1.An Unusual Cause of Gastrointestinal Hemorrhage: Gastrocolic Fistula Caused by Colon Cancer Invasion.
Jeong Hyeon CHO ; In Tae KIM ; Jin Yi CHOI ; Song Wook CHUN ; Beo Deul KANG ; Sang Kyun BAE ; Hee Man KIM ; Ji Sun SONG
Yeungnam University Journal of Medicine 2013;30(1):43-46
Gastrocolic fistula is a fistulous communication between the stomach and the colon. It is a passage between the gastric epithelium and the colonic epithelium. This uncommon complication is caused by benign and malignant diseases of the stomach or the colon. Its clinical manifestations include weight loss, diarrhea and fecal vomiting; occasionally, anemia, poor oral intake, fatigue and dizziness; and very rarely, gastrointestinal bleeding. In this paper, an unusual case of gastrocolic fistula accompanied by hematochezia, which was revealed to have been caused by colon cancer invasion, is described.
Anemia
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Colon
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Colonic Neoplasms
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Diarrhea
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Epithelium
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Fatigue
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Fistula
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Gastrointestinal Hemorrhage
;
Hemorrhage
;
Intestinal Fistula
;
Stomach
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Weight Loss
2.A Case of Successful Treatment of Portal Venous Gas Caused by Acute Pancreatitis.
Shi Heon DONG ; Hyeon Geun CHO ; Jeong Hoon BAEK ; Beo Deul KANG ; Mi Sung KIM ; Jae Hee CHO ; Jeong Hoon LEE ; Song Wook CHUN
The Korean Journal of Gastroenterology 2013;61(3):170-173
Hepatic portal venous gas (HPVG) has been considered a rare entity associated with a poor prognosis. Portal vein gas is most commonly caused by mesenteric ischemia but may have a variety other causes. HPVG can be associated with ischemic bowel disease, inflammatory bowel disease, intra-abdominal abscess, small bowel obstruction, acute pancreatitis, and gastric ulcer. Because of high mortality rate, most HPVG requires emergent surgical interventions and intensive medical management. We experienced a case of hepatic portal venous gas caused by acute pancreatitis and successfully treated with medical management.
Acute Disease
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Anti-Bacterial Agents/therapeutic use
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Gases/metabolism
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Humans
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Male
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Middle Aged
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Pancreatitis/*diagnosis/drug therapy/radiography
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Portal Vein/radiography
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Tomography, X-Ray Computed
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Vascular Diseases/*diagnosis/drug therapy/radiography
3.Extrinsic Indentation at Gastric Fundus by Splenic Lymphangioma.
Shi Heon DONG ; Hee Man KIM ; Jae Hee CHO ; Hee Woo LEE ; Seung Won LEE ; Beo Deul KANG ; Sun Ok SONG ; Sang Yeop YI
Korean Journal of Gastrointestinal Endoscopy 2011;42(1):20-23
Splenic lymphangioma is a very rare benign condition, and it is classified as one of the cystic proliferations of the spleen. This is considered to result from developmental malformation of the lymphatic system. Splenic lymphangioma is usually seen in children and it is often found incidentally. Herein, we report on an unusual case of splenic lymphangioma in an adult. A 66-year-old woman presented with abdominal pain. On esophagogastroduodenoscopy, the gastric fundus was externally compressed by an extrinsic mass. Computed tomography revealed multiple cystic masses in the spleen. Laparoscopic splenectomy was then performed. The histology revealed multiple splenic lymphangiomas. This case showed an unusual presentation of splenic lymphangioma as gastric extrinsic compression, and this should be examined by imaging studies.
Abdominal Pain
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Adult
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Aged
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Child
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Endoscopy, Digestive System
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Female
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Gastric Fundus
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Humans
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Lymphangioma
;
Lymphatic System
;
Spleen
;
Splenectomy
4.A Case of IgG4 Associated Sclerosing Cholangitis without Clinical Manifestations of Autoimmune Pancreatitis.
Song Wook CHUN ; Ja Sung CHOI ; Beo Deul KANG ; Yu Jin KIM ; Ki Jun HAN ; Hyeon Geun CHO ; Hwa Eun OH ; Jae Hee CHO
The Korean Journal of Gastroenterology 2013;62(1):69-74
IgG4-related systemic diseases are characterized by a diffuse or mass forming inflammatory reaction rich in lymphocytes and IgG4-positive plasma cells (lymphoplasmacytic infiltration), fibrosclerosis of variable organs and obliterative phlebitis. They usually involve various organs including the pancreas, bile duct, gallbladder, salivary gland, retroperitoneum, kidney, lung, and prostate. However, most of them are accompanied by autoimmune pancreatitis, and good response to steroid treatment is one of the hallmarks of this disease. We report a case of an 67-year-old man with IgG4 associated sclerosing cholangitis, who was diagnosed by endoscopic retrograde cholangiopancreatography and successfully treated with steroid therapy.
Aged
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Anti-Inflammatory Agents/therapeutic use
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Autoimmune Diseases/complications/diagnosis
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Bile Ducts, Intrahepatic/pathology/ultrasonography
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Cholangiopancreatography, Endoscopic Retrograde
;
Cholangitis, Sclerosing/complications/*diagnosis/drug therapy
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Common Bile Duct/pathology/ultrasonography
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Humans
;
Immunoglobulin G/*blood
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Immunohistochemistry
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Male
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Pancreatitis/complications/diagnosis
;
Prednisolone/therapeutic use
;
Tomography, X-Ray Computed
5.Ifosfamide-induced Fanconi syndrome with diabetes insipidus.
Ah Young LEEM ; Han Sang KIM ; Byung Woo YOO ; Beo Deul KANG ; Min Hwan KIM ; Sun Young RHA ; Hyo Song KIM
The Korean Journal of Internal Medicine 2014;29(2):246-249
Ifosfamide-induced Fanconi syndrome is a rare complication that typically occurs in young patients due to a cumulative dose of ifosfamide > 40-60 g/m2, a reduction in kidney mass, or concurrent cisplatin treatment. It is usually characterized by severe and fatal progression accompanied by type II proximal renal tubular dysfunction, as evidenced by glycosuria, proteinuria, electrolyte loss, and metabolic acidosis. Diabetes insipidus is also a rare complication of ifosfamide-induced renal disease. We herein describe a case involving a 61-year-old man who developed ifosfamide-induced Fanconi syndrome accompanied by diabetes insipidus only a few days after the first round of chemotherapy. He had no known risk factors. In addition, we briefly review the mechanisms and possible therapeutic options for this condition based on other cases in the literature. Patients who receive ifosfamide must be closely monitored for renal impairment to avoid this rare but fatal complication.
Acidosis/chemically induced
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Antineoplastic Agents, Alkylating/*adverse effects
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Chemotherapy, Adjuvant
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Diabetes Insipidus/*chemically induced/diagnosis/therapy
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Fanconi Syndrome/*chemically induced/diagnosis/therapy
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Fatal Outcome
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Histiocytoma, Malignant Fibrous/*drug therapy/pathology
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Humans
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Ifosfamide/*adverse effects
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Male
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Middle Aged
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Neoadjuvant Therapy/*adverse effects
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Time Factors
6.A Case of IgA Nephropathy with a Membranoproliferative Glomerulonephritis-like Pattern Presenting as Massive Ascites in a Patient with Alcoholic Liver Disease.
Sun Ok SONG ; Seung Won LEE ; Hee Woo LEE ; Beo Deul KANG ; Shi Heon DONG ; Ja Sung CHOI ; Ji Sun SONG ; Soo Young YOON ; Sang Choel LEE
Korean Journal of Nephrology 2009;28(2):152-156
IgA nephropathy can occur commonly in alcoholic liver cirrhosis and is the most common form of secondary IgA nephropathy. Defective clearance of IgA-containing complexes by liver is thought to contribute to the development of IgA nephropathy in alcoholic cirrhosis. Although IgA deposition is found up to 64% in autopsy cases of alcoholic cirrhosis, most patients have mild clinical symptoms, and nephrotic range proteinuria occurs rarely in these patients. We report a case of IgA nephropathy with a membranoproliferative pattern that is detected with unusual massive ascites in a patient with alcoholic liver disease. A 60-year-old male patient was referred to our hospital for evaluation and management of abdominal distension. Abdominal ultrasonographic findings were compatible with diffuse liver cirrhosis with splenomegaly and large amount of ascites. He had nephrotic range proteinuria, azotemia, hyperlipidemia, and hematuria in dipstick. Renal biopsy performed under the impression of acute nephritis revealed mesangial and endocapillary proliferative glomerulonephritis with double contour of capillary loop. Immunofluorescence findings showed mesangial IgA and C3 deposit, compatible with IgA nephropathy. He was treated with high dose steroid, and steroid was tapered during 2 months. Steroid treatment induced complete remission state, and ascites was resolved.
Alcoholics
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Ascites
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Autopsy
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Azotemia
;
Biopsy
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Capillaries
;
Fluorescent Antibody Technique
;
Glomerulonephritis
;
Glomerulonephritis, IGA
;
Glomerulonephritis, Membranoproliferative
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Hematuria
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Humans
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Hyperlipidemias
;
Immunoglobulin A
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Liver
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Liver Cirrhosis
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Liver Cirrhosis, Alcoholic
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Liver Diseases, Alcoholic
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Male
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Middle Aged
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Nephritis
;
Proteinuria
;
Splenomegaly
7.Guillain-Barre Syndrome in a Patient with Chemotherapy for HIV Related Burkitt's Lymphoma.
Hee Woo LEE ; Sun Ok SONG ; Beo Deul KANG ; Shi Heon DONG ; Yuri KIM ; Youn Hee PARK ; Hyuk Min LEE ; Kkot Sil LEE
Infection and Chemotherapy 2011;43(2):206-209
Human immunodeficiency virus infection is not a common cause of Guillain-Barre syndrome. Guillain-Barre syndrome with cerebrospinal fluid pleocytosis has been associated with early human immunodeficiency virus (HIV) infection, occasionally as the presenting manifestation. We report a case of 73-year-old Korean malen with acute motor axonal variant of Guillain-Barre syndrome during chemotherapy for HIV- related Burkitt's lymphoma. This is the first report of Guillain-Barre syndrome occurringed within HIV infection in Korea.
Aged
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Axons
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Burkitt Lymphoma
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Guillain-Barre Syndrome
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HIV
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HIV Infections
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Humans
;
Korea
;
Leukocytosis
8.A Case of Occupational Hypersensitivity Pneumonitis Associated with Trichloroethylene.
Young Jae KIM ; Eu Dong HWANG ; Ah Young LEEM ; Beo Deul KANG ; Soo Yun CHANG ; Ho Keun KIM ; In Kyu PARK ; Song Yee KIM ; Eun Young KIM ; Ji Ye JUNG ; Young Ae KANG ; Moo Suk PARK ; Young Sam KIM ; Se Kyu KIM ; Joon CHANG ; Kyung Soo CHUNG
Tuberculosis and Respiratory Diseases 2014;76(2):75-79
Trichloroethylene (TCE) is a toxic chemical commonly used as a degreasing agent, and it is usually found in a colorless or blue liquid form. TCE has a sweet, chloroform-like odor, and this volatile chlorinated organic chemical can cause toxic hepatitis, neurophysiological disorders, skin disorders, and hypersensitivity syndromes. However, the hypersensitivity pneumonitis (HP) attributed to TCE has rarely been reported. We hereby describe a case of HP associated with TCE in a 29-year-old man who was employed as a lead welder at a computer repair center. He was installing the capacitors on computer chip boards and had been wiped down with TCE. He was admitted to our hospital with complaints of dry coughs, night sweats, and weight losses for the past two months. HP due to TCE exposure was being suspected due to his occupational history, and the results of a video-associated thoracoscopic biopsy confirmed the suspicions. Symptoms have resolved after the steroid pulse therapy and his occupational change. TCE should be taken into consideration as a potential trigger of HP. Early recognition and avoidance of the TCE exposure in the future is important for the treatment of TCE induced HP.
Adult
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Alveolitis, Extrinsic Allergic*
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Biopsy
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Cough
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Drug-Induced Liver Injury
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Humans
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Hypersensitivity*
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Lung Diseases
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Occupational Exposure
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Odors
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Skin
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Sweat
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Trichloroethylene*
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Weight Loss