
;
Dialysis
;
Homeostasis
;
Humans
;
Hyperkalemia
;
Plasma*
;
Potassium*
;
Renal Dialysis*
9.Hyperkalemia from preoperative non-steroidal anti-inflammatory drugs and angiotensin II receptor blockers in patients with nephropathy.
Ho Kyung SONG ; Yeon JANG ; Jin Woo NAM ; Ju Hyun YOU
Korean Journal of Anesthesiology 2011;61(6):533-534
No abstract available.
Angiotensin II
;
Angiotensin Receptor Antagonists
;
Angiotensins
;
Hyperkalemia
;
Receptors, Angiotensin
10.A Fatal Adverse Effect of Barbiturate Coma Therapy: Dyskalemia.
Hyun Mook KWON ; Jin Wook BAEK ; Sang Pyung LEE ; Jae Ik CHO
Korean Journal of Neurotrauma 2016;12(2):156-158
The management guideline for traumatic brain injury (TBI) recommends high-dose barbiturate therapy to control increased intracranial pressure refractory to other therapeutic options. High-dose barbiturate therapy, however, may cause many severe side effects; the commonly recognized ones include hypotension, immunosuppression, hepatic dysfunction, renal dysfunction, and prolonged decrease of cortical activity. Meanwhile, dyskalemia remains relatively uncommon. In this study, we report the case of a hypokalemic patient with severe rebound hyperkalemia, which occurred as a result of barbiturate coma therapy administered for TBI treatment.
Brain Injuries
;
Coma*
;
Humans
;
Hyperkalemia
;
Hypokalemia
;
Hypotension
;
Immunosuppression
;
Intracranial Pressure