1.Does Helicobacter pylori Colonization Ameliorate Obese Host Glucose Metabolism?.
The Korean Journal of Gastroenterology 2012;60(6):394-396
No abstract available.
2.Helicobacter pylori Eradication Therapy, the Reasonable First Line Therapy for Gastric Mucosa-Associated Lymphoid Tissue Lymphoma Irrespective of Infection Status and Disease Stages.
Gut and Liver 2016;10(5):659-660
No abstract available.
Helicobacter pylori*
;
Helicobacter*
;
Lymphoma, B-Cell, Marginal Zone*
3.Atrophic Gastritis Increases the Risk of Gastric Cancer in Asymptomatic Population in Korea.
Gut and Liver 2017;11(5):575-576
No abstract available.
Gastritis, Atrophic*
;
Korea*
;
Stomach Neoplasms*
4.Atrophic Gastritis Increases the Risk of Gastric Cancer in Asymptomatic Population in Korea.
Gut and Liver 2017;11(5):575-576
No abstract available.
Gastritis, Atrophic*
;
Korea*
;
Stomach Neoplasms*
5.Helicobacter pylori in Human Stomach: Can It Be Called Mutualism or a Disease?.
The Korean Journal of Gastroenterology 2012;59(5):329-337
Helicobacter pylori (H. pylori) has been a major concern as a gastric pathogen with unique features since discovered in the end of the 20th century. Recent data on comparative genome study have revealed that H. pylori has successfully survived with its host though over 58,000 years of evolution and migration from continent to continent. To maintain the symbiotic relationship with human, H. pylori has come up with ways to induce host tolerance as well as exert harmful injuries. Studies about H. pylori have accumulated the knowledge about how the cellular and molecular interactions are controlled and regulated to decide whether the symbiotic relationship is directed to diseases or peaceful mutualism. We reviewed recent literatures and research outcomes about the H. pylori and host interaction in molecular and cellular basis.
Adaptive Immunity
;
Epithelial Cells/metabolism/microbiology/pathology
;
Helicobacter Infections/immunology/metabolism/*pathology
;
Helicobacter pylori/*immunology
;
*Host-Pathogen Interactions
;
Humans
;
Symbiosis
;
T-Lymphocytes/immunology/metabolism
6.Status and Literature Review of Self-Expandable Metallic Stents for Malignant Colorectal Obstruction.
Dae Young CHEUNG ; Yong Kook LEE ; Chang Heon YANG
Clinical Endoscopy 2014;47(1):65-73
Use of colorectal stents has increased dramatically over the last decades. Colorectal stents offer an alternative way to relieve fatal intestinal obstruction and can take place of emergency surgery, which associated with significant morbidity and mortality and a high incidence of stoma creation, to elective resection. Although there remain a few concerns regarding the use of stents as a bridge to surgical resection, use of self-expandable metallic stents for palliation in patients with unresectable disease has come to be generally accepted. Advantages of colorectal stents include acute restoration of luminal patency and allowance of time for proper staging and surgical optimization, and the well-known disadvantages are procedure-related complications including perforation, migration, and stent failure. General indications, procedures, and clinical outcomes as well as recent evidences regarding the use of colorectal stents will be discussed in this review.
Colorectal Neoplasms
;
Emergencies
;
Humans
;
Incidence
;
Intestinal Obstruction
;
Mortality
;
Phenobarbital
;
Stents*
8.Must-Have Knowledge about the Helicobacter pylori-Negative Gastric Cancer.
Jeong Ho KIM ; Dae Young CHEUNG
Gut and Liver 2016;10(2):157-159
No abstract available.
Antibodies, Bacterial
;
Helicobacter
;
Helicobacter Infections
;
Helicobacter pylori/*immunology
;
Humans
;
*Stomach Neoplasms
9.How to Interpret the Pathological Report before and after Endoscopic Submucosal Dissection of Early Gastric Cancer.
Dae Young CHEUNG ; Soo Heon PARK
Clinical Endoscopy 2016;49(4):327-331
Possible lymph node metastasis (LNM) and residual cancer are major concerns in endoscopic submucosal dissection (ESD) for early gastric cancer. To reduce the risk of LNM and cancer recurrence, the proper indications for ESD should be considered. Histology, size, depth of invasion, and presence of ulceration should be thoroughly evaluated before proceeding with ESD. However, with incomplete information, discrepancies often arise between the pathological diagnosis based on the forceps biopsy and that based on the totally resected specimen. In addition, the presence of lymphovascular involvement and histological homogeneity can be clarified only after ESD. If the pathological diagnosis changes after ESD, we should reevaluate the curativeness and reformulate the goal of treatment. Additional surgery is a reasonable strategy for non-curative ESD, but a patient's other health conditions should also be considered. It is simple to read pathological reports before and after ESD, but it can be a complicated art to interpret the report and formulate an optimal approach. In this review, various considerations regarding the pathological diagnosis will be discussed.
Biopsy
;
Diagnosis
;
Lymph Nodes
;
Neoplasm Metastasis
;
Neoplasm, Residual
;
Pathology
;
Recurrence
;
Stomach Neoplasms*
;
Surgical Instruments
;
Ulcer
10.Helicobacter pylori Eradication, a Gordian Knot for Idiopathic Thrombocytopenic Purpura?.
Gut and Liver 2016;10(3):323-324
No abstract available.
Helicobacter pylori*
;
Helicobacter*
;
Purpura, Thrombocytopenic, Idiopathic*
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