Premature constriction of the ductus arteriosus is rare, but it can occur during fetal life idiopathically or secondary to medications or structural lesions. Premature constriction of the ductus arteriosus can lead to progressive right heart dysfunction, heart failure, subsequent hydrops fetalis, and even fetal death. Herein, we describe a case of fetal ductus arteriosus constriction of unknown etiology with a severely enlarged and hypertrophied right ventricle, which resolved dramatically soon after birth.
Constriction* ; Ductus Arteriosus* ; Fetal Death ; Heart ; Heart Failure ; Heart Ventricles ; Hydrops Fetalis ; Parturition* ; Prenatal Diagnosis

No Abstract available.
Fetal Heart*

No abstract available.
Female ; Fetal Heart* ; Heart Rate, Fetal* ; Pregnancy

OBJECTIVE: We aim to analyze each variable affecting FHR to build objective decision basis using canonical correlation analysis METHODS: The sixty four hundred and fifty five cases of NST from 1988 to 1997 at Hanyang University hospital were collected. We used FHR interpretation softwares, HYFM-I & II those were already developed by author, for extraction of each variable of FHR. We classified the variables into 2 groups, F.A.M(FHR, Amplitude, Mean minute range) and N.S.F(NST weeks, Signal loss, Fetal movement) groups. The Canonical correlation was compared between each variable and group. RESULTS: The canonical correlation between F.A.M & N.S.F are as follows; r=0.40 (p=0.001) in preterm, r=0.34 (p=0.001) in term, and r=0.41 (p=0.004) in postterm. The high linear dependency is shown as r=0.79 (p=0.013) before 23 weeks, the irregular changes shown from 23 weeks to 30 weeks (r=0.47 in 23-24 weeks, r=0.29 in 25-26 weeks, r=0.56 in 27-28 weeks, r=0.24 in 29-30 weeks) and the stable relative changes shown (about r=0.3-0.4) after 30 weeks (r=0.32 in 31-32 weeks, r=0.33 in 33-34 weeks, r=0.37 in 35-36, r=0.33 in 37-38 weeks, r=0.37 in 39-40 weeks, r=0.35 after 41 weeks) (p<0.02). In the linear correlation of FHR, two indicies of FHR variables, AMP and MMR pertaining to N.S.F., FHR(r) is 0.06, AMP and MMR seem to be identical as r=0.30 and then the canonization in term and postterm[FHR(r)=-0.27, AMR(r)=0.23, and MMR(r)=0.35]lies in the regular pattern. Moreover, there is rather a linear correlation between F.A.M and the gestational weeks in preterm and it is changed into linearity similar to zero in term and postterm pregnancies[term(r)=0.01, postterm(r)=0.06] CONCLUSIONS: This study shows the linearity of FHR and the variable which are based on the gestational weeks organically, comprehensibly, and quantitatively. It would be utilized as the fundamental standard of the linearity. The changes from the irregular linear correlation to the regular pattern according to advancing gestational weeks means the changes from the immature and transitional level to the mature level. The closer investigation of the linear and
Female ; Fetal Heart* ; Heart Rate, Fetal* ; Pregnancy

No abstract available.
Female ; Fetal Heart* ; Fetal Movement* ; Heart Rate, Fetal* ; Music* ; Pregnancy

The purpose of this pictorial review was to describe various echocardiographic techniques that can be used for the functional assessment of the fetal heart. The systolic and diastolic assessments of the fetal heart are presented separately, with an emphasis on 2-dimensional Doppler methods and an overview of new technologies. The aim of this summary was to review the tools that can be used by the echocardiographer, and on that basis, to systematize the process of performing a functional assessment.
Echocardiography ; Fetal Heart

Fetal Heart ; Heart Failure ; Heart Rate, Fetal ; Humans

We report a case of premature constriction of the fetal ductus arteriosus following maternal ingestion of a cyclooxygenase-2 (COX-2) inhibitor at 37 weeks' gestation. Fetal sonography at 38+2 weeks' gestation revealed tricuspid regurgitation, absent transpulmonary valve flow, right heart enlargement, and pericardial effusion. An immediate delivery resulted in a good postnatal outcome with dramatic improvement in the clinical and echocardiographic findings. Maternal exposure to Non-steroidal anti-inflammatory drugs (NSAIDs), especially late in gestation, can cause premature constriction of the ductus arteriosus, heart failure, and fetal death. Therefore, the use of NSAIDs late in gestation should be considered in limited cases with close fetal heart monitoring.
Anti-Inflammatory Agents, Non-Steroidal ; Cardiomegaly ; Constriction ; Cyclooxygenase 2 ; Ductus Arteriosus ; Eating ; Female ; Fetal Death ; Fetal Heart ; Heart ; Heart Failure ; Maternal Exposure ; Pericardial Effusion ; Pregnancy ; Sulfonamides ; Tricuspid Valve Insufficiency

No abstract available.
Female ; Fetal Heart* ; Heart Rate, Fetal* ; Pregnancy ; Telemetry*

No abstract available.
Female ; Fetal Heart* ; Hearing* ; Heart Rate, Fetal* ; Pregnancy