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RESULTSFive patients all presented with subacute onset with a history of direct exposure to 1,2-DCE. Lumbar cerebrospinal fluid pressures were all increased in 5 patients. All 5 patients had obvious intracranial hypertension. Liver and kidney function had no obvious abnormalities; Cranial MRI showed T1WI low signal and T2WI high signal in bilateral hemispheric white matter, cerebellar dentate nucleus and globus pallidus. After the treatment of dehydrating agent, glucocorticoid and supportive treatment, four patients were clearly improved, and one patient had cerebral hernia formation.

CONCLUSIONThe main neurological clinical features in patients with 1,2-DEC poisoning is obvious intracranial hypertension. The prognosis is usually good with early and long term use of glucocorticoids and dehydrating agent in poisoning patients.

Adult ; Ethylene Dichlorides ; poisoning ; Female ; Humans ; Magnetic Resonance Imaging ; Male ; Neurotoxicity Syndromes ; diagnosis ; therapy ; Young Adult

10.Experience of treatment of subacute encephalopathy induced by 1, 2-dichloroethane poisoning.

Yuan-lin ZHOU ; Wei-jun HONG ; Shao-fa KE

Chinese Journal of Industrial Hygiene and Occupational Diseases 2009;27(4):253-254

1.One Death Case Caused by Subacute Poisoning of 1,2-Dichloroethane.

2.Successful rescue of a case with 1, 2-dichlorethane and cyclohexanone intoxication.

3.Two cases of occupational acute severe 1, 2-dichloroethane poisoning.

4.Therapy of encephalopathy induced by severe dichloroethane poisoning.

5.One case report of treating the sequelae of encephalopathy due to dichloroethane poisoning.

6.Investigation on subacute poisoning accident in dichlorethane construction site.

7.One case of ethylene dichloride poisoning with wrong diagnosis as epilepsy.

8.Discussion on the treatment of increased intracranial pressure caused by the occupational acute 1,2-dichloroethane poisoning.

9.Clinical and cranial MRI analysis on five cases of toxic encephalopathy induced by dichloroethane.

10.Experience of treatment of subacute encephalopathy induced by 1, 2-dichloroethane poisoning.

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