No abstract available.
Electrocardiography* ; Hyperkalemia*

No abstract available.
Humans ; Hyperglycemia ; Hyperkalemia

No abstract available.
Hyperkalemia ; Paralysis ; Polyneuropathies ; Spironolactone

Hypokalemia and hyperkalemia are the most commonly encountered electrolyte abnormalities in hospitalized patients. Because untreated hypokalemia or hyperkalemia is associated with high morbidity and mortality, it is important to recognize and treat them immediately. Hypokalemia and hyperkalemia can result from disruptions in transcellular homeostasis or in the renal regulation of K+ excretion. Although the recognition is simple, appropriate management requires an understanding of normal K+ homeostasis and pathophysiology. In this article, normal K+ homeostasis, pathophysiology, diagnosis and management of hypokalemia and hypokalemia are discussed.
Diagnosis ; Homeostasis ; Humans ; Hyperkalemia* ; Hypokalemia* ; Mortality ; Potassium

2 cases of fatal neurological complications following D.P.T. vaccination were presented with a brief review of literature. One case was a 6 mo. Old baby who developed symptoms 5 hrs. after his. 3rd D.P.T. inoculation and expired about 29 hrs. after vaccination. The other case was 5 mo. Old male who developed symptom 3 2/1 days after his 2nd D.P.T. incoulation and expired about 4 2/1 days after vaccination. The interesting findings in these 2 cases were hyperglycemia, severe acidosis, and hyperkalemia.
Acidosis ; Humans ; Hyperglycemia ; Hyperkalemia ; Male ; Vaccination*

Female ; Humans ; Hyperkalemia ; therapy ; Infant, Newborn ; Male ; Peritoneal Dialysis ; instrumentation

Aged ; Electrocardiography ; Female ; Humans ; Hyperkalemia ; complications ; physiopathology ; Myocardial Ischemia ; etiology

No abstract available.
Angiotensin II ; Angiotensin Receptor Antagonists ; Angiotensins ; Hyperkalemia ; Receptors, Angiotensin

Transient hyperkalemia is well known to occur in man following intravenous administration of succinylcholine chloride. To study on change of the serum level of K+, Na+ & Cl- after injection of succinylcholine in healthy adults, physical status 1 or 2 adopted by the American society of Anesthesiologists, we studied in two groups: pentothal sodium-succinylcholine injection group (group 1) as control, flaxedil-pentothal sodium-succinylcholine injetion group(group 2) as experimental. The following results were obtained: 1. Serum K+ was slightly increased in both groups, 2mEq/L at 3rd minute in control group, 2mEq/L at 1st minute in experimental group after succinylcholine injection, but no statistical significance was noticed in either group. 2. Serum Na+ was decreased 3 mEq/L immediately after pentothal sodium injection and then sliyhtly increased until 10th minute in control group. In experimental group lower level than control was observed until 15th minute, with the maximum decrease of 7mEq/L at 5th minute. But no statistical significance was noticed in either group. 3. Serum Cl was slightly decreased until 10th minute with the maximum decrease of 4mEq/L at 5th minute in control group. In experimental group, it was slightly increased until 5th minute. But no significance was noticed in either group.
Administration, Intravenous ; Adult ; Humans ; Hyperkalemia ; Sodium ; Succinylcholine ; Thiopental

To evaluate potassium(K) homeostasis during in-terdialytic and dialytic phases in chronic hemodialysis patients, we analyzed pre- and post- dialysis plasma K concentration(n=28) over n week with an interdialytic interval of 7Zhrs, 48hrs(l), and 48hrs(II), respectively, and the quantity of total dialytic K removal via dialysate. The predialysis plasma K at 72h interval(prePK72h: 4.89+/-0.17mEq/L) was significantly higher than those at 48h interval(prePK48h-I: 4.57+/-0.15mEq/L, and prePK48h-II: 4.40+/-15mEq/L) (p=0.000, p=0.000). 10.7% in prePK72h were categorized into severe hyperkalemia more than 6.0mEq/L, but none in prePK48h-I, II(p=0.000, p=0.000). In contrast no difference between 72-h and 42-h intervals was found in the postdialysis plasma K(postPK72h: 3.59+/-0.07 vs postPK48h-I : 3.530+/-08mEq/L, p>0.05) and in the quantity of total dialytic K removal via dialysate(delta Ktota172h : 74+/-2.6 vs delta Ktota148h-I:71+/-2.2mEq, p>0.05). On approach to this with two-compartment model, there was significant difference in dialytic K removal from ECF(delta Kecf72h:22.2+/-1.6 vs delta Kecf48h-I:17.7+/-1.6mEq, p<0.01), but not in that from ICF(delta Kicf72h:51.6+/-3.1 vs delta Kicf48h-I: 53.5+/-2.7mEq, p>0.05). In all 28 patients, age, sex and body weight were not correlated with either pre- and post- plasma K levels or total K removal per kg body weight. In conclusion, the majority of dialytic K removal is from the replenishment of the ICF potassium and it has rather constant feature in that there was no autoregulatory increment even with the higher predialysis plasma K concentration. So the plasma K concentration on chronic maintenace hemodialysis is more dependent on the potassium gain during interdialytic phase than the potassium removal during dialytic phase. Also it is reasonable to restrict dietary K intake and apply K-exalate orientating to the interdialytic phase of 72hrs because severe hyperkalemia is rare in that of 48hrs.
Body Weight ; Dialysis ; Homeostasis ; Humans ; Hyperkalemia ; Plasma* ; Potassium* ; Renal Dialysis*