No abstract available.
Brain Stem Infarctions*

Research on 150 patients suffer brain stroke at The Nervous Department of 103 Hospital from 4/2000 to 4/2002. Results: brain stroke rate take 8,18% inner patients. The rate of woman per man is 1/.2.20. The highest incidence rate is at 60-69 ages group (38,67%). 109 patients (72,67%) have risk factors and 144 times risk factors were reckon up. The typies of brain stroke distribute to brain infarct 76% (in which 53,33% brain artery thrombosis, 16/675 vacant syndrome, 6% vessel stuck, 24% haemorrhage inner brain (in which 13,33% intracerebral haemorrhage, 9,33% subarachnoid hemorrhage, 1,33% ventricle haemorrhage)
Brain Stem Infarctions ; Brain ; Hemorrhage

We reviewed 27 patients wlth cerebellar infarction which was demonstrated by brain CT and/or MRI. Infarction occurred in the territory of posterior inferior cerebellar artery (PICAj in 16 patients, and the territory of the superior cerebellar artery(SCA) was involved in 5 patients. Antenor inferior cerebellar artery(AICA) infarcts occurred in 3 patients. Both PICA and SCA temtories were involved in 2 patients. In the remaining 1 patient, the infarct encompassed the borderzone between the SCA and PICA territories. The main symptoms and signs were sudden onset of vertigo, dizziness, nausea, vomiting, dysmetria, ataxia, nystagmus, and headache. There were signs of associated brain stem infarction or occipitotemporal infarction; rostral basilar artery syndrome, classic SCA syndrome, Wallenberg syndrome, internuclear ophthalmoplegia, facial palsy, hearing impairment. Presumed cerebral embolism was the main stroke mechanism in the SCA terntories. Six patients with brainstem compression or brainstem involvement showed consciousness deterioration, and only one of them died as a result of extensive cerebellar infarctions involving both SCA and PICA territories Cerebellar infarction may run a more benign course than previously thought.
Arteries ; Ataxia ; Basilar Artery ; Brain ; Brain Stem ; Brain Stem Infarctions ; Cerebellar Ataxia ; Consciousness ; Dizziness ; Facial Paralysis ; Headache ; Hearing Loss ; Humans ; Infarction* ; Intracranial Embolism ; Lateral Medullary Syndrome ; Magnetic Resonance Imaging ; Nausea ; Ocular Motility Disorders ; Pica ; Stroke ; Vertigo ; Vomiting

Lateral medullary syndrome is one of the most well known and best characterized vascular syndromes of the vertebrobasilar circulation. This syndrome is most often caused by unilateral occlusive disease of the ipsilateral vertebral artery or its posterior inferior cerebellar artery branch. Bilateral lateral medullary syndromes are rare and the prognosis of this condition is grave. A 70-year-old hypertensive, diabetic woman developed sudden dizziness and gait ataxia. On neurologic examination, she had dysarthria, hoarseness, nystagmus, absent gag reflex, sensory changes on the right hemibody, and left limb and gait ataxia. Brain MRI revealed left middle and inferior lateral medullar infarction. Brain MR angiography revealed occlusion of the right vertebral artery and mild stenosis at the origin of the left vertebral artery with distal occlusion. Seven months later, she developed right lateral medullary syndrome. Repeated brain MRI revealed right upper lateral medullar and cerebellar infarction. We report a case of bilateral lateral medullary syndromes due to two consecutive medullary infarcts.
Aged ; Angiography ; Arteries ; Brain ; Brain Stem Infarctions ; Constriction, Pathologic ; Dizziness ; Dysarthria ; Extremities ; Female ; Gait Ataxia ; Hoarseness ; Humans ; Infarction ; Lateral Medullary Syndrome* ; Magnetic Resonance Imaging ; Neurologic Examination ; Prognosis ; Reflex ; Vertebral Artery

No abstract available.
Brain Stem Infarctions ; Infarction ; Trigeminal Neuralgia

No abstract available.
Brain ; Brain Stem ; Brain Stem Infarctions ; Cerebellar Ataxia ; Cerebral Infarction

Paralytic pontine extropia(PPE) is a exotropia in contralateral eye of lesion side, associated with one-and- a-half syndrome in acute phase of brainstem infarction and after then, often followed by non-paralytic pontine extropia(NPPE) and medial longitudinal fasciculus (MLF) syndrome in each clinical improvement. NPPE is pontine exotropia without lateral gaze palsy to ipsilateral side and is considered to be due to partial impairment of the unilateral paramedian pontine reticular formation(PPRF). We reported a case, 75 yearold man, of brainstem infarction who initially presented NPPE in acute phase, followed by ipsialteral MLF syndrome after 5th days.
Brain Stem Infarctions* ; Brain Stem* ; Brain* ; Exotropia* ; Paralysis

Lateral medullary infarction can lead to central respiratory failure. However, in Wallenberg's syndrome, obstructive respiratory failure rarely develops. Here, we report a case of obstructive respiratory failure with stridor caused by unilateral lateral medullary infarction. A 74-year-old woman was admitted for acute right lateral medullary infarction. On the 12th day after the stroke, the patient developed respiratory failure necessitating endotracheal intubation. She could maintain good oxygen saturation and ventilation in intubated status without the assistance of mechanical ventilator, even during sleep. The pharyngo-laryngoscopy demonstrated the hypomobility of bilateral vocal cords. This case suggests that unilateral medullary infarction might be a cause of bilateral vocal cord palsy.
Aged ; Airway Obstruction ; Brain Stem Infarctions ; Female ; Humans ; Infarction ; Intubation, Intratracheal ; Lateral Medullary Syndrome ; Medulla Oblongata ; Oxygen ; Respiratory Insufficiency ; Respiratory Sounds ; Stroke ; Ventilation ; Ventilators, Mechanical ; Vocal Cord Paralysis ; Vocal Cords

Brainstem infarction secondary to basilar artery occlusion often causes very-severe and life-threatening neurological conditions. For better recovery, early diagnosis is essential; however, this is not always easy because of ambiguous symptoms and limited information in some cases. We experienced two patients with basilar artery occlusion who presented with convulsive-like movements as an initial manifestation.
Basilar Artery ; Brain Stem Infarctions ; Early Diagnosis ; Humans ; Vertebrobasilar Insufficiency

Hypertrophic olivary degeneration is a form of transynaptic degeneration, which is caused by a lesion in the dentate-rubro-olivary pathway. Commonly described lesions were brainstem stroke, neoplasm, demyelination, and trauma. It's clinical presentations are Holmes tremor, and palatal tremor. This case was a 49-year-old man who was diagnosed as bilateral brainstem hemorrhage. About 2 months later, he had developed bilateral Holmes tremor of upper extremities and oculopalatal termor. Brain MRI was performed at 13 months after onset. MRI showed hyperintense and hypertrophied lesion on T2-weighted image in both inferior olivary nuclei.
Brain ; Brain Stem ; Brain Stem Infarctions ; Demyelinating Diseases ; Hemorrhage ; Humans ; Middle Aged ; Tremor ; Upper Extremity