No abstract available.
Asthma*

No abstract available.

No abstract available.
Angioedema* ; Angioedemas, Hereditary* ; Complement C1 Inhibitor Protein* ; Complement C1s*

No abstract available.
Antibody Formation* ; Chickenpox* ; Child* ; Female ; Herpesvirus 3, Human* ; Humans ; Immunoblotting* ; Immunoglobulin G* ; Pregnant Women*

No abstract available.
Chlamydia trachomatis* ; Chlamydia* ; Immunoglobulin A* ; Immunoglobulin G* ; Immunoglobulin M*

Giant lymphnode hyperplasias are an uncommon entity. They are difficult to image on plain film. The CTapearance of this condition has been described inrecent radiologic literature. In this case report, combination of ultrasonogram and CT permitted differentiation of other space occupying lesions from giant retroperitoneallymphnode hyperplasia.
Hyperplasia ; Ultrasonography

The ulnar nerve entrapment at the wrist is usually caused by carpal ganglion, occupational neuritis, ulnar artery disease, fractures of the carpal bones, tumors, rheumatioid arthritis, etc. The ganglion is the most common cause of the distal ulnar nerve entrapment. A fort-two years old woman complained of insidious motor weakness of the left hand. The electromyogram revealed distal ulnar nerve palsy. On examination, her sensibility of affected hand was normal; there were no Tinel's sign and palpable mass on the Guyon's canal and palm; there was obvious wasting of all the interossei. On surgical exploration, the deep branch of the ulnar nerve was compressed by a ganglion at the mid-palmar space, not in the Guyon's canal. Four months after removal, the clawing of the 4th and 5th fingers disappeared, and the pinch power of the left hand recovered normally seventeen months later.
Animals ; Arteries ; Arthritis ; Carpal Bones ; Female ; Fingers ; Ganglion Cysts ; Hand ; Hoof and Claw ; Humans ; Paralysis ; Ulnar Nerve Compression Syndromes ; Ulnar Nerve ; Ulnar Neuropathies

During recent 3 years, the authors had treated surgically 31 patients who have had old nerve injuries. 19 patients(21 nerves) of them could be followed for more than 1 year after operation. Of these patients, children were 5 and adults were 14. The elapsed time from the injury were from 3 weeks to 20 months(18 cases under 6months and 3 cases above 6 months). The methods of operation were epineural suture(4 cases), grouped interfascicular suture(8 cases), neurolysis(8cases) and nerve graft(1 case). There were satisfactory or good results in 9 nerves of the 21 old nerve injuries. In 4 of 5 children and 5 of 16 adults, good results were obtained. There were 2, 6 satisfactory or good results in 4 epineural sutures and 8 grouped interfascicular sutures and all poor results in 8 neurolyses. One case treated with nerve graft with sural nerve was showed good result. 9 cases of 18 old injuried nerves under 6 months from injury were good results. All 3 cases over 6 months from injury were poor results.
Adult ; Child ; Humans ; Sural Nerve ; Sutures ; Transplants

To evaulate the possible pathogenetic significance of allergen-specific IgA antibody in respiratory secretion from asthmatics, we measured house dust mite(HDM)-specific IgA antibody in 3% saline-induced sputum from 23 HDM-sensitive asthmatics, 4 atopic asthmatics without mite-sensitivity, 6 non-atopic asthmatics, and 13 non-atopic, non-asthmatic controls (including 6 non-atopic healthy controls, 4 patients with chronic bronchitis, and 3 patients with rheumatoid arthritis) by ELISA. We also measured HDM-specific IgA antibody in serum and numbers of eosinophils in sputum. 1) Levels of HDM-specific IgA antibody in sputum from mite-sensitive asthmatics were significantly higher than those from non-atopic, non-asthmatic controls and non-atopic asthmatics(p<0.05). Levels of HDM-specific IgA antibody in sputum from atopic asthmatics without mite-sensitivity were significantly higher than those from non-atopic, non-asthmatic controls (p<0.05), however HDM-specific IgA/albumin raito was not significantly different between two groups (p>0.05). 2) The ratio of HDM-specific IgA antibody to albumin in sputum was not significantly different in mite-sensitive asthmatics with sputum eosinophila (> or = 5% of 200 counted leukocytes) and those without sputum eosinophilia (p>0.05). 3) The ratio of HDM-specific IgA to albumin in sputum from asthmatics was higher than that of serum. 4) There was no significant correlation of HDM-specific IgA/albumin ratios between serum and sputum (p>0.05). 5) When comparing sputum and saliva samples from 7 mite-sensitive asthmatics, levels of HDM-specific IgA antibody in sputum were significantly higher than those in saliva (p<0.05). In conclusion, HDM-specific IgA anti-body was increased in sputum from HDM-sensitive asthmatics, and it might be locally produced from bronchial mucosa. To evlauate the pathogenetic significance of allergen-specific IgA antibody in respiratory secretion from asthmatics, further studies might be needed.
Bronchitis, Chronic ; Dust* ; Enzyme-Linked Immunosorbent Assay ; Eosinophilia ; Eosinophils ; Humans ; Immunoglobulin A* ; Mucous Membrane ; Saliva ; Sputum*

No abstract available.
Cerebrospinal Fluid* ; Humans ; Polymerase Chain Reaction* ; Streptococcus pneumoniae* ; Streptococcus*