1.OBSERVATION OF THE SUPERIOR MESENTERIC ARTERY IN CHINESE ADULTS
Eryu CHEN ; Suchen CHANG ; Lishing WANG
Acta Anatomica Sinica 1953;0(01):-
The origin, branching and distribution of the superior mesenteric artery were observed on 100 Chinese adult cadavers. The average level of the origin of the superior mesenteric artery lay on the middle third of the body of the first lumbar vertebra. The distance between the origins of the superior mesenteric artery and the coeliac artery and the diameter of the superior mesenteric artery were measured. The superior mesenteric artery usually gave off two (47%) or three (39%) colic branches. The relationships of the origin of each colic branch, originating directly from the superior mesenteric artery, to the third part of duodenum or the head of pancreas and also to the origins of the intestinal arteries were observed. The extent of distribution of each colic branch was estimated. Most of the superior mesenteric arteries (82%) did not distribute so far as the left colic flexure. The pattern of branching of the ileocolic artery may be divided into four types. The symmetrical and ansal types were the prevailing types, they were present in 45.26% and 42.11% respectively. Nearly half of the appendicular arteries (45.56%) arose from the ileocolic artery, about 34.44% appendicular arteries arose from the ileocolic ansa. Usually the appendicular artery was the first branch of the ileocolic trunk. The superior mesenteric artery gave off 15.9 intestinal branches in average. In 76% cases, the inferior pancreatic-duodenal artery and the first intestinal artery originated from a common trunk and this trunk usually arose from the posterior aspect of the superior mesenteric artery. We found that in 13 cases the superior mesenteric artery gave off common hepatic artery or right hepatic artery.
2.The Zinc Nutriture of Teenagers in Wuhan
Qinghua ZHU ; Eryu WANG ; Liqun TIAN ; Xiping ZHANG ; Wei ZHANG
Acta Nutrimenta Sinica 1956;0(03):-
3.A hemodynamics model describing cardiac deficiency of qi(vital energy).
Wei YAO ; Guanghong DING ; Xueyong SHEN ; Jing YANG ; Eryu CHEN ; Ruishang DANG ; Huansheng CHEN ; Qi GU ; Shengzhang WANG ; Hu WEI
Journal of Biomedical Engineering 2002;19(1):53-56
Cardiac deficiency of qi(vital energy) is one of the main syndromes in terms of TCM (Traditional Chinese Medicine). Based on our analysis of blood-tissue fluid circulation, we set up a hemodynamics model describing cardiac deficiency of qi. The model's theoretical results can reflect the manifestations of cardiac deficiency of qi, and are identical to those of clinical experimental discoveries, therefore it has substantiated our theory-cell's abnormal physiological function caused by undernourishment of tissue fluid is the reason of qi-deficiency.
Animals
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Cardiovascular Diseases
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physiopathology
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Hemodynamics
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physiology
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Humans
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Medicine, Chinese Traditional
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Models, Cardiovascular
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Qi
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Rabbits
4.Advances in biotic and abiotic mutual promoting mechanism for chlorinated aliphatic hydrocarbons degradation.
Shuai LIU ; Tiantao ZHAO ; Zhilin XING ; Xu YANG ; Eryu WANG
Chinese Journal of Biotechnology 2018;34(4):510-524
Chlorinated aliphatic hydrocarbons (CAHs) with characteristics of high toxicity, biological accumulation and recalcitrance to degradation as well as carcinogenicity, teratogenesis and mutagenicity, are seriously harmful to human health and ecological environment. CAHs degradation depends on biotic and abiotic responses that exist diversified interactive effects, so it is important to clarify the mechanism of CAHs degradation via biotic and abiotic mutual promoting to significantly enhance the CAHs-contaminated site restoration. In this work, a series of pathways for CAHs degradation was first introduced and summarized as three means on reductive dechlorination, aerobic cometabolism and direct oxidation, and biotic and abiotic typical factors affecting CAHs degradation were concluded from these. Then, mechanisms of induced degradation and synergistic degradation were indicated from the perspective of mutual promoting degradation both with biotic and abiotic responses, and furthermore, the application and technical limitations of CAHs degradation enhanced via biotic and abiotic mutual promoting were reviewed and analyzed. Finally, the development of CAHs degradation technology in future was prospected.