No abstract available.
Dyskinesias

No abstract available.
Dyskinesias

No abstract available.
Dyskinesias ; Pregnancy

Oro-facial dyskinesia (OFD) is involuntary, abnormal, uncontrolled and stereotyped movements, consisting of forehead furrowing, eye opening and closing, smacking and pursing of the lips, lateral deviation and protrusion of the tongue, and occasionally lateral deviation and protrusion of the jaw.1 OFD is known to have various complications including speech difficulty, chewing and eating disorders, and social embarrassment; facial muscle stiffness, mucosal and gingival traumatic lesions. In addition, it may leads to cranio-mandibular joint (TMJ) complications in the presence of intense and prolonged abnormal movements, with pain and degeneration.1,2 There is no previous report of TMJ dislocation due to OFD. In this report, we describe a patient who developed bilateral anterior TMJ dislocation due to OFD which occurred following intra-cranial hemorrhage (ICH).
Movement Disorders ; Dyskinesias

No abstract available.
Dyskinesias ; Myotonia Congenita ; Myotonia

No abstract available.
Arm* ; Dyskinesias* ; Hand* ; Multiple Sclerosis*

Organic causes of astasia or asterixis have been described in the literature. However, concurrent unilateral manifestation of the two symptoms is extremely rare. We report two cases presenting with astasia and asterixis due to infarcts involving the ventrolateral nucleus of the left thalamus. Acute onset of astasia or asterixis in patients without significant metabolic disorder should alert the clinician on the possibility of acute stroke involving the thalamus.
Dyskinesias* ; Humans ; Infarction* ; Stroke ; Thalamus

It is well recognized that antiepileptic drugs (AEDs) can cause generalized or bilateral asterixis. However, the unilat-eral asterixis induced by AEDs has been rarely reported in patients with structural brain lesions. We report 2 patients who developed unilateral asterixis within therapeutic AEDs levels. In both patients, the unilateral asterixis disappeared when the daily dosage of AEDs was decreased. Our cases suggest that the cerebral hemisphere with structural lesions might be more vulnerable to the development of AEDs-induced asterixis.
Anticonvulsants* ; Brain* ; Cerebrum ; Dyskinesias* ; Humans

It has been reported that the antiparkinsonian efficacy of levodopa is reduced after long-term administration However, main parkinsonian motor symptoms, since they are mainly caused by the deficiency of nigrostriatal dopamine, should be corrected if sufficient dopamine is supplied exogenously. Therefore, the functional decline in patients with long-term levodopa therapy may result either from side effects such as response fluctuation and abnormal involuntary movements or from progression of doPa -unresponsive parkinsonian symptoms, instead of reduction in levodopa efficacy itself. To adress this question, we measured residual motor disability during and at 6 hours after continuous intravenous levodopa infusion with optimal dose for at least 16 hours in 54 patients with idiopathic Parkinson's disease. While the basal motor disability is increased according to the advance of symptom duration as well as Hoehn and Yahr stage, the residual motor disability after levodpa supplement is not changed. The duration of levodopa therapy until development of motor fluctuation is significantly shorter in good responder (residual motor disability<2. 0) than in poor r-ponder(residual motor disability>2.0), and positively correlated to the residual motor disability. These findings suggest; first, the functional decline observed in parkinsonian patients with chronic levodopa therapy mainly results from motor fluctuation and/or progression of dopa-unresponsive symptoms, not from decline of levodopa efficacy itself on cardinal motor symptoms; second, the parkinsonian patients with good levodopa response may develop motor fluctuation earlier than those with poor response.
Dihydroxyphenylalanine ; Dopamine ; Dyskinesias ; Humans ; Levodopa* ; Parkinson Disease

Diphenylbydantoin-induced movement disorders have been rarely reported. They include choreoathetosis, orofacial dyskinesia, asterixis, dystonia, and ballismus. A patient with epllepsy, who showed orofacial dyskinesia with toxic cerebellar syrnptoms after longterm use of diphenylhydantoin is presented. The involuntary movement rapidily disappeared with a reduction of diphenylhydantoin dose.
Dyskinesias ; Dystonia ; Humans ; Movement Disorders* ; Phenytoin