The adrenal cortex of 11 adult cats of both sexes were partially removedand the mode of their degeneration and regeneration was studied. The regenera-tion began from the peripheral glomerulosa in form of bands protruding into thewound. The newly formed glomerulosal layer would give rise to the fasciculataand successively the reticularis. When the glomerulosa alone was injured or re-moved, such as in the shallow part of the wound, the fasciculata inside remain-ed healthy and came in direct contact with the regenerated capsule. Similarly,when the fasciculata degenerated, the reticularis still remained intact. In view of these findings, it is concluded that the cells of the superficiallayer may proliferate and give rise to cells in the deeper layers during regenera-tion. Once the zones are well differentiated each of them is maintained as anindependent unit, as there is no degeneration of inner zones following the removalof outer zones. Previous hypotheses of cellular migration or zonation formulatedby various authors are discussed. Since there was no transitional form betweenthe capsular cells and the cells of fasciculata, the hypothesis that glomerulosalcells originated from the capsule is denied. Although some glomerulosal cells wereseen in the substance of the capsule in certain sections, they were found, whentraced in serial sections, to be a protrution of the neighbouring glomerulosallayer. Extracellular pigments were found in the early stages of the wound butin the later stages pigments were found only inside the cells. The pigmentcells, therefore, are believed to be macrophage in nature.

A total of 26 adult rabbits were used in this study. Six were used as control andthe remaining 20 rabbits were injected with adrenaline to produce focal cardiac lesions.These animals were sacrificed alternatively either by medulla shock or calcium arrest after1, 3, 5, 7, 10, 14 and 21 days of experiment. The left ventricle was excised and sectioned.To the unfixed frozen sections, Chevremont and Frederic method for SH-group were used(Mecurric chloride or Mecurric acetate block were used as control), also Sudan blackB were used for staining fat. To paraffin sections, H-E and McManus PAS method(saliva-digestion as control) were stained to demonstrate the general structure and theglycogen. The following points of conclusion were drawn: 1) The adrenaline induced necrotic myocardial fibers were saliva-resisted PASpositive. 2) There were many degenerative fibers at the sides of the necrotic fibers orarround the lesions. These fibers showed fatty change, deposition of abnormal glycogenand deeper stain for SH-reaction. These fibers might regain to normal or progress tonecrotic. 3) The fatty change and the deposition of abnormal glycogen first appeared at thefirst day of the experiment. On the recovery of the muscle fibers, the abnormal glycogenregained earlier to normal than that of the fatty change. From this, the authersbelieved: The fatty change might be caused by the abnormal metabolism of the glycogen.This again proved the authorized work of pathologists: Fat burns in the flame of carbo-hydrate. 4) The relationship between the increase of SH-group and both the fatty changeand the abnormal glycogen deposition was discussed. 5) Of the fragmented cardiac fibers, the glycogen granules disappeared before itsnecrosis, and the fatty granules were removed later by the macrophages.

By using intracranial illumination, the surface outlines of the frontal sinuses of 65male Chinese skulls (85 cases) were studied. The frontal sinuses were grouped into 5types according to the relationship of the sinus with the orbit. Three types were visibleon the surface of the squamous portion of the frontal bones (76.5%), whereas the other2 types were only visible intraorbitally (23.5%). Of the 5 types, 4 types of sinuseswere intimately related with the orbit (84.7%). The clinical significance of the above mentioned facts was discussed.