The data of arterial pressure and diameter were measured in vivo before and after normal anastomosis and tensional anastomosis in canine femoral arteries at different time intervals. The arterial compliance was calculated, and the exponential form C = bemPa was employed to fit the compliance (C)- average pressure (Pa) curve. The relationship between compliance and average pressure was determined. The results showed that the compliance of arteries decreased after anastomosis, and the compliance of arteries after tensional anastomosis was lower than that of normal anastomosis at different time intervals. It was decreased most markedly at 14 days after tensional anastomosis. This indicated that the distensibility of tensional anastomotic artery was decreased more markedly than that of normal anastomotic artery. The function of artery was evidently affected by tensional anastomosis.
Anastomosis, Surgical ; methods ; Animals ; Compliance ; Dogs ; Female ; Femoral Artery ; physiopathology ; surgery ; Male ; Pressure

Based on the morphology and function of lymphatic vessel, and on the achievements of researches in the regulatory mechanism of lymphatic circulation, we fully considered the dynamic interaction of blood, interstitial fluid and lymph fluid; then we imitated and used Sungawa's method of analyzing the heart output, and finally set up a dynamic model for describing lymphatic circulation. Comparison of our calculating results with the data from Ikomi's experiment showed that they were identical, thus indicating that our model is of value in explaining the dynamic mechanism of lymphatic circulation. In this paper is especially calculated the relationship between lymph flow and massage frequency, which is useful for analyzing the effect of massage on the lymph flow rate with respect to this model.
Animals ; Computer Simulation ; Endothelium, Lymphatic ; cytology ; physiology ; Humans ; Lymph ; physiology ; Lymphatic Vessels ; physiology ; Models, Biological ; Nonlinear Dynamics ; Pressure ; Rabbits ; Rheology

Edema is a common pathological symptom, but its development mechanism is unknown. Based on the bearings of pressure upon interstitium structure and substantial exchange between plasma and interstitial fluid, a dymamics model describing the development of edema was set up. The model's theoretical results showed that the variations of interstitium pressure and structure due to imbalance of substantial exchange may lead to the development of edema, which is in accordance with recent clinical researches. Discussions on the dynamic mechanism of the development of edema proposed that the best way to prevent edema is instituting treatment before the interstitial structure being destroyed.
Edema ; physiopathology ; Humans ; Models, Biological ; Nonlinear Dynamics