1.Broken Guidewire Fragment in the Radio-brachial Artery During Transradial Sheath Placement: Percutaneous Retrieval via Femoral Approach.
Jang Young KIM ; Junghan YOON ; Hyun Sook JUNG ; Woo Jea KIM ; Byung Su YOO ; Seung Hwan LEE ; Kyung Hoon CHOI
Yonsei Medical Journal 2005;46(1):166-168
A case in which a 0.014" wire was broken during the sheath placement in the radial artery for transradial coronary procedure is described here, and a successful retrieval of it using conventional methods is also described. Through the left femoral artery, the 6 Fr guiding catheter was advanced down to the tip of the broken wire at the brachial artery, and the distal part of the broken guidewire was captivated into the guiding catheter. By inflating the balloon catheter inside of the guiding catheter, seized broken guidewire between the inflated balloon and the guiding catheter was removed successfully by withdrawing the whole system en bloc.
Aged
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Angioplasty, Transluminal, Percutaneous Coronary/*adverse
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effects/instrumentation/*methods
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Brachial Artery
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Coronary Arteriosclerosis/diagnosis
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Equipment Failure
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Female
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Femoral Artery
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Foreign Bodies/*etiology/*therapy
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Humans
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Radial Artery
2.An Autopsy Case of Postpartum Acute Myocardial Infarction Associated with Postpartum Ergot Alkaloids Administration in Old-Aged Pregnant Women.
Minseob EOM ; Jeong Heon LEE ; Jae Hun CHUNG ; Ho LEE
Yonsei Medical Journal 2005;46(6):866-869
Cases of acute myocardial infarction (AMI) that occur during pregnancy or postpartum are rarely reported. Ergot derivatives are known to induce the spasmodic contraction of coronary arteries. Administration of ergot derivatives can cause AMI, even in normal healthy people. In several reported cases, ergot derivatives triggered severe AMI during the postpartum period. Here, we report the case of a forty-year-old woman who was successfully impregnated by artificial fertilization and died after treatment with ergot derivatives. The autopsy revealed AMI with severe coronary atherosclerosis. This is the first case that reports aggravation of pre-existent severe coronary atherosclerosis after postpartum infusion of ergot derivtives.
Pregnancy
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*Postpartum Period
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Myocardial Infarction/*chemically induced/diagnosis/pathology
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Maternal Age
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Humans
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Female
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Ergot Alkaloids/*adverse effects
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Coronary Arteriosclerosis/chemically induced/diagnosis/pathology
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Adult
3.Is Thyroid Hormone a Risk Factor of Coronary Atherosclerosis in Korean Patients?.
Kyung Ho YUN ; Myung Ho JEONG ; Dong Goo KANG ; Kye Hun KIM ; Sang Yup LIM ; Yeon Sang LEE ; Sang Hyun LEE ; Seo Na HONG ; Young Joon HONG ; Ju Han KIM ; Young Keun AHN ; Jeong Gwan CHO ; Jong Chun PARK ; Nam Ho KIM ; Seok Kyu OH ; Jin Won JEONG ; Jung Chaee KANG
Korean Circulation Journal 2005;35(1):43-48
BACKGROUND AND OBJECTIVES: Overt and subclinical hypothyroidism is known to be associated with developing atherosclerosis and adverse changes in blood lipid. There has been no data regarding the effects of a normal range of thyroid hormone on the presence of coronary atherosclerosis. SUBJECTS AND METHODS: We studied 1 25 consecutive patients (age: 60.0 +/-11.1 years, male: female=84:41) who underwent diagnostic coronary angiography. The clinical diagnoses on admission were stable angina (32.0%), unstable angina (53.6%), and acute myocardial infarction (14.4%). The thyroid hormones [thyroid stimulating hormone (TSH), free thyroxine and free 3 -iodothyronine], serum lipid levels, high sensitivity C-reactive protein (hsCRP) level, homocysteine and fibrinogen levels were measured. The coronary angiographic results were compared with laboratory findings. RESULTS: The angiographic diagnoses of coronary artery disease were no significant stenosis in 4 1 patients (32.8%), single vessel disease in 47 patients (37.6%) and multivessel disease in 37 patients (29.6%). The serum TSH levels showed a trend toward higher levels in the patients with multivessel disease compared to the patients with no significant stenosis (1.22+/-0.96 uIU/mL vs. 0.73+/-0.53 uIU/mL, respectively, p=0.053). According to the levels of TSH (<1.0 uIU/mL and > or =1.0 IU/mL), the incidence of multivessel disease was significantly higher in the patients with high serum TSH levels (23.1 % vs. 40.0%, respectively, p=0.038). A significant correlation was observed between the levels of TSH and the monocyte count (r=0.251, p=0.005), hsCRP level (r=0.208, p=0.023) and homocysteine level (r=0.279, p=0.002). CONCLUSION: The high level of serum TSH is associated with multivessel disease, abnormal inflammatory markers and high homocysteine levels.
Angina, Stable
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Angina, Unstable
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Arteriosclerosis
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Atherosclerosis
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C-Reactive Protein
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Constriction, Pathologic
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Coronary Angiography
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Coronary Artery Disease*
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Coronary Disease
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Diagnosis
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Fibrinogen
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Homocysteine
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Humans
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Hypothyroidism
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Incidence
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Male
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Monocytes
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Myocardial Infarction
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Reference Values
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Risk Factors*
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Thyroid Gland*
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Thyroid Hormones
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Thyroxine
4.Characteristics of Acute Cerebral Infarction in Patients with Familial Hypercholesterolemia
Jiah KIM ; So Hyun PARK ; Jeong Min KIM ; Sang Hak LEE ; Kwang Yeol PARK
Journal of the Korean Neurological Association 2019;37(4):361-367
BACKGROUND: The patients with familial hypercholesterolemia (FH) suffer from early onset atherosclerotic vascular disease due to high level of cholesterol and subsequent vascular inflammation, especially in the form of coronary artery disease. We investigated the clinical characteristics of FH associated cerebral infarction and its possible mechanism. METHODS: Between January 2014 and May 2017, acute cerebral infarction patients who admitted to Chung-Ang University Hospital were reviewed from stroke registry and the diagnosis of FH was made based on the Dutch Lipid Clinic Network Diagnostic Criteria for FH. We reviewed their initial laboratory and brain imaging information, prescribed medication and followed lipid profile after discharge. Stroke mechanism was determined based on Trial of ORG 10172 in Acute Stroke Treatment classification. RESULTS: Among 1,401 acute cerebral infarction or transient ischemic attack patients, one probable and three possible FH stroke patients were detected. All the patients denied of previous coronary artery disease history and initial lipid panel revealed high levels of total cholesterol (378±75 mg/dL) and low-density lipoprotein-cholesterol (238±56 mg/dL). Stroke mechanisms were heterogeneous, including one atherosclerotic, two vertebral artery dissection cases and one coagulation disorder. All the patients were combined with noticeable degree of intracranial atherosclerosis and were maintained with statin treatment. CONCLUSIONS: This study illustrates diverse stroke mechanism among stroke patients with FH. Further research is required to disclose exact incidence of FH among stroke population and effective treatment strategy.
Atherosclerosis
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Cerebral Infarction
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Cholesterol
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Classification
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Coronary Artery Disease
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Diagnosis
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Humans
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Hydroxymethylglutaryl-CoA Reductase Inhibitors
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Hyperlipoproteinemia Type II
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Incidence
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Inflammation
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Intracranial Arteriosclerosis
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Ischemic Attack, Transient
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Neuroimaging
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Stroke
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Vascular Diseases
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Vertebral Artery Dissection